Conference Paper: In search of shared deficits underlying SLI and dyslexia in Chinese

TitleIn search of shared deficits underlying SLI and dyslexia in Chinese
Authors
KeywordsSLI
Dyslexia
Chinese
Language disorders
Issue Date2010
PublisherUniversity of Wisconsin-Madion.
Citation
The 31st Annual Symposium on Research in Child Language Disorders (SRCLD 2010), University of Wisconsin-Madion, WI., 3-5 June 2010. How to Cite?
AbstractChildren with Specific Language Impairment (SLI) are at heightened risk for dyslexia. Some studies have found SLI children with concomitant dyslexia poorer at discriminating fine-grained auditory stimuli; in separate samples of SLI and reading-impaired children, others have found poorer quality of phonological representations. We asked whether auditory frequency discrimination (FD), frequency modulation (FM) detection and speech gating, putative measures of these skills, could identify SLI children ‘at-risk’ for dyslexia. Cantonese-speaking children aged 5-6 years were classified as SLI+ (n=15), SLI- (n=22), NL+ (n=14), or NL- (n=18) (+ and – denote at-risk status for dyslexia; NL denotes Normal Language). Group differences were evident on FM: SLI+ and SLI- had worse thresholds than NL+ and NL-, which did not differ from each other. SLI- children performed significantly less well on speech gating than the NL+ group, but did not differ from NL- or SLI+. These results suggest that poor FM and poor phonological representations might characterize SLI but not ‘at-risk’ status for dyslexia in Cantonese-Chinese children.
DescriptionPoster Presentation
Persistent Identifierhttp://hdl.handle.net/10722/127168

 

DC FieldValueLanguage
dc.contributor.authorKidd, JCen_HK
dc.contributor.authorWong, AMYen_HK
dc.contributor.authorHo, CSHen_HK
dc.contributor.authorAu, TKFen_HK
dc.contributor.authorLam, CCCen_HK
dc.contributor.authorYip, LPWen_HK
dc.contributor.authorLam, FWFen_HK
dc.date.accessioned2010-10-31T13:10:05Z-
dc.date.available2010-10-31T13:10:05Z-
dc.date.issued2010en_HK
dc.identifier.citationThe 31st Annual Symposium on Research in Child Language Disorders (SRCLD 2010), University of Wisconsin-Madion, WI., 3-5 June 2010.en_HK
dc.identifier.urihttp://hdl.handle.net/10722/127168-
dc.descriptionPoster Presentation-
dc.description.abstractChildren with Specific Language Impairment (SLI) are at heightened risk for dyslexia. Some studies have found SLI children with concomitant dyslexia poorer at discriminating fine-grained auditory stimuli; in separate samples of SLI and reading-impaired children, others have found poorer quality of phonological representations. We asked whether auditory frequency discrimination (FD), frequency modulation (FM) detection and speech gating, putative measures of these skills, could identify SLI children ‘at-risk’ for dyslexia. Cantonese-speaking children aged 5-6 years were classified as SLI+ (n=15), SLI- (n=22), NL+ (n=14), or NL- (n=18) (+ and – denote at-risk status for dyslexia; NL denotes Normal Language). Group differences were evident on FM: SLI+ and SLI- had worse thresholds than NL+ and NL-, which did not differ from each other. SLI- children performed significantly less well on speech gating than the NL+ group, but did not differ from NL- or SLI+. These results suggest that poor FM and poor phonological representations might characterize SLI but not ‘at-risk’ status for dyslexia in Cantonese-Chinese children.-
dc.languageengen_HK
dc.publisherUniversity of Wisconsin-Madion.-
dc.relation.ispartofAnnual Symposium on Research in Child Language Disorders, SRCLD 2010-
dc.subjectSLI-
dc.subjectDyslexia-
dc.subjectChinese-
dc.subjectLanguage disorders-
dc.titleIn search of shared deficits underlying SLI and dyslexia in Chineseen_HK
dc.typeConference_Paperen_HK
dc.identifier.emailWong, AMY: amywong@hku.hken_HK
dc.identifier.emailHo, CSH: shhoc@hkucc.hku.hken_HK
dc.identifier.emailAu, TKF: terryau@hkucc.hku.hken_HK
dc.identifier.authorityWong, AMY=rp00973en_HK
dc.identifier.authorityHo, CSH=rp00631en_HK
dc.identifier.authorityAu, TKF=rp00580en_HK
dc.description.naturelink_to_OA_fulltext-
dc.identifier.hkuros175531en_HK
dc.publisher.placeUnited States-

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