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Article: Biodegradation and enzymatic responses in the marine diatom Skeletonema costatum upon exposure to 2,4-dichlorophenol

TitleBiodegradation and enzymatic responses in the marine diatom Skeletonema costatum upon exposure to 2,4-dichlorophenol
Authors
Keywords2,4-Dichlorophenol
Degradation
Detoxification
Diatom
Enzyme
Issue Date2002
PublisherElsevier BV. The Journal's web site is located at http://www.elsevier.com/locate/aquatox
Citation
Aquatic Toxicology, 2002, v. 59 n. 3-4, p. 191-200 How to Cite?
AbstractThe biodegradation and responses of selected detoxification and antioxidant enzymes in the marine diatom, Skeletonema costatum, upon exposure to sublethal concentrations of 2,4-dichlorophenol (2,4-DCP) were investigated. Results show that 2,4-DCP was readily metabolised, but bioaccumulation and adsorption were negligible. Glutathione S-transferase, ascorbate peroxidase and superoxide dismutase activities were increased markedly after exposure to 2,4-DCP for 96 h, while no appreciable change in peroxidase activity was observed. The addition of exogeneous glutathione to diatom culture enhanced the degradation of 2,4-DCP, and promoted diatom growth. The inhibition of glutathione synthesis enhanced the toxicity of 2,4-DCP. These results suggest that glutathione conjugation was one of the principal mechanisms involved in the degradation of 2,4-DCP in this diatom. Copyright © 2002 Elsevier Science B.V.
Persistent Identifierhttp://hdl.handle.net/10722/92783
ISSN
2023 Impact Factor: 4.1
2023 SCImago Journal Rankings: 1.099
ISI Accession Number ID
References

 

DC FieldValueLanguage
dc.contributor.authorYang, Sen_HK
dc.contributor.authorWu, RSSen_HK
dc.contributor.authorKong, RYCen_HK
dc.date.accessioned2010-09-17T10:57:02Z-
dc.date.available2010-09-17T10:57:02Z-
dc.date.issued2002en_HK
dc.identifier.citationAquatic Toxicology, 2002, v. 59 n. 3-4, p. 191-200en_HK
dc.identifier.issn0166-445Xen_HK
dc.identifier.urihttp://hdl.handle.net/10722/92783-
dc.description.abstractThe biodegradation and responses of selected detoxification and antioxidant enzymes in the marine diatom, Skeletonema costatum, upon exposure to sublethal concentrations of 2,4-dichlorophenol (2,4-DCP) were investigated. Results show that 2,4-DCP was readily metabolised, but bioaccumulation and adsorption were negligible. Glutathione S-transferase, ascorbate peroxidase and superoxide dismutase activities were increased markedly after exposure to 2,4-DCP for 96 h, while no appreciable change in peroxidase activity was observed. The addition of exogeneous glutathione to diatom culture enhanced the degradation of 2,4-DCP, and promoted diatom growth. The inhibition of glutathione synthesis enhanced the toxicity of 2,4-DCP. These results suggest that glutathione conjugation was one of the principal mechanisms involved in the degradation of 2,4-DCP in this diatom. Copyright © 2002 Elsevier Science B.V.en_HK
dc.languageengen_HK
dc.publisherElsevier BV. The Journal's web site is located at http://www.elsevier.com/locate/aquatoxen_HK
dc.relation.ispartofAquatic Toxicologyen_HK
dc.subject2,4-Dichlorophenolen_HK
dc.subjectDegradationen_HK
dc.subjectDetoxificationen_HK
dc.subjectDiatomen_HK
dc.subjectEnzymeen_HK
dc.titleBiodegradation and enzymatic responses in the marine diatom Skeletonema costatum upon exposure to 2,4-dichlorophenolen_HK
dc.typeArticleen_HK
dc.identifier.emailWu, RSS: rudolfwu@hku.hken_HK
dc.identifier.authorityWu, RSS=rp01398en_HK
dc.description.naturelink_to_subscribed_fulltext-
dc.identifier.doi10.1016/S0166-445X(01)00252-1en_HK
dc.identifier.pmid12127736-
dc.identifier.scopuseid_2-s2.0-0035996782en_HK
dc.relation.referenceshttp://www.scopus.com/mlt/select.url?eid=2-s2.0-0035996782&selection=ref&src=s&origin=recordpageen_HK
dc.identifier.volume59en_HK
dc.identifier.issue3-4en_HK
dc.identifier.spage191en_HK
dc.identifier.epage200en_HK
dc.identifier.isiWOS:000177503600005-
dc.publisher.placeNetherlandsen_HK
dc.identifier.scopusauthoridYang, S=7406950684en_HK
dc.identifier.scopusauthoridWu, RSS=7402945079en_HK
dc.identifier.scopusauthoridKong, RYC=7005290687en_HK
dc.identifier.issnl0166-445X-

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