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Conference Paper: The role of the tumor suppressor gene THY1 in suppression of epithelial-mesenchymal transition (EMT) in nasopharyngeal carcinoma

TitleThe role of the tumor suppressor gene THY1 in suppression of epithelial-mesenchymal transition (EMT) in nasopharyngeal carcinoma
Other TitlesThe role of the tumor suppressor gene THY1 in suppression of EMT in NPC
Authors
KeywordsMedical sciences
Oncology
Issue Date2014
PublisherAmerican Association for Cancer Research. The Journal's web site is located at http://cancerres.aacrjournals.org/
Citation
The 105th Annual Meeting of the American Association for Cancer Research (AACR 2014), San Diego, CA., 5-9 April 2014. In Cancer Research, 2014, v. 74 n. 19 suppl., abstract no. 1150 How to Cite?
AbstractBACKGROUND AND AIMS: THY1 has been successfully identified as a tumor suppressor gene (TSG) in nasopharyngeal carcinoma (NPC) in our laboratory. The frequency of down-regulated THY1 protein expression was found to be significantly associated with NPC lymph node metastases. However, direct functional evidence is still lacking for THY1 being anti-metastatic in NPC. Epithelial-Mesenchymal Transition (EMT) is well-known as a closely associated process for stem cells as well as tumor cells to invade to other part of the body. We aim to study the association of EMT with the THY1 in NPC. METHODS: Functional analysis of ...
DescriptionTumor Biology - Poster Presentations - Proffered Abstracts - Poster Presentations - Epithelial-Mesenchymal Transitions in Tumor Metastasis: abstract no. 1150
This journal suppl. entitled: Proceedings: AACR Annual Meeting 2014; April 5-9, 2014 ...
Persistent Identifierhttp://hdl.handle.net/10722/198638
ISSN
2023 Impact Factor: 12.5
2023 SCImago Journal Rankings: 3.468
ISI Accession Number ID

 

DC FieldValueLanguage
dc.contributor.authorLung, HL-
dc.contributor.authorLung, ML-
dc.date.accessioned2014-07-07T08:26:46Z-
dc.date.available2014-07-07T08:26:46Z-
dc.date.issued2014-
dc.identifier.citationThe 105th Annual Meeting of the American Association for Cancer Research (AACR 2014), San Diego, CA., 5-9 April 2014. In Cancer Research, 2014, v. 74 n. 19 suppl., abstract no. 1150-
dc.identifier.issn0008-5472-
dc.identifier.urihttp://hdl.handle.net/10722/198638-
dc.descriptionTumor Biology - Poster Presentations - Proffered Abstracts - Poster Presentations - Epithelial-Mesenchymal Transitions in Tumor Metastasis: abstract no. 1150-
dc.descriptionThis journal suppl. entitled: Proceedings: AACR Annual Meeting 2014; April 5-9, 2014 ...-
dc.description.abstractBACKGROUND AND AIMS: THY1 has been successfully identified as a tumor suppressor gene (TSG) in nasopharyngeal carcinoma (NPC) in our laboratory. The frequency of down-regulated THY1 protein expression was found to be significantly associated with NPC lymph node metastases. However, direct functional evidence is still lacking for THY1 being anti-metastatic in NPC. Epithelial-Mesenchymal Transition (EMT) is well-known as a closely associated process for stem cells as well as tumor cells to invade to other part of the body. We aim to study the association of EMT with the THY1 in NPC. METHODS: Functional analysis of ...-
dc.languageeng-
dc.publisherAmerican Association for Cancer Research. The Journal's web site is located at http://cancerres.aacrjournals.org/-
dc.relation.ispartofCancer Research-
dc.subjectMedical sciences-
dc.subjectOncology-
dc.titleThe role of the tumor suppressor gene THY1 in suppression of epithelial-mesenchymal transition (EMT) in nasopharyngeal carcinoma-
dc.title.alternativeThe role of the tumor suppressor gene THY1 in suppression of EMT in NPC-
dc.typeConference_Paper-
dc.identifier.emailLung, HL: hllung2@hku.hk-
dc.identifier.emailLung, ML: mlilung@hku.hk-
dc.identifier.authorityLung, HL=rp00299-
dc.identifier.authorityLung, ML=rp00300-
dc.description.naturepostprint-
dc.identifier.doi10.1158/1538-7445.AM2014-1150-
dc.identifier.hkuros229926-
dc.identifier.volume74-
dc.identifier.issue19 suppl., abstract no. 1150-
dc.identifier.isiWOS:000349906900388-
dc.publisher.placeUnited States-
dc.identifier.issnl0008-5472-

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