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Conference Paper: Spectral changes in resting-state fMRI connectivity induced by corpus callosum transection

TitleSpectral changes in resting-state fMRI connectivity induced by corpus callosum transection
Authors
Issue Date2013
PublisherInternational Society for Magnetic Resonance in Medicine (ISMRM).
Citation
The 21st Annual Meeting & Exhibition of the International Society for Magnetic Resonance in Medicine (ISMRM 2013), Salt Lake City, UT., 20-26 April 2013. In Society of Magnetic Resonance in Medicine Proceedings, 2013, v. 21, p. 3268 How to Cite?
AbstractChanges in connectivity strength or spatial map of the low-frequency fluctuations in resting-state fMRI are the common focus when comparing the normal and diseased brains. However, limited work has studied the underlying spectral alterations. In this study, we investigated the spectral changes of resting-state connectivity in a rat model of complete corpus callosum (CC) transection. Interhemispheric connectivity was disappeared in cortical regions where the corresponding callosal connections were severed. Spectral analysis of these regions showed stronger power at relative high frequency around 0.1 Hz in transection group than in sham group. These spectral changes were observed in brain regions showing predominately intrahemispheric connectivity and therefore may arise from the faster intrahemispheric communication. Our experimental findings indicate that spectral characteristics of rsfMRI connectivity can be modulated by neural disruption and spectral analysis of rsfMRI data may provide a new dimension of information regarding the brain organization and connectivity. (Abstract by ISMRM)
DescriptionElectronic Poster Session: Functional MRI (neuro) - Resting State Connectivity: Methods
ISMRM Merit Award: Magna cum Laude
Persistent Identifierhttp://hdl.handle.net/10722/191642
ISSN

 

DC FieldValueLanguage
dc.contributor.authorZhou, IYen_US
dc.contributor.authorLiang, YXen_US
dc.contributor.authorChan, RWen_US
dc.contributor.authorCheng, JSen_US
dc.contributor.authorGao, PPen_US
dc.contributor.authorSo, KFen_US
dc.contributor.authorWu, EX-
dc.date.accessioned2013-10-15T07:14:49Z-
dc.date.available2013-10-15T07:14:49Z-
dc.date.issued2013en_US
dc.identifier.citationThe 21st Annual Meeting & Exhibition of the International Society for Magnetic Resonance in Medicine (ISMRM 2013), Salt Lake City, UT., 20-26 April 2013. In Society of Magnetic Resonance in Medicine Proceedings, 2013, v. 21, p. 3268en_US
dc.identifier.issn1557-3672-
dc.identifier.urihttp://hdl.handle.net/10722/191642-
dc.descriptionElectronic Poster Session: Functional MRI (neuro) - Resting State Connectivity: Methods-
dc.descriptionISMRM Merit Award: Magna cum Laude-
dc.description.abstractChanges in connectivity strength or spatial map of the low-frequency fluctuations in resting-state fMRI are the common focus when comparing the normal and diseased brains. However, limited work has studied the underlying spectral alterations. In this study, we investigated the spectral changes of resting-state connectivity in a rat model of complete corpus callosum (CC) transection. Interhemispheric connectivity was disappeared in cortical regions where the corresponding callosal connections were severed. Spectral analysis of these regions showed stronger power at relative high frequency around 0.1 Hz in transection group than in sham group. These spectral changes were observed in brain regions showing predominately intrahemispheric connectivity and therefore may arise from the faster intrahemispheric communication. Our experimental findings indicate that spectral characteristics of rsfMRI connectivity can be modulated by neural disruption and spectral analysis of rsfMRI data may provide a new dimension of information regarding the brain organization and connectivity. (Abstract by ISMRM)-
dc.languageengen_US
dc.publisherInternational Society for Magnetic Resonance in Medicine (ISMRM).-
dc.relation.ispartofSociety of Magnetic Resonance in Medicine Proceedingsen_US
dc.titleSpectral changes in resting-state fMRI connectivity induced by corpus callosum transectionen_US
dc.typeConference_Paperen_US
dc.identifier.emailZhou, IY: iriszhou@eee.hku.hken_US
dc.identifier.emailSo, KF: hrmaskf@hku.hken_US
dc.identifier.emailWu, EX: ewu1@hkucc.hku.hk-
dc.identifier.authorityZhou, IY=rp01739en_US
dc.identifier.authoritySo, KF=rp00329en_US
dc.description.naturelink_to_OA_fulltext-
dc.identifier.hkuros226015en_US
dc.identifier.volume21-
dc.identifier.spage3268en_US
dc.identifier.epage3268en_US
dc.publisher.placeUnited Statesen_US
dc.identifier.issnl1065-9889-

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