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Conference Paper: Endothelium-selective activation of AMP-activated protein kinase alleviates diabetes-induced endothelial dysfunction by enhancing reendothelialisation and endothelial progenitor cell mobilisation in mice

TitleEndothelium-selective activation of AMP-activated protein kinase alleviates diabetes-induced endothelial dysfunction by enhancing reendothelialisation and endothelial progenitor cell mobilisation in mice
Authors
Issue Date2012
PublisherHong Kong Academy of Medicine Press. The Journal's web site is located at http://www.hkmj.org.hk
Citation
The 17th Medical Research Conference (MRC), Department of Medicine, University of Hong Kong, Hong Kong, 14 January 2012. In Hong Kong Medical Journal, 2012, v. 18 suppl. 1, p. 37, abstract no. 50 How to Cite?
AbstractINTRODUCTION: Vascular dysfunction is commonly observed in diabetic patients. Endothelial injury can be repaired in part by endothelial progenitor cells (EPCs). In diabetes, impaired functionality and reduced number of EPCs are reported. AMP-activated kinase (AMPK) is a well-known target of several anti-diabetic and cardiovascular drugs. The objective of this study was to examine whether the activation of AMPK in endothelium alone is sufficient to prevent diabetes-induced impairment in vascular function, possibly by improving EPC function and endothelial repair using a tissue-specific transgenic mouse model. METHODS: Transgenic mice with endothelium-selective expression of a …
DescriptionThis journal supplement contains abstracts from the 17th MRC; Dept. of Medicine, The University of Hong Kong, Queen Mary Hospital, Hong Kong
Topics: Basic science, translational and clinical research
Oral Presentation
Persistent Identifierhttp://hdl.handle.net/10722/146021
ISSN
2015 Impact Factor: 0.887
2015 SCImago Journal Rankings: 0.279

 

DC FieldValueLanguage
dc.contributor.authorLi, FYLen_US
dc.contributor.authorWang, Yen_US
dc.contributor.authorLam, KSLen_US
dc.contributor.authorXu, Aen_US
dc.date.accessioned2012-03-27T09:06:17Z-
dc.date.available2012-03-27T09:06:17Z-
dc.date.issued2012en_US
dc.identifier.citationThe 17th Medical Research Conference (MRC), Department of Medicine, University of Hong Kong, Hong Kong, 14 January 2012. In Hong Kong Medical Journal, 2012, v. 18 suppl. 1, p. 37, abstract no. 50en_US
dc.identifier.issn1024-2708-
dc.identifier.urihttp://hdl.handle.net/10722/146021-
dc.descriptionThis journal supplement contains abstracts from the 17th MRC; Dept. of Medicine, The University of Hong Kong, Queen Mary Hospital, Hong Kong-
dc.descriptionTopics: Basic science, translational and clinical research-
dc.descriptionOral Presentation-
dc.description.abstractINTRODUCTION: Vascular dysfunction is commonly observed in diabetic patients. Endothelial injury can be repaired in part by endothelial progenitor cells (EPCs). In diabetes, impaired functionality and reduced number of EPCs are reported. AMP-activated kinase (AMPK) is a well-known target of several anti-diabetic and cardiovascular drugs. The objective of this study was to examine whether the activation of AMPK in endothelium alone is sufficient to prevent diabetes-induced impairment in vascular function, possibly by improving EPC function and endothelial repair using a tissue-specific transgenic mouse model. METHODS: Transgenic mice with endothelium-selective expression of a …-
dc.languageengen_US
dc.publisherHong Kong Academy of Medicine Press. The Journal's web site is located at http://www.hkmj.org.hk-
dc.relation.ispartofHong Kong Medical Journalen_US
dc.rightsHong Kong Medical Journal. Copyright © Hong Kong Academy of Medicine Press.-
dc.rightsCreative Commons: Attribution 3.0 Hong Kong License-
dc.titleEndothelium-selective activation of AMP-activated protein kinase alleviates diabetes-induced endothelial dysfunction by enhancing reendothelialisation and endothelial progenitor cell mobilisation in miceen_US
dc.typeConference_Paperen_US
dc.identifier.emailLi, FYL: francois.li@hku.hken_US
dc.identifier.emailWang, Y: yiqunw@hku.hken_US
dc.identifier.emailLam, KSL: ksllam@hku.hken_US
dc.identifier.emailXu, A: amxu@hkucc.hku.hken_US
dc.identifier.authorityLam, KSL=rp00343en_US
dc.identifier.authorityXu, A=rp00485en_US
dc.description.naturepublished_or_final_version-
dc.identifier.hkuros198910en_US
dc.identifier.volume18-
dc.identifier.issuesuppl. 1-
dc.identifier.spage37-
dc.identifier.epage37-
dc.publisher.placeHong Kong-
dc.description.otherThe 17th Medical Research Conference (MRC), Department of Medicine, University of Hong Kong, Hong Kong, 14 January 2012. In Hong Kong Medical Journal, 2012, v. 18 suppl. 1, p. 37, abstract no. 50-

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