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postgraduate thesis: Role of Epac1 and Epac2 in gut microbiota (GM) dysregulation and metabolic syndrome

TitleRole of Epac1 and Epac2 in gut microbiota (GM) dysregulation and metabolic syndrome
Authors
Virwani, Preeti Dinesh
Advisors
Advisor(s):Chung, SKWang, Y
Issue Date2018
PublisherThe University of Hong Kong (Pokfulam, Hong Kong)
Citation
Virwani, P. D.. (2018). Role of Epac1 and Epac2 in gut microbiota (GM) dysregulation and metabolic syndrome. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR.
DegreeDoctor of Philosophy
SubjectMetabolic syndrome
Gastrointestinal system - Microbiology
Cyclic adenylic acid
Dept/ProgramBiomedical Sciences
Persistent Identifierhttp://hdl.handle.net/10722/312804

 

DC FieldValueLanguage
dc.contributor.advisorChung, SK-
dc.contributor.advisorWang, Y-
dc.contributor.authorVirwani, Preeti Dinesh-
dc.date.accessioned2022-05-13T06:30:35Z-
dc.date.available2022-05-13T06:30:35Z-
dc.date.issued2018-
dc.identifier.citationVirwani, P. D.. (2018). Role of Epac1 and Epac2 in gut microbiota (GM) dysregulation and metabolic syndrome. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR.-
dc.identifier.urihttp://hdl.handle.net/10722/312804-
dc.languageeng-
dc.publisherThe University of Hong Kong (Pokfulam, Hong Kong)-
dc.relation.ispartofHKU Theses Online (HKUTO)-
dc.rightsThe author retains all proprietary rights, (such as patent rights) and the right to use in future works.-
dc.rightsThis work is licensed under a Creative Commons Attribution-NonCommercial-NoDerivatives 4.0 International License.-
dc.subject.lcshMetabolic syndrome-
dc.subject.lcshGastrointestinal system - Microbiology-
dc.subject.lcshCyclic adenylic acid-
dc.titleRole of Epac1 and Epac2 in gut microbiota (GM) dysregulation and metabolic syndrome-
dc.typePG_Thesis-
dc.description.thesisnameDoctor of Philosophy-
dc.description.thesislevelDoctoral-
dc.description.thesisdisciplineBiomedical Sciences-
dc.description.naturepublished_or_final_version-
dc.date.hkucongregation2019-
dc.identifier.mmsid991044505314803414-

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