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Conference Paper: Macrophages promoted the colony forming ability of putative endometrial stromal stem cells
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TitleMacrophages promoted the colony forming ability of putative endometrial stromal stem cells
 
AuthorsChan, RWS
Chan, YY
Lee, CL
Ng, EHY
Yeung, WSB
 
Issue Date2011
 
CitationThe 2011 Meeting of the Days of Molecular Medicine (DMM), Hong Kong, 10-12 November 2011. [How to Cite?]
 
AbstractWomen with endometriosis have a decreased cell-mediated immunity1 and contain more activated macrophages2. Human endometrial and endometriotic stem/progenitor cells have been identified using the clonogenic assay3, 4 ENREF 4 ENREF 4. Large colony forming units (CFUs, >4000 cells) are initiated from stem/progenitor cells and small CFUs (<4000 cells) are from transit-amplifying cells. Retrograded endometrial stem cells may have a role in the pathogenesis of endometriosis. In this study, the regulatory mechanism between macrophages and putative stem cells was examined. Endometrium (n=12)/ovarian endometrioma (n=16) were obtained from women undergoing hysterectomy and …
 
DescriptionDMM 2011 entitled: Re-engineering Regenerative Medicine
Poster Session - Blood Disorders & Stem Cell Immunology: no. 69
 
DC FieldValue
dc.contributor.authorChan, RWS
 
dc.contributor.authorChan, YY
 
dc.contributor.authorLee, CL
 
dc.contributor.authorNg, EHY
 
dc.contributor.authorYeung, WSB
 
dc.date.accessioned2012-09-20T08:26:44Z
 
dc.date.available2012-09-20T08:26:44Z
 
dc.date.issued2011
 
dc.description.abstractWomen with endometriosis have a decreased cell-mediated immunity1 and contain more activated macrophages2. Human endometrial and endometriotic stem/progenitor cells have been identified using the clonogenic assay3, 4 ENREF 4 ENREF 4. Large colony forming units (CFUs, >4000 cells) are initiated from stem/progenitor cells and small CFUs (<4000 cells) are from transit-amplifying cells. Retrograded endometrial stem cells may have a role in the pathogenesis of endometriosis. In this study, the regulatory mechanism between macrophages and putative stem cells was examined. Endometrium (n=12)/ovarian endometrioma (n=16) were obtained from women undergoing hysterectomy and …
 
dc.description.naturepostprint
 
dc.descriptionDMM 2011 entitled: Re-engineering Regenerative Medicine
 
dc.descriptionPoster Session - Blood Disorders & Stem Cell Immunology: no. 69
 
dc.identifier.citationThe 2011 Meeting of the Days of Molecular Medicine (DMM), Hong Kong, 10-12 November 2011. [How to Cite?]
 
dc.identifier.hkuros209969
 
dc.identifier.urihttp://hdl.handle.net/10722/166050
 
dc.languageeng
 
dc.relation.ispartofDay of Molecular Medicine, DMM 2011
 
dc.rightsCreative Commons: Attribution 3.0 Hong Kong License
 
dc.titleMacrophages promoted the colony forming ability of putative endometrial stromal stem cells
 
dc.typeConference_Paper
 
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<contributor.author>Chan, YY</contributor.author>
<contributor.author>Lee, CL</contributor.author>
<contributor.author>Ng, EHY</contributor.author>
<contributor.author>Yeung, WSB</contributor.author>
<date.accessioned>2012-09-20T08:26:44Z</date.accessioned>
<date.available>2012-09-20T08:26:44Z</date.available>
<date.issued>2011</date.issued>
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<description>DMM 2011 entitled: Re-engineering Regenerative Medicine</description>
<description>Poster Session - Blood Disorders &amp; Stem Cell Immunology: no. 69</description>
<description.abstract>Women with endometriosis have a decreased cell-mediated immunity1 and contain more activated macrophages2. Human endometrial and endometriotic stem/progenitor cells have been identified using the clonogenic assay3, 4 ENREF 4 ENREF 4. Large colony forming units (CFUs, &gt;4000 cells) are initiated from stem/progenitor cells and small CFUs (&lt;4000 cells) are from transit-amplifying cells. Retrograded endometrial stem cells may have a role in the pathogenesis of endometriosis. In this study, the regulatory mechanism between macrophages and putative stem cells was examined. Endometrium (n=12)/ovarian endometrioma (n=16) were obtained from women undergoing hysterectomy and &#8230;</description.abstract>
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