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Article: Targeting adipocyte ESRRA promotes osteogenesis and vascular formation in adipocyte-rich bone marrow

TitleTargeting adipocyte ESRRA promotes osteogenesis and vascular formation in adipocyte-rich bone marrow
Authors
Issue Date4-May-2024
PublisherNature Portfolio
Citation
Nature Communications, 2024, v. 15, n. 1 How to Cite?
AbstractExcessive bone marrow adipocytes (BMAds) accumulation often occurs under diverse pathophysiological conditions associated with bone deterioration. Estrogen-related receptor α (ESRRA) is a key regulator responding to metabolic stress. Here, we show that adipocyte-specific ESRRA deficiency preserves osteogenesis and vascular formation in adipocyte-rich bone marrow upon estrogen deficiency or obesity. Mechanistically, adipocyte ESRRA interferes with E2/ESR1 signaling resulting in transcriptional repression of secreted phosphoprotein 1 (Spp1); yet positively modulates leptin expression by binding to its promoter. ESRRA abrogation results in enhanced SPP1 and decreased leptin secretion from both visceral adipocytes and BMAds, concertedly dictating bone marrow stromal stem cell fate commitment and restoring type H vessel formation, constituting a feed-forward loop for bone formation. Pharmacological inhibition of ESRRA protects obese mice against bone loss and high marrow adiposity. Thus, our findings highlight a therapeutic approach via targeting adipocyte ESRRA to preserve bone formation especially in detrimental adipocyte-rich bone milieu.
Persistent Identifierhttp://hdl.handle.net/10722/346279

 

DC FieldValueLanguage
dc.contributor.authorHuang, Tongling-
dc.contributor.authorLu, Zhaocheng-
dc.contributor.authorWang, Zihui-
dc.contributor.authorCheng, Lixin-
dc.contributor.authorGao, Lu-
dc.contributor.authorGao, Jun-
dc.contributor.authorZhang, Ning-
dc.contributor.authorGeng, Chang An-
dc.contributor.authorZhao, Xiaoli-
dc.contributor.authorWang, Huaiyu-
dc.contributor.authorWong, Chi Wai-
dc.contributor.authorYeung, Kelvin WK-
dc.contributor.authorPan, Haobo-
dc.contributor.authorLu, William Weijia-
dc.contributor.authorGuan, Min-
dc.date.accessioned2024-09-13T00:30:10Z-
dc.date.available2024-09-13T00:30:10Z-
dc.date.issued2024-05-04-
dc.identifier.citationNature Communications, 2024, v. 15, n. 1-
dc.identifier.urihttp://hdl.handle.net/10722/346279-
dc.description.abstractExcessive bone marrow adipocytes (BMAds) accumulation often occurs under diverse pathophysiological conditions associated with bone deterioration. Estrogen-related receptor α (ESRRA) is a key regulator responding to metabolic stress. Here, we show that adipocyte-specific ESRRA deficiency preserves osteogenesis and vascular formation in adipocyte-rich bone marrow upon estrogen deficiency or obesity. Mechanistically, adipocyte ESRRA interferes with E2/ESR1 signaling resulting in transcriptional repression of secreted phosphoprotein 1 (Spp1); yet positively modulates leptin expression by binding to its promoter. ESRRA abrogation results in enhanced SPP1 and decreased leptin secretion from both visceral adipocytes and BMAds, concertedly dictating bone marrow stromal stem cell fate commitment and restoring type H vessel formation, constituting a feed-forward loop for bone formation. Pharmacological inhibition of ESRRA protects obese mice against bone loss and high marrow adiposity. Thus, our findings highlight a therapeutic approach via targeting adipocyte ESRRA to preserve bone formation especially in detrimental adipocyte-rich bone milieu.-
dc.languageeng-
dc.publisherNature Portfolio-
dc.relation.ispartofNature Communications-
dc.rightsThis work is licensed under a Creative Commons Attribution-NonCommercial-NoDerivatives 4.0 International License.-
dc.titleTargeting adipocyte ESRRA promotes osteogenesis and vascular formation in adipocyte-rich bone marrow-
dc.typeArticle-
dc.identifier.doi10.1038/s41467-024-48255-8-
dc.identifier.pmid38704393-
dc.identifier.scopuseid_2-s2.0-85192051887-
dc.identifier.volume15-
dc.identifier.issue1-
dc.identifier.eissn2041-1723-
dc.identifier.issnl2041-1723-

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