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Conference Paper: Downregulation of miR-199b-5p is associated with chemoresistance via Jagged1 mediated notch signaling in human ovarian cancer cells

TitleDownregulation of miR-199b-5p is associated with chemoresistance via Jagged1 mediated notch signaling in human ovarian cancer cells
Authors
Liu, XChan, WNgan, YS
KeywordsMedical sciences
Oncology
Issue Date2012
PublisherPergamon. The Journal's web site is located at http://www.elsevier.com/locate/ejca
Citation
The 22nd Annual Congress of the European Association of Cancer Research (EACR-22), Barcelona, Spain, 7-10 July 2012. In European Journal of Cancer, 2012, v. 48 suppl. 5, p. S129, abstract no. 545 How to Cite?
DOI: http://dx.doi.org/10.1016/S0959-8049(12)71204-2
AbstractBACKGROUND: Ovarian cancer is one of the most deadly gynaecological malignancies worldwide. Adjuvant chemotherapy combined with other treatments is the current management of the advanced ovarian cancers. However, the acquisition of resistance to initially responsive tumors limits the successful curative rate. The underlying molecular mechanism remains largely unknown. Here, we report that the downregulation of miR-199b-5p leading to elevated JAG1-mediated NOTCH signal...
DescriptionThis journal suppl. entitled: 22nd Biennial Congress of the European Association for Cancer Research - proceedings of EACR 22
Persistent Identifierhttp://hdl.handle.net/10722/153220
ISSN
0959-8049
2023 Impact Factor: 7.6
2023 SCImago Journal Rankings: 2.501
ISI Accession Number ID
WOS:000313036501048

 

DC FieldValueLanguage
dc.contributor.authorLiu, Xen_US
dc.contributor.authorChan, Wen_US
dc.contributor.authorNgan, YSen_US
dc.date.accessioned2012-07-16T10:00:23Z-
dc.date.available2012-07-16T10:00:23Z-
dc.date.issued2012en_US
dc.identifier.citationThe 22nd Annual Congress of the European Association of Cancer Research (EACR-22), Barcelona, Spain, 7-10 July 2012. In European Journal of Cancer, 2012, v. 48 suppl. 5, p. S129, abstract no. 545en_US
dc.identifier.issn0959-8049-
dc.identifier.urihttp://hdl.handle.net/10722/153220-
dc.descriptionThis journal suppl. entitled: 22nd Biennial Congress of the European Association for Cancer Research - proceedings of EACR 22-
dc.description.abstractBACKGROUND: Ovarian cancer is one of the most deadly gynaecological malignancies worldwide. Adjuvant chemotherapy combined with other treatments is the current management of the advanced ovarian cancers. However, the acquisition of resistance to initially responsive tumors limits the successful curative rate. The underlying molecular mechanism remains largely unknown. Here, we report that the downregulation of miR-199b-5p leading to elevated JAG1-mediated NOTCH signal...-
dc.languageengen_US
dc.publisherPergamon. The Journal's web site is located at http://www.elsevier.com/locate/ejca-
dc.relation.ispartofEuropean Journal of Canceren_US
dc.rightsNOTICE: this is the author’s version of a work that was accepted for publication in European Journal of Cancer. Changes resulting from the publishing process, such as peer review, editing, corrections, structural formatting, and other quality control mechanisms may not be reflected in this document. Changes may have been made to this work since it was submitted for publication. A definitive version was subsequently published in European Journal of Cancer, v. 48 suppl. 5, July 2012, doi: 10.1016/S0959-8049(12)71204-2-
dc.rightsThis work is licensed under a Creative Commons Attribution-NonCommercial-NoDerivatives 4.0 International License.-
dc.subjectMedical sciences-
dc.subjectOncology-
dc.titleDownregulation of miR-199b-5p is associated with chemoresistance via Jagged1 mediated notch signaling in human ovarian cancer cellsen_US
dc.typeConference_Paperen_US
dc.identifier.emailChan, W: dwchan@hku.hken_US
dc.identifier.emailNgan, YS: hysngan@hkucc.hku.hken_US
dc.identifier.authorityChan, W=rp00543en_US
dc.identifier.authorityNgan, YS=rp00346en_US
dc.description.naturepostprint-
dc.identifier.doi10.1016/S0959-8049(12)71204-2-
dc.identifier.hkuros201592en_US
dc.identifier.volume48-
dc.identifier.issuesuppl. 5-
dc.identifier.spageS129, abstract no. 545-
dc.identifier.epageS129, abstract no. 545-
dc.identifier.isiWOS:000313036501048-
dc.publisher.placeUnited Kingdom-
dc.identifier.issnl0959-8049-

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