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Article: SREBP1c-CRY1 signalling represses hepatic glucose production by promoting FOXO1 degradation during refeeding

TitleSREBP1c-CRY1 signalling represses hepatic glucose production by promoting FOXO1 degradation during refeeding
Authors
Jang, HLee, GYSelby, CPLee, GJeon, YGLee, JHCheng, KYTitchenell, PBirnbaum, MJXu, ASancar, AKim, JB
Issue Date2016
PublisherNature Publishing Group. The Journal's web site is located at http://www.nature.com/ncomms/index.html
Citation
Nature Communications, 2016, v. 7, p. 12180:1-14 How to Cite?
DOI: http://dx.doi.org/10.1038/ncomms12180
Persistent Identifierhttp://hdl.handle.net/10722/232082
ISSN
2041-1723
2023 Impact Factor: 14.7
2023 SCImago Journal Rankings: 4.887
PubMed Central ID
PMC4947181
ISI Accession Number ID
WOS:000380747700001

 

DC FieldValueLanguage
dc.contributor.authorJang, H-
dc.contributor.authorLee, GY-
dc.contributor.authorSelby, CP-
dc.contributor.authorLee, G-
dc.contributor.authorJeon, YG-
dc.contributor.authorLee, JH-
dc.contributor.authorCheng, KY-
dc.contributor.authorTitchenell, P-
dc.contributor.authorBirnbaum, MJ-
dc.contributor.authorXu, A-
dc.contributor.authorSancar, A-
dc.contributor.authorKim, JB-
dc.date.accessioned2016-09-20T05:27:36Z-
dc.date.available2016-09-20T05:27:36Z-
dc.date.issued2016-
dc.identifier.citationNature Communications, 2016, v. 7, p. 12180:1-14-
dc.identifier.issn2041-1723-
dc.identifier.urihttp://hdl.handle.net/10722/232082-
dc.languageeng-
dc.publisherNature Publishing Group. The Journal's web site is located at http://www.nature.com/ncomms/index.html-
dc.relation.ispartofNature Communications-
dc.rightsThis work is licensed under a Creative Commons Attribution-NonCommercial-NoDerivatives 4.0 International License.-
dc.titleSREBP1c-CRY1 signalling represses hepatic glucose production by promoting FOXO1 degradation during refeeding-
dc.typeArticle-
dc.identifier.emailCheng, KY: dorncky@hkucc.hku.hk-
dc.identifier.emailXu, A: amxu@hkucc.hku.hk-
dc.identifier.authorityCheng, KY=rp01672-
dc.identifier.authorityXu, A=rp00485-
dc.description.naturepublished_or_final_version-
dc.identifier.doi10.1038/ncomms12180-
dc.identifier.pmcidPMC4947181-
dc.identifier.scopuseid_2-s2.0-84978629050-
dc.identifier.hkuros267178-
dc.identifier.volume7-
dc.identifier.spage12180:1-
dc.identifier.epage14-
dc.identifier.isiWOS:000380747700001-
dc.publisher.placeUnited Kingdom-
dc.identifier.f1000726505493-
dc.identifier.issnl2041-1723-

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