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Article: Antralization at the edge of proximal gastric ulcers: Does Helicobacter pylori infection play a role?

TitleAntralization at the edge of proximal gastric ulcers: Does Helicobacter pylori infection play a role?
Authors
Issue Date2003
PublisherBeijing Baishideng BioMed Scientific Co., Ltd. The Journal's web site is located at http://www.wjgnet.com/1007-9327/index.htm
Citation
World Journal Of Gastroenterology, 2003, v. 9 n. 6, p. 1265-1269 How to Cite?
AbstractAim: To determine the prevalence of antralization at the edge of proximal gastric ulcers, and the effect of H. pylori eradication on the mucosal appearances. Methods: Biopsies were taken from the antrum, body and the ulcer edge of patients with benign proximal gastric ulcers before and one year after treatment. Gastric mucosa was classified as antral, transitional or body type. H. pylori positive patients received either triple therapy, or omeprazole. Results: Patients with index ulcers in the incisura, body or fundus (n=116) were analyzed. Antral-type mucosa was more prevalent at the ulcer edge in H. pylori-positive patients than H. pylori-negative patients (93 % vs 60 %, OR=8.95, 95 %CI: 2.47-32.4, P=0.001). At one year, there was a significant reduction in the prevalence of antralization (from 93 % to 61 %, P=0.004) at the ulcer edge in patients with H. pylori being eradicated. However, there was no difference in the prevalence of antralization at the ulcer edge in those with persistent infection. Conclusion: H. pylori infection is associated with antralization at the edge of proximal gastric ulcers, which may be reversible in some patients after eradication of the infection.
Persistent Identifierhttp://hdl.handle.net/10722/76699
ISSN
2014 Impact Factor: 2.369
2013 SCImago Journal Rankings: 1.022
ISI Accession Number ID
References

 

DC FieldValueLanguage
dc.contributor.authorXia, HHXen_HK
dc.contributor.authorLam, SKen_HK
dc.contributor.authorWong, WMen_HK
dc.contributor.authorHu, WHCen_HK
dc.contributor.authorLai, KCen_HK
dc.contributor.authorWong, SHen_HK
dc.contributor.authorLeung, SYen_HK
dc.contributor.authorYuen, STen_HK
dc.contributor.authorWright, NAen_HK
dc.contributor.authorWong, BCYen_HK
dc.date.accessioned2010-09-06T07:24:00Z-
dc.date.available2010-09-06T07:24:00Z-
dc.date.issued2003en_HK
dc.identifier.citationWorld Journal Of Gastroenterology, 2003, v. 9 n. 6, p. 1265-1269en_HK
dc.identifier.issn1007-9327en_HK
dc.identifier.urihttp://hdl.handle.net/10722/76699-
dc.description.abstractAim: To determine the prevalence of antralization at the edge of proximal gastric ulcers, and the effect of H. pylori eradication on the mucosal appearances. Methods: Biopsies were taken from the antrum, body and the ulcer edge of patients with benign proximal gastric ulcers before and one year after treatment. Gastric mucosa was classified as antral, transitional or body type. H. pylori positive patients received either triple therapy, or omeprazole. Results: Patients with index ulcers in the incisura, body or fundus (n=116) were analyzed. Antral-type mucosa was more prevalent at the ulcer edge in H. pylori-positive patients than H. pylori-negative patients (93 % vs 60 %, OR=8.95, 95 %CI: 2.47-32.4, P=0.001). At one year, there was a significant reduction in the prevalence of antralization (from 93 % to 61 %, P=0.004) at the ulcer edge in patients with H. pylori being eradicated. However, there was no difference in the prevalence of antralization at the ulcer edge in those with persistent infection. Conclusion: H. pylori infection is associated with antralization at the edge of proximal gastric ulcers, which may be reversible in some patients after eradication of the infection.en_HK
dc.languageengen_HK
dc.publisherBeijing Baishideng BioMed Scientific Co., Ltd. The Journal's web site is located at http://www.wjgnet.com/1007-9327/index.htm en_HK
dc.relation.ispartofWorld Journal of Gastroenterologyen_HK
dc.subject.meshAgeden_HK
dc.subject.meshAnti-Bacterial Agentsen_HK
dc.subject.meshAnti-Ulcer Agents - therapeutic useen_HK
dc.subject.meshDrug Therapy, Combination - therapeutic useen_HK
dc.subject.meshFemaleen_HK
dc.subject.meshGastric Mucosa - pathologyen_HK
dc.subject.meshHelicobacter Infections - complications - drug therapyen_HK
dc.subject.meshHelicobacter pylorien_HK
dc.subject.meshHumansen_HK
dc.subject.meshMaleen_HK
dc.subject.meshMiddle Ageden_HK
dc.subject.meshOmeprazole - therapeutic useen_HK
dc.subject.meshPyloric Antrum - pathologyen_HK
dc.subject.meshStomach Ulcer - drug therapy - microbiology - pathologyen_HK
dc.titleAntralization at the edge of proximal gastric ulcers: Does Helicobacter pylori infection play a role?en_HK
dc.typeArticleen_HK
dc.identifier.openurlhttp://library.hku.hk:4550/resserv?sid=HKU:IR&issn=1007-9327&volume=9&issue=6&spage=1265&epage=1269&date=2003&atitle=Antralization+at+the+Edge+of+Proximal+Gastric+Ulcers:+Does+Helicobacter+pylori+Infection+Play+a+Role?en_HK
dc.identifier.emailLeung, SY:suetyi@hkucc.hku.hken_HK
dc.identifier.emailWong, BCY:bcywong@hku.hken_HK
dc.identifier.authorityLeung, SY=rp00359en_HK
dc.identifier.authorityWong, BCY=rp00429en_HK
dc.description.naturelink_to_subscribed_fulltext-
dc.identifier.pmid12800237en_HK
dc.identifier.scopuseid_2-s2.0-0037713705en_HK
dc.identifier.hkuros80443en_HK
dc.relation.referenceshttp://www.scopus.com/mlt/select.url?eid=2-s2.0-0037713705&selection=ref&src=s&origin=recordpageen_HK
dc.identifier.volume9en_HK
dc.identifier.issue6en_HK
dc.identifier.spage1265en_HK
dc.identifier.epage1269en_HK
dc.identifier.isiWOS:000183763000026-
dc.publisher.placeChinaen_HK
dc.identifier.scopusauthoridXia, HHX=36784990000en_HK
dc.identifier.scopusauthoridLam, SK=7402279473en_HK
dc.identifier.scopusauthoridWong, WM=7403972413en_HK
dc.identifier.scopusauthoridHu, WHC=7404359791en_HK
dc.identifier.scopusauthoridLai, KC=7402135595en_HK
dc.identifier.scopusauthoridWong, SH=7404590306en_HK
dc.identifier.scopusauthoridLeung, SY=7202044886en_HK
dc.identifier.scopusauthoridYuen, ST=7103160927en_HK
dc.identifier.scopusauthoridWright, NA=7201531909en_HK
dc.identifier.scopusauthoridWong, BCY=7402023340en_HK

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