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Article: Impact of intrauterine growth retardation on the gastrointestinal tract and the pancreas in newborn pigs

TitleImpact of intrauterine growth retardation on the gastrointestinal tract and the pancreas in newborn pigs
Authors
KeywordsGrowth retardation
Intestine
Newborn
Pancreas
Pig
Stomach
Issue Date1994
PublisherLippincott Williams & Wilkins. The Journal's web site is located at http://www.jpgn.org
Citation
Journal Of Pediatric Gastroenterology And Nutrition, 1994, v. 18 n. 2, p. 231-240 How to Cite?
AbstractEffects of intrauterine growth retardation (IUGR) on the gastrointestinal tract and the pancreas were examined using a model of piglets with naturally occurring IUGR. IUGR was associated with significant reductions in gastrointestinal and pancreatic tissue weights. The reduction in tissue weight in the gastrointestinal tract was due to lowered cell numbers and was proportionate to the body as a whole. In contrast to the gastrointestinal tract, the reduction in the pancreas was due to both lowered cell numbers and smaller cell size and was disproportionately greater than the body as a whole. IUGR was associated with precocious occurrence of maltase and sucrase activities in the small-intestinal mucosa. The exocrine cells of the pancreas in the animals with IUGR appeared immature when compared with those in normal piglets. Further studies are required to examine the postnatal effects of IUGR on the gastrointestinal tract, the pancreas, and somatic development.
Persistent Identifierhttp://hdl.handle.net/10722/84665
ISSN
2015 Impact Factor: 2.4
2015 SCImago Journal Rankings: 1.227
ISI Accession Number ID

 

DC FieldValueLanguage
dc.contributor.authorXu, RJen_HK
dc.contributor.authorMellor, DJen_HK
dc.contributor.authorBirtles, MJen_HK
dc.contributor.authorReynolds, GWen_HK
dc.contributor.authorSimpson, HVen_HK
dc.date.accessioned2010-09-06T08:55:41Z-
dc.date.available2010-09-06T08:55:41Z-
dc.date.issued1994en_HK
dc.identifier.citationJournal Of Pediatric Gastroenterology And Nutrition, 1994, v. 18 n. 2, p. 231-240en_HK
dc.identifier.issn0277-2116en_HK
dc.identifier.urihttp://hdl.handle.net/10722/84665-
dc.description.abstractEffects of intrauterine growth retardation (IUGR) on the gastrointestinal tract and the pancreas were examined using a model of piglets with naturally occurring IUGR. IUGR was associated with significant reductions in gastrointestinal and pancreatic tissue weights. The reduction in tissue weight in the gastrointestinal tract was due to lowered cell numbers and was proportionate to the body as a whole. In contrast to the gastrointestinal tract, the reduction in the pancreas was due to both lowered cell numbers and smaller cell size and was disproportionately greater than the body as a whole. IUGR was associated with precocious occurrence of maltase and sucrase activities in the small-intestinal mucosa. The exocrine cells of the pancreas in the animals with IUGR appeared immature when compared with those in normal piglets. Further studies are required to examine the postnatal effects of IUGR on the gastrointestinal tract, the pancreas, and somatic development.en_HK
dc.languageengen_HK
dc.publisherLippincott Williams & Wilkins. The Journal's web site is located at http://www.jpgn.orgen_HK
dc.relation.ispartofJournal of Pediatric Gastroenterology and Nutritionen_HK
dc.rightsJournal of Pediatric Gastroenterology and Nutrition. Copyright © Lippincott Williams & Wilkins.en_HK
dc.subjectGrowth retardationen_HK
dc.subjectIntestineen_HK
dc.subjectNewbornen_HK
dc.subjectPancreasen_HK
dc.subjectPigen_HK
dc.subjectStomachen_HK
dc.titleImpact of intrauterine growth retardation on the gastrointestinal tract and the pancreas in newborn pigsen_HK
dc.typeArticleen_HK
dc.identifier.openurlhttp://library.hku.hk:4550/resserv?sid=HKU:IR&issn=0277-2116&volume=18&spage=231&epage=240&date=1994&atitle=Impact+of+intrauterine+growth+retardation+on+the+gastrointestinal+tract+and+the+pancreas+in+newborn+pigsen_HK
dc.identifier.emailXu, RJ: xuruojun@hkucc.hku.hken_HK
dc.identifier.authorityXu, RJ=rp00820en_HK
dc.description.naturelink_to_subscribed_fulltext-
dc.identifier.pmid8014773-
dc.identifier.scopuseid_2-s2.0-0028107621en_HK
dc.identifier.hkuros5794en_HK
dc.identifier.volume18en_HK
dc.identifier.issue2en_HK
dc.identifier.spage231en_HK
dc.identifier.epage240en_HK
dc.identifier.isiWOS:A1994MV01800018-
dc.publisher.placeUnited Statesen_HK
dc.identifier.scopusauthoridXu, RJ=7402813973en_HK
dc.identifier.scopusauthoridMellor, DJ=7103269442en_HK
dc.identifier.scopusauthoridBirtles, MJ=6603615393en_HK
dc.identifier.scopusauthoridReynolds, GW=7201487841en_HK
dc.identifier.scopusauthoridSimpson, HV=7202391355en_HK

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