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Conference Paper: Interplay between ER stress and hypoxia pathways in chondrocytes
Title | Interplay between ER stress and hypoxia pathways in chondrocytes |
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Authors | |
Issue Date | 2014 |
Citation | The 2014 Hong Kong Inter-University Biochemistry Postgraduate Symposium, Hong Kong, 14 June 2014. In Program Booklet, 2014, p. 41, poster no. 54 How to Cite? |
Abstract | Interplay between stress signals can regulate cell fate. Potential connections are emerging between hypoxia and ER homeostasis. It is well established that presence of mis-folded protein in the endoplasmic reticulum (ER) triggers the unfolded protein response (UPR) as part of the ER-stress signal for cell survival. Under low oxygen tension or hypoxia, the cell activates the hypoxic stress signal via the activation of Hif genes, and utilize components of the ER-stress pathways for cell survival and adaptation to the stress condition. However, whether activation of ER stress by accumulation of mis-folded protein has a direct relationship with activation of hypoxia and components ... |
Description | Poster Presentation - Theme 3: Development & stem cells |
Persistent Identifier | http://hdl.handle.net/10722/203869 |
DC Field | Value | Language |
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dc.contributor.author | Yao, AQ | en_US |
dc.contributor.author | Chan, WCW | en_US |
dc.contributor.author | Ng, VCW | en_US |
dc.contributor.author | Tan, Z | en_US |
dc.contributor.author | Tsang, KY | en_US |
dc.contributor.author | Cheah, KSE | en_US |
dc.contributor.author | Chan, D | en_US |
dc.date.accessioned | 2014-09-19T16:42:28Z | - |
dc.date.available | 2014-09-19T16:42:28Z | - |
dc.date.issued | 2014 | en_US |
dc.identifier.citation | The 2014 Hong Kong Inter-University Biochemistry Postgraduate Symposium, Hong Kong, 14 June 2014. In Program Booklet, 2014, p. 41, poster no. 54 | en_US |
dc.identifier.uri | http://hdl.handle.net/10722/203869 | - |
dc.description | Poster Presentation - Theme 3: Development & stem cells | - |
dc.description.abstract | Interplay between stress signals can regulate cell fate. Potential connections are emerging between hypoxia and ER homeostasis. It is well established that presence of mis-folded protein in the endoplasmic reticulum (ER) triggers the unfolded protein response (UPR) as part of the ER-stress signal for cell survival. Under low oxygen tension or hypoxia, the cell activates the hypoxic stress signal via the activation of Hif genes, and utilize components of the ER-stress pathways for cell survival and adaptation to the stress condition. However, whether activation of ER stress by accumulation of mis-folded protein has a direct relationship with activation of hypoxia and components ... | - |
dc.language | eng | en_US |
dc.relation.ispartof | Hong Kong Inter-University 2014 Biochemistry Postgraduate Symposium | en_US |
dc.title | Interplay between ER stress and hypoxia pathways in chondrocytes | en_US |
dc.type | Conference_Paper | en_US |
dc.identifier.email | Chan, WCW: cwilson@hkucc.hku.hk | en_US |
dc.identifier.email | Ng, VCW: vcwng@hkucc.hku.hk | en_US |
dc.identifier.email | Tan, Z: tanzj@hku.hk | en_US |
dc.identifier.email | Tsang, KY: kytsang@hku.hk | en_US |
dc.identifier.email | Cheah, KSE: hrmbdkc@hku.hk | en_US |
dc.identifier.email | Chan, D: chand@hku.hk | en_US |
dc.identifier.authority | Cheah, KSE=rp00342 | en_US |
dc.identifier.authority | Chan, D=rp00540 | en_US |
dc.description.nature | postprint | - |
dc.identifier.hkuros | 240398 | en_US |
dc.identifier.spage | 41, poster no. 54 | - |
dc.identifier.epage | 41, poster no. 54 | - |