Article: Adipose tissue and the metabolic syndrome: Focusing on adiponectin and several novel adipokines
| Title | Adipose tissue and the metabolic syndrome: Focusing on adiponectin and several novel adipokines |
|---|---|
| Authors | Tso, AWK1 Xu, A1 Chow, WS1 Lam, KSL1 |
| Keywords | Adipocyte fatty acid-binding protein Adipokines Adiponectin Insulin resistance Lipocalin-2 Retinol-binding protein 4 The Metabolic syndrome |
| Issue Date | 2008 |
| Publisher | Future Medicine Ltd. The Journal's web site is located at http://www.futuremedicine.com/loi/bmm |
| Citation | Biomarkers In Medicine, 2008, v. 2 n. 3, p. 239-252 [How to Cite?] DOI: http://dx.doi.org/10.2217/17520363.2.3.239 |
| Abstract | The metabolic syndrome represents a cluster of metabolic risk factors that predispose an individual to an increased risk for Type 2 diabetes, cardiovascular diseases and their associated morbidity and mortality. Visceral obesity is thought to be a major culprit. Adipokines secreted from the adipose tissue are now believed to be key factors mediating the metabolic and inflammatory effects of obesity. In this review, we shall examine the evidence suggesting that several novel adipokines, adiponectin, adipocyte fatty acid-binding protein, retinol-binding protein-4 and lipocalin-2, may hold promise as important clinical biomarkers to identify individuals at risk for the metabolic syndrome and related comorbidities. © 2008 Future Medicine Ltd. |
| ISSN | 1752-0363 2011 Impact Factor: 2.63 2011 SCImago Journal Rankings: 0.163 |
| DOI | http://dx.doi.org/10.2217/17520363.2.3.239 |
| ISI Accession Number ID | WOS:000258921400009 |
| References | References in Scopus |
| dc.contributor.author | Tso, AWK |
|---|---|
| dc.contributor.author | Xu, A |
| dc.contributor.author | Chow, WS |
| dc.contributor.author | Lam, KSL |
| dc.date.accessioned | 2010-09-06T07:33:09Z |
| dc.date.available | 2010-09-06T07:33:09Z |
| dc.date.issued | 2008 |
| dc.description.abstract | The metabolic syndrome represents a cluster of metabolic risk factors that predispose an individual to an increased risk for Type 2 diabetes, cardiovascular diseases and their associated morbidity and mortality. Visceral obesity is thought to be a major culprit. Adipokines secreted from the adipose tissue are now believed to be key factors mediating the metabolic and inflammatory effects of obesity. In this review, we shall examine the evidence suggesting that several novel adipokines, adiponectin, adipocyte fatty acid-binding protein, retinol-binding protein-4 and lipocalin-2, may hold promise as important clinical biomarkers to identify individuals at risk for the metabolic syndrome and related comorbidities. © 2008 Future Medicine Ltd. |
| dc.description.nature | Link_to_subscribed_fulltext |
| dc.identifier.citation | Biomarkers In Medicine, 2008, v. 2 n. 3, p. 239-252 [How to Cite?] DOI: http://dx.doi.org/10.2217/17520363.2.3.239 |
| dc.identifier.doi | http://dx.doi.org/10.2217/17520363.2.3.239 |
| dc.identifier.epage | 252 |
| dc.identifier.hkuros | 145457 |
| dc.identifier.isi | WOS:000258921400009 |
| dc.identifier.issn | 1752-0363 2011 Impact Factor: 2.63 2011 SCImago Journal Rankings: 0.163 |
| dc.identifier.issue | 3 |
| dc.identifier.openurl | ![]() |
| dc.identifier.scopus | eid_2-s2.0-48249114800 |
| dc.identifier.spage | 239 |
| dc.identifier.uri | http://hdl.handle.net/10722/77553 |
| dc.identifier.volume | 2 |
| dc.language | eng |
| dc.publisher | Future Medicine Ltd. The Journal's web site is located at http://www.futuremedicine.com/loi/bmm |
| dc.publisher.place | United Kingdom |
| dc.relation.ispartof | Biomarkers in Medicine |
| dc.relation.references | References in Scopus |
| dc.subject | Adipocyte fatty acid-binding protein |
| dc.subject | Adipokines |
| dc.subject | Adiponectin |
| dc.subject | Insulin resistance |
| dc.subject | Lipocalin-2 |
| dc.subject | Retinol-binding protein 4 |
| dc.subject | The Metabolic syndrome |
| dc.title | Adipose tissue and the metabolic syndrome: Focusing on adiponectin and several novel adipokines |
| dc.type | Article |
Author Affiliations
- The University of Hong Kong


