Article: Telbivudine versus lamivudine in patients with chronic hepatitis B
| Title | Telbivudine versus lamivudine in patients with chronic hepatitis B |
|---|---|
| Authors | Lai, CL3 Gane, E7 Liaw, YF12 Hsu, CW12 Thongsawat, S6 Wang, Y11 Chen, Y4 Heathcote, EJ13 Rasenack, J14 Bzowej, N9 Naoumov, NV1 Di Bisceglie, AM8 Zeuzem, S10 Moon, YM5 Goodman, Z2 Chao, G15 Constance, BF15 Brown, NA15 |
| Issue Date | 2007 |
| Publisher | Massachusetts Medical Society. The Journal's web site is located at http://content.nejm.org/ |
| Citation | New England Journal Of Medicine, 2007, v. 357 n. 25, p. 2576-2588 [How to Cite?] DOI: http://dx.doi.org/10.1056/NEJMoa066422 |
| Abstract | BACKGROUND: Reducing hepatitis B virus (HBV) replication to minimal levels is emerging as a key therapeutic goal for chronic hepatitis B. METHODS: In this double-blind, phase 3 trial, 1370 patients with chronic hepatitis B were randomly assigned to receive 600 mg of telbivudine or 100 mg of lamivudine once daily. The primary efficacy end point was noninferiority of telbivudine to lamivudine for therapeutic response (i.e., a reduction in serum HBV DNA levels to fewer than 5 log 10 copies per milliliter, along with loss of hepatitis B e antigen [HBeAg] or normalization of alanine aminotransferase levels). Secondary efficacy measures included histologic response, changes in serum HBV DNA levels, and HBeAg responses. RESULTS: At week 52, a significantly higher proportion of HBeAg-positive patients receiving telbivudine than of those receiving lamivudine had a therapeutic response (75.3% vs. 67.0%, P = 0.005) or a histologic response (64.7% vs. 56.3%, P = 0.01); telbivudine also was not inferior to lamivudine for these end points in HBeAg-negative patients. In HBeAg-positive and HBeAg-negative patients, telbivudine was superior to lamivudine with respect to the mean reduction in the number of copies of HBV DNA from baseline, the proportion of patients with a reduction in HBV DNA to levels undetectable by polymerase-chain-reaction assay, and development of resistance to the drug. Elevated creatine kinase levels were more common in patients who received telbivudine, whereas elevated alanine aminotransferase and aspartate aminotransferase levels were more common in those who received lamivudine. CONCLUSIONS: Among patients with HBeAg-positive chronic hepatitis B, the rates of therapeutic and histologic response at 1 year were significantly higher in patients treated with telbivudine than in patients treated with lamivudine. In both the HBeAg-negative and the HBeAg-positive groups, telbivudine demonstrated greater HBV DNA suppression with less resistance than did lamivudine. (ClinicalTrials.gov number, NCT00057265.) Copyright © 2007 Massachusetts Medical Society. |
| ISSN | 0028-4793 2011 Impact Factor: 53.298 2011 SCImago Journal Rankings: 3.412 |
| DOI | http://dx.doi.org/10.1056/NEJMoa066422 |
| ISI Accession Number ID | WOS:000251728900008 |
| References | References in Scopus |
| dc.contributor.author | Lai, CL |
|---|---|
| dc.contributor.author | Gane, E |
| dc.contributor.author | Liaw, YF |
| dc.contributor.author | Hsu, CW |
| dc.contributor.author | Thongsawat, S |
| dc.contributor.author | Wang, Y |
| dc.contributor.author | Chen, Y |
| dc.contributor.author | Heathcote, EJ |
| dc.contributor.author | Rasenack, J |
| dc.contributor.author | Bzowej, N |
| dc.contributor.author | Naoumov, NV |
| dc.contributor.author | Di Bisceglie, AM |
| dc.contributor.author | Zeuzem, S |
| dc.contributor.author | Moon, YM |
| dc.contributor.author | Goodman, Z |
| dc.contributor.author | Chao, G |
| dc.contributor.author | Constance, BF |
| dc.contributor.author | Brown, NA |
| dc.date.accessioned | 2010-04-12T01:39:08Z |
| dc.date.available | 2010-04-12T01:39:08Z |
| dc.date.issued | 2007 |
| dc.description.abstract | BACKGROUND: Reducing hepatitis B virus (HBV) replication to minimal levels is emerging as a key therapeutic goal for chronic hepatitis B. METHODS: In this double-blind, phase 3 trial, 1370 patients with chronic hepatitis B were randomly assigned to receive 600 mg of telbivudine or 100 mg of lamivudine once daily. The primary efficacy end point was noninferiority of telbivudine to lamivudine for therapeutic response (i.e., a reduction in serum HBV DNA levels to fewer than 5 log 10 copies per milliliter, along with loss of hepatitis B e antigen [HBeAg] or normalization of alanine aminotransferase levels). Secondary efficacy measures included histologic response, changes in serum HBV DNA levels, and HBeAg responses. RESULTS: At week 52, a significantly higher proportion of HBeAg-positive patients receiving telbivudine than of those receiving lamivudine had a therapeutic response (75.3% vs. 67.0%, P = 0.005) or a histologic response (64.7% vs. 56.3%, P = 0.01); telbivudine also was not inferior to lamivudine for these end points in HBeAg-negative patients. In HBeAg-positive and HBeAg-negative patients, telbivudine was superior to lamivudine with respect to the mean reduction in the number of copies of HBV DNA from baseline, the proportion of patients with a reduction in HBV DNA to levels undetectable by polymerase-chain-reaction assay, and development of resistance to the drug. Elevated creatine kinase levels were more common in patients who received telbivudine, whereas elevated alanine aminotransferase and aspartate aminotransferase levels were more common in those who received lamivudine. CONCLUSIONS: Among patients with HBeAg-positive chronic hepatitis B, the rates of therapeutic and histologic response at 1 year were significantly higher in patients treated with telbivudine than in patients treated with lamivudine. In both the HBeAg-negative and the HBeAg-positive groups, telbivudine demonstrated greater HBV DNA suppression with less resistance than did lamivudine. (ClinicalTrials.gov number, NCT00057265.) Copyright © 2007 Massachusetts Medical Society. |
| dc.description.nature | published_or_final_version |
| dc.identifier.citation | New England Journal Of Medicine, 2007, v. 357 n. 25, p. 2576-2588 [How to Cite?] DOI: http://dx.doi.org/10.1056/NEJMoa066422 |
| dc.identifier.doi | http://dx.doi.org/10.1056/NEJMoa066422 |
| dc.identifier.epage | 2588 |
| dc.identifier.hkuros | 149250 |
| dc.identifier.isi | WOS:000251728900008 |
| dc.identifier.issn | 0028-4793 2011 Impact Factor: 53.298 2011 SCImago Journal Rankings: 3.412 |
| dc.identifier.issue | 25 |
| dc.identifier.openurl | ![]() |
| dc.identifier.pmid | 18094378 |
| dc.identifier.scopus | eid_2-s2.0-37349120537 |
| dc.identifier.spage | 2576 |
| dc.identifier.uri | http://hdl.handle.net/10722/57525 |
| dc.identifier.volume | 357 |
| dc.language | eng |
| dc.publisher | Massachusetts Medical Society. The Journal's web site is located at http://content.nejm.org/ |
| dc.publisher.place | United States |
| dc.relation.ispartof | New England Journal of Medicine |
| dc.relation.references | References in Scopus |
| dc.rights | Creative Commons: Attribution 3.0 Hong Kong License |
| dc.rights | New England Journal of Medicine. Copyright © Massachusetts Medical Society. |
| dc.subject.mesh | Antiviral Agents - adverse effects - therapeutic use |
| dc.subject.mesh | Lamivudine - adverse effects - therapeutic use |
| dc.subject.mesh | Hepatitis B, Chronic - drug therapy |
| dc.subject.mesh | Nucleosides - adverse effects - therapeutic use |
| dc.subject.mesh | Pyrimidinones - adverse effects - therapeutic use |
| dc.title | Telbivudine versus lamivudine in patients with chronic hepatitis B |
| dc.type | Article |
Author Affiliations
- UCL
- Armed Forces Institute of Pathology
- The University of Hong Kong
- Zhejiang University
- Yonsei University College of Medicine
- Chiang Mai University
- Middlemore Hospital, Auckland
- St. Louis University
- Sutter Health
- null
- Third Military Medical University
- Chang Gung University
- University of Toronto
- Universität Freiburg im Breisgau
- Idenix Pharmaceuticals, Inc.


