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Article: Dinucleotide repeat polymorphisms within the Flt-1 gene in minimal change nephropathy

TitleDinucleotide repeat polymorphisms within the Flt-1 gene in minimal change nephropathy
Authors
Parry, RGGillespie, KMClark, AGBMathieson, PW
Issue Date1999
Citation
European Journal of Immunogenetics, 1999, v. 26 n. 5, p. 321-323 How to Cite?
DOI: http://dx.doi.org/10.1046/j.1365-2370.1999.00161.x
AbstractDysregulation of Flt-1, a major receptor for vascular permeability factor (VPF), may provide a mechanism for the development of proteinuria in minimal change nephropathy (MCN). The gene for Flt-1 has a polymorphic dinucleotide repeat. We have demonstrated an 88% predominance of one allele and an 80% rate of homozygosity for this polymorphism with no association with MCN.
Persistent Identifierhttp://hdl.handle.net/10722/195380
ISSN
0960-7420
ISI Accession Number ID
WOS:000083338400001

 

DC FieldValueLanguage
dc.contributor.authorParry, RG-
dc.contributor.authorGillespie, KM-
dc.contributor.authorClark, AGB-
dc.contributor.authorMathieson, PW-
dc.date.accessioned2014-02-28T06:12:04Z-
dc.date.available2014-02-28T06:12:04Z-
dc.date.issued1999-
dc.identifier.citationEuropean Journal of Immunogenetics, 1999, v. 26 n. 5, p. 321-323-
dc.identifier.issn0960-7420-
dc.identifier.urihttp://hdl.handle.net/10722/195380-
dc.description.abstractDysregulation of Flt-1, a major receptor for vascular permeability factor (VPF), may provide a mechanism for the development of proteinuria in minimal change nephropathy (MCN). The gene for Flt-1 has a polymorphic dinucleotide repeat. We have demonstrated an 88% predominance of one allele and an 80% rate of homozygosity for this polymorphism with no association with MCN.-
dc.languageeng-
dc.relation.ispartofEuropean Journal of Immunogenetics-
dc.titleDinucleotide repeat polymorphisms within the Flt-1 gene in minimal change nephropathy-
dc.typeArticle-
dc.description.naturelink_to_subscribed_fulltext-
dc.identifier.doi10.1046/j.1365-2370.1999.00161.x-
dc.identifier.pmid10553496-
dc.identifier.scopuseid_2-s2.0-0032872758-
dc.identifier.volume26-
dc.identifier.issue5-
dc.identifier.spage321-
dc.identifier.epage323-
dc.identifier.isiWOS:000083338400001-
dc.identifier.issnl0960-7420-

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