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Article: Potential role for platelet apheresis for post-liver transplant thrombocytosis complicating portal vein thrombosis

TitlePotential role for platelet apheresis for post-liver transplant thrombocytosis complicating portal vein thrombosis
Authors
KeywordsBleeding
Liver transplantation
Platelet apheresis
Portal vein thrombosis
Thrombocytosis
Issue Date2004
PublisherJohn Wiley & Sons, Inc. The Journal's web site is located at http://www3.interscience.wiley.com/cgi-bin/jhome/35674
Citation
Journal Of Clinical Apheresis, 2004, v. 19 n. 4, p. 192-196 How to Cite?
AbstractExcept for patients with underlying myeloproliferative diseases (MPD), thrombocytosis is rarely encountered in cirrhotic patients after liver transplantation. Although the long-term control of primary thrombocytosis is important for the prevention of graft thrombosis in MPD patients, the threshold for intervention and best mode for the control of persistent reactive thrombocytosis after liver transplantation is unclear. We present two patients with extreme reactive thrombocytosis (over 1,000 × 10 9/l) due to intra-abdominal sepsis after liver transplantation. Furthermore, both patients suffered from bleeding problems as well as ongoing venous thrombosis of the graft. Rapid control of the platelet count was achieved using platelet apheresis. The use of cell separation procedures may be a relatively rapid, reversible, and reasonably safe way to control platelet counts peri-operatively in liver transplant recipients. © 2004 Wiley-Liss, Inc.
Persistent Identifierhttp://hdl.handle.net/10722/84529
ISSN
2015 Impact Factor: 2.432
2015 SCImago Journal Rankings: 0.782
ISI Accession Number ID
References

 

DC FieldValueLanguage
dc.contributor.authorAu, WYen_HK
dc.contributor.authorLiu, CLen_HK
dc.contributor.authorLo, CMen_HK
dc.contributor.authorFan, STen_HK
dc.contributor.authorMa, SKen_HK
dc.date.accessioned2010-09-06T08:54:01Z-
dc.date.available2010-09-06T08:54:01Z-
dc.date.issued2004en_HK
dc.identifier.citationJournal Of Clinical Apheresis, 2004, v. 19 n. 4, p. 192-196en_HK
dc.identifier.issn0733-2459en_HK
dc.identifier.urihttp://hdl.handle.net/10722/84529-
dc.description.abstractExcept for patients with underlying myeloproliferative diseases (MPD), thrombocytosis is rarely encountered in cirrhotic patients after liver transplantation. Although the long-term control of primary thrombocytosis is important for the prevention of graft thrombosis in MPD patients, the threshold for intervention and best mode for the control of persistent reactive thrombocytosis after liver transplantation is unclear. We present two patients with extreme reactive thrombocytosis (over 1,000 × 10 9/l) due to intra-abdominal sepsis after liver transplantation. Furthermore, both patients suffered from bleeding problems as well as ongoing venous thrombosis of the graft. Rapid control of the platelet count was achieved using platelet apheresis. The use of cell separation procedures may be a relatively rapid, reversible, and reasonably safe way to control platelet counts peri-operatively in liver transplant recipients. © 2004 Wiley-Liss, Inc.en_HK
dc.languageengen_HK
dc.publisherJohn Wiley & Sons, Inc. The Journal's web site is located at http://www3.interscience.wiley.com/cgi-bin/jhome/35674en_HK
dc.relation.ispartofJournal of Clinical Apheresisen_HK
dc.rightsJournal of Clinical Apheresis. Copyright © John Wiley & Sons, Inc.en_HK
dc.subjectBleedingen_HK
dc.subjectLiver transplantationen_HK
dc.subjectPlatelet apheresisen_HK
dc.subjectPortal vein thrombosisen_HK
dc.subjectThrombocytosisen_HK
dc.titlePotential role for platelet apheresis for post-liver transplant thrombocytosis complicating portal vein thrombosisen_HK
dc.typeArticleen_HK
dc.identifier.openurlhttp://library.hku.hk:4550/resserv?sid=HKU:IR&issn=0733-2459&volume=19&issue=4&spage=192&epage=196&date=2004&atitle=Potential+role+for+platelet+apheresis+for+post-liver+transplant+thrombocytosis+complicating+portal+vein+thrombosisen_HK
dc.identifier.emailLo, CM: chungmlo@hkucc.hku.hken_HK
dc.identifier.emailFan, ST: stfan@hku.hken_HK
dc.identifier.emailMa, SK: sma@pathology.hku.hken_HK
dc.identifier.authorityLo, CM=rp00412en_HK
dc.identifier.authorityFan, ST=rp00355en_HK
dc.identifier.authorityMa, SK=rp00506en_HK
dc.description.naturelink_to_subscribed_fulltext-
dc.identifier.doi10.1002/jca.20028en_HK
dc.identifier.pmid15597348-
dc.identifier.scopuseid_2-s2.0-12844257445en_HK
dc.identifier.hkuros96813en_HK
dc.relation.referenceshttp://www.scopus.com/mlt/select.url?eid=2-s2.0-12844257445&selection=ref&src=s&origin=recordpageen_HK
dc.identifier.volume19en_HK
dc.identifier.issue4en_HK
dc.identifier.spage192en_HK
dc.identifier.epage196en_HK
dc.identifier.isiWOS:000225891300006-
dc.publisher.placeUnited Statesen_HK
dc.identifier.scopusauthoridAu, WY=7202383089en_HK
dc.identifier.scopusauthoridLiu, CL=7409789712en_HK
dc.identifier.scopusauthoridLo, CM=7401771672en_HK
dc.identifier.scopusauthoridFan, ST=7402678224en_HK
dc.identifier.scopusauthoridMa, SK=16444895800en_HK

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