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Article: Nomenclature of Toso, Fas apoptosis inhibitory molecule 3, and IgM FcR

TitleNomenclature of Toso, Fas apoptosis inhibitory molecule 3, and IgM FcR
Authors
Issue Date2015
Citation
Journal of Immunology, 2015, v. 194, n. 9, p. 4055-4057 How to Cite?
AbstractCopyright © 2015 by The American Association of Immunologists, Inc. Hiromi Kubagawa and John E. Coligan coordinated an online meeting to define an appropriate nomenclature for the cell surface glycoprotein presently designated by different names: Toso, Fas apoptosis inhibitory molecule 3 (FAIM3), and IgM FcR (FcμR). FAIM3 and Faim3 are the currently approved symbols for the human and mouse genes, respectively, in the National Center for Biotechnology Information, Ensembl, and other databases. However, recent functional results reported by several groups of investigators strongly support a recommendation for renaming FAIM3/Faim3 as FCMR/Fcmr, a name better reflecting its physiological function as the FcR for IgM. Participants included 12 investigators involved in studying Toso/FAIM3(Faim3)/FμR, representatives from the Human Genome Nomenclature Committee (Ruth Seal) and the Mouse Genome Nomenclature Committee (Monica McAndrews), and an observer from the IgM research field (Michael Carroll). In this article, we provide a brief background of the key research on the Toso/FAIM3(Faim3)/FcμR proteins, focusing on the ligand specificity and functional activity, followed by a brief summary of discussion about adopting a single name for this molecule and its gene and a resulting recommendation for genome nomenclature committees.
Persistent Identifierhttp://hdl.handle.net/10722/292877
ISSN
2023 Impact Factor: 3.6
2023 SCImago Journal Rankings: 1.558
ISI Accession Number ID

 

DC FieldValueLanguage
dc.contributor.authorKubagawa, Hiromi-
dc.contributor.authorCarroll, Michael C.-
dc.contributor.authorJacob, Chaim O.-
dc.contributor.authorLang, Karl S.-
dc.contributor.authorLee, Kyeong Hee-
dc.contributor.authorMak, Tak-
dc.contributor.authorMcAndrews, Monica-
dc.contributor.authorMorse, Herbert C.-
dc.contributor.authorNolan, Garry P.-
dc.contributor.authorOhno, Hiroshi-
dc.contributor.authorRichter, Günther H.-
dc.contributor.authorSeal, Ruth-
dc.contributor.authorWang, Ji Yang-
dc.contributor.authorWiestner, Adrian-
dc.contributor.authorColigan, John E.-
dc.date.accessioned2020-11-17T14:57:24Z-
dc.date.available2020-11-17T14:57:24Z-
dc.date.issued2015-
dc.identifier.citationJournal of Immunology, 2015, v. 194, n. 9, p. 4055-4057-
dc.identifier.issn0022-1767-
dc.identifier.urihttp://hdl.handle.net/10722/292877-
dc.description.abstractCopyright © 2015 by The American Association of Immunologists, Inc. Hiromi Kubagawa and John E. Coligan coordinated an online meeting to define an appropriate nomenclature for the cell surface glycoprotein presently designated by different names: Toso, Fas apoptosis inhibitory molecule 3 (FAIM3), and IgM FcR (FcμR). FAIM3 and Faim3 are the currently approved symbols for the human and mouse genes, respectively, in the National Center for Biotechnology Information, Ensembl, and other databases. However, recent functional results reported by several groups of investigators strongly support a recommendation for renaming FAIM3/Faim3 as FCMR/Fcmr, a name better reflecting its physiological function as the FcR for IgM. Participants included 12 investigators involved in studying Toso/FAIM3(Faim3)/FμR, representatives from the Human Genome Nomenclature Committee (Ruth Seal) and the Mouse Genome Nomenclature Committee (Monica McAndrews), and an observer from the IgM research field (Michael Carroll). In this article, we provide a brief background of the key research on the Toso/FAIM3(Faim3)/FcμR proteins, focusing on the ligand specificity and functional activity, followed by a brief summary of discussion about adopting a single name for this molecule and its gene and a resulting recommendation for genome nomenclature committees.-
dc.languageeng-
dc.relation.ispartofJournal of Immunology-
dc.titleNomenclature of Toso, Fas apoptosis inhibitory molecule 3, and IgM FcR-
dc.typeArticle-
dc.description.naturelink_to_OA_fulltext-
dc.identifier.doi10.4049/jimmunol.1500222-
dc.identifier.pmid25888699-
dc.identifier.scopuseid_2-s2.0-84928473008-
dc.identifier.volume194-
dc.identifier.issue9-
dc.identifier.spage4055-
dc.identifier.epage4057-
dc.identifier.eissn1550-6606-
dc.identifier.isiWOS:000353727400001-
dc.identifier.issnl0022-1767-

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