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Article: Anti-ganglioside antibodies were not detected in human subjects infected with or vaccinated against 2009 pandemic influenza A (H1N1) virus

TitleAnti-ganglioside antibodies were not detected in human subjects infected with or vaccinated against 2009 pandemic influenza A (H1N1) virus
Authors
Lei, TSiu, KLKok, KHChan, KHChan, EYTHung, IFNTo, KKWLi, PCKZhou, JZheng, BJYuen, KYWang, MJin, DY
KeywordsGanglioside GM1 antibody
Influenza vaccine
2009 H1N1 influenza
Animal experiment
Antibody detection
Issue Date2012
PublisherElsevier Ltd. The Journal's web site is located at http://www.elsevier.com/locate/vaccine
Citation
Vaccine, 2012, v. 30 n. 16, p. 2605-2610 How to Cite?
DOI: http://dx.doi.org/10.1016/j.vaccine.2012.02.009
AbstractRecipients of influenza A (H1N1) vaccine in 1976 had an increased risk for the neurologic disorder Guillain-Barre syndrome (GBS). Anti-ganglioside antibodies, which might be associated with the development of GBS, were previously reported to be induced in mice immunized with an H1N1 vaccine of 1976 or another influenza vaccine. In this study we analyzed anti-ganglioside antibodies in human subjects infected with or vaccinated against 2009 pandemic H1N1, including eight patients diagnosed to have post-vaccination GBS. Antibodies against GM1 or another ganglioside were not detected in any subject or in vaccinated mice. Our results did not support the induction of anti-ganglioside antibodies by influenza viruses or vaccines.
Persistent Identifierhttp://hdl.handle.net/10722/163502
ISSN
0264-410X
2021 Impact Factor: 4.169
2020 SCImago Journal Rankings: 1.585
ISI Accession Number ID
WOS:000302663200001
References
References in Scopus

 

DC FieldValueLanguage
dc.contributor.authorLei, Ten_US
dc.contributor.authorSiu, KLen_US
dc.contributor.authorKok, KHen_US
dc.contributor.authorChan, KHen_US
dc.contributor.authorChan, EYTen_US
dc.contributor.authorHung, IFNen_US
dc.contributor.authorTo, KKWen_US
dc.contributor.authorLi, PCKen_US
dc.contributor.authorZhou, Jen_US
dc.contributor.authorZheng, BJen_US
dc.contributor.authorYuen, KYen_US
dc.contributor.authorWang, Men_US
dc.contributor.authorJin, DYen_US
dc.date.accessioned2012-09-05T05:32:33Z-
dc.date.available2012-09-05T05:32:33Z-
dc.date.issued2012en_US
dc.identifier.citationVaccine, 2012, v. 30 n. 16, p. 2605-2610en_US
dc.identifier.issn0264-410Xen_US
dc.identifier.urihttp://hdl.handle.net/10722/163502-
dc.description.abstractRecipients of influenza A (H1N1) vaccine in 1976 had an increased risk for the neurologic disorder Guillain-Barre syndrome (GBS). Anti-ganglioside antibodies, which might be associated with the development of GBS, were previously reported to be induced in mice immunized with an H1N1 vaccine of 1976 or another influenza vaccine. In this study we analyzed anti-ganglioside antibodies in human subjects infected with or vaccinated against 2009 pandemic H1N1, including eight patients diagnosed to have post-vaccination GBS. Antibodies against GM1 or another ganglioside were not detected in any subject or in vaccinated mice. Our results did not support the induction of anti-ganglioside antibodies by influenza viruses or vaccines.en_US
dc.languageengen_US
dc.publisherElsevier Ltd. The Journal's web site is located at http://www.elsevier.com/locate/vaccineen_US
dc.relation.ispartofVaccineen_US
dc.subjectGanglioside GM1 antibodyen_US
dc.subjectInfluenza vaccineen_US
dc.subject2009 H1N1 influenzaen_US
dc.subjectAnimal experimenten_US
dc.subjectAntibody detection-
dc.titleAnti-ganglioside antibodies were not detected in human subjects infected with or vaccinated against 2009 pandemic influenza A (H1N1) virusen_US
dc.typeArticleen_US
dc.identifier.emailSiu, KL: sklsfx@hkucc.hku.hken_US
dc.identifier.emailKok, KH: khkok@hku.hken_US
dc.identifier.emailChan, KH: chankh2@hkucc.hku.hk-
dc.identifier.emailChan, EYT: eytchan@hkucc.hku.hk-
dc.identifier.emailHung, IFN: ivanhung@hkucc.hku.hk-
dc.identifier.emailTo, KKW: kelvinto@hkucc.hku.hk-
dc.identifier.emailZhou, J: jiezhou@hku.hk-
dc.identifier.emailZheng, BJ: bzheng@hkucc.hku.hk-
dc.identifier.emailYuen, KY: kyyuen@hkucc.hku.hk-
dc.identifier.emailJin, DY: dyjin@hku.hk-
dc.identifier.authorityHung, IFN=rp00508en_US
dc.identifier.authorityJin, DY=rp00452en_US
dc.description.naturelink_to_subscribed_fulltexten_US
dc.identifier.doi10.1016/j.vaccine.2012.02.009en_US
dc.identifier.pmid22342549-
dc.identifier.scopuseid_2-s2.0-84862809138en_US
dc.identifier.hkuros208363-
dc.relation.referenceshttp://www.scopus.com/mlt/select.url?eid=2-s2.0-84862809138&selection=ref&src=s&origin=recordpageen_US
dc.identifier.volume30en_US
dc.identifier.issue16en_US
dc.identifier.spage2605en_US
dc.identifier.epage2610en_US
dc.identifier.isiWOS:000302663200001-
dc.publisher.placeUnited Kingdomen_US
dc.identifier.scopusauthoridJin, DY=7201973614en_US
dc.identifier.scopusauthoridWang, M=53265130500en_US
dc.identifier.scopusauthoridYuen, KY=55018434500en_US
dc.identifier.scopusauthoridZheng, BJ=54935584800en_US
dc.identifier.scopusauthoridZhou, J=55262021600en_US
dc.identifier.scopusauthoridLi, PCK=55262189900en_US
dc.identifier.scopusauthoridTo, KKW=55024912100en_US
dc.identifier.scopusauthoridHung, IFN=7006103457en_US
dc.identifier.scopusauthoridChan, EYT=55262166100en_US
dc.identifier.scopusauthoridChan, KH=7406034307en_US
dc.identifier.scopusauthoridKok, KH=55261897900en_US
dc.identifier.scopusauthoridSiu, KL=55261997500en_US
dc.identifier.scopusauthoridLei, T=37000963500en_US
dc.identifier.citeulike10366680-
dc.identifier.issnl0264-410X-

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