Article: A genome-wide association study identifies colorectal cancer susceptibility loci on chromosomes 10p14 and 8q23.3
| Title | A genome-wide association study identifies colorectal cancer susceptibility loci on chromosomes 10p14 and 8q23.3 |
|---|---|
| Authors | Tomlinson, IPM16 Webb, E4 CarvajalCarmona, L16 Broderick, P4 Howarth, K16 Pittman, AM4 Spain, S16 Lubbe, S4 Walther, A16 Sullivan, K4 Jaeger, E16 Fielding, S4 Rowan, A16 Vijayakrishnan, J4 Domingo, E16 Chandler, I4 Kemp, Z16 Qureshi, M4 Farrington, SM21 Tenesa, A21 Prendergast, JGD21 Barnetson, RA21 Penegar, S4 Barclay, E16 Wood, W4 Martin, L12 16 26 Gorman, M16 Thomas, H25 Peto, J4 9 Bishop, DT17 Gray, R12 Maher, ER12 Lucassen, A3 Kerr, D18 Evans, DGR26 Schafmayer, C14 Buch, S14 Völzke, H20 Hampe, J14 Schreiber, S14 John, U20 Koessler, T15 Pharoah, P15 Van Wezel, T23 Morreau, H23 Wijnen, JT23 Hopper, JL22 Southey, MC13 Giles, GG2 22 Severi, G2 CastellvíBel, S1 RuizPonte, C5 Carracedo, A5 Castells, A1 Försti, A11 27 Hemminki, K11 27 Vodicka, P24 Naccarati, A24 Lipton, L19 Ho, JWC6 Cheng, KK6 Sham, PC6 Luk, J6 Agúndez, JAG7 Ladero, JM8 De La Hoya, M8 Caldés, T8 Niittymäki, I10 Tuupanen, S10 Karhu, A10 Aaltonen, L10 Cazier, JB16 Campbell, H21 Dunlop, MG21 Houlston, RS4 |
| Issue Date | 2008 |
| Publisher | Nature Publishing Group. The Journal's web site is located at http://www.genetics.nature.com |
| Citation | Nature Genetics, 2008, v. 40 n. 5, p. 623-630 [How to Cite?] DOI: http://dx.doi.org/10.1038/ng.111 |
| Abstract | To identify colorectal cancer (CRC) susceptibility alleles, we conducted a genome-wide association study. In phase 1, we genotyped 550,163 tagSNPs in 940 familial colorectal tumor cases (627 CRC, 313 high-risk adenoma) and 965 controls. In phase 2, we genotyped 42,708 selected SNPs in 2,873 CRC cases and 2,871 controls. In phase 3, we evaluated 11 SNPs showing association at P < 10 -4 in a joint analysis of phases 1 and 2 in 4,287 CRC cases and 3,743 controls. Two SNPs were taken forward to phase 4 genotyping (10,731 CRC cases and 10,961 controls from eight centers). In addition to the previously reported 8q24, 15q13 and 18q21 CRC risk loci, we identified two previously unreported associations: rs10795668, located at 10p14 (P = 2.5 × 10 -13 overall; P = 6.9 × 10 -12 replication), and rs16892766, at 8q23.3 (P = 3.3 × 10 -18 overall; P = 9.6 × 10 -17 replication), which tags a plausible causative gene, EIF3H. These data provide further evidence for the 'common-disease common-variant' model of CRC predisposition. © 2008 Nature Publishing Group. |
| ISSN | 1061-4036 2011 Impact Factor: 35.532 2011 SCImago Journal Rankings: 8.923 |
| DOI | http://dx.doi.org/10.1038/ng.111 |
| ISI Accession Number ID | WOS:000255366700031 |
| References | References in Scopus |
| dc.contributor.author | Tomlinson, IPM |
|---|---|
| dc.contributor.author | Webb, E |
| dc.contributor.author | CarvajalCarmona, L |
| dc.contributor.author | Broderick, P |
| dc.contributor.author | Howarth, K |
| dc.contributor.author | Pittman, AM |
| dc.contributor.author | Spain, S |
| dc.contributor.author | Lubbe, S |
| dc.contributor.author | Walther, A |
| dc.contributor.author | Sullivan, K |
| dc.contributor.author | Jaeger, E |
| dc.contributor.author | Fielding, S |
| dc.contributor.author | Rowan, A |
| dc.contributor.author | Vijayakrishnan, J |
| dc.contributor.author | Domingo, E |
| dc.contributor.author | Chandler, I |
| dc.contributor.author | Kemp, Z |
| dc.contributor.author | Qureshi, M |
| dc.contributor.author | Farrington, SM |
| dc.contributor.author | Tenesa, A |
| dc.contributor.author | Prendergast, JGD |
| dc.contributor.author | Barnetson, RA |
| dc.contributor.author | Penegar, S |
| dc.contributor.author | Barclay, E |
| dc.contributor.author | Wood, W |
| dc.contributor.author | Martin, L |
| dc.contributor.author | Gorman, M |
| dc.contributor.author | Thomas, H |
| dc.contributor.author | Peto, J |
| dc.contributor.author | Bishop, DT |
| dc.contributor.author | Gray, R |
| dc.contributor.author | Maher, ER |
| dc.contributor.author | Lucassen, A |
| dc.contributor.author | Kerr, D |
| dc.contributor.author | Evans, DGR |
| dc.contributor.author | Schafmayer, C |
| dc.contributor.author | Buch, S |
| dc.contributor.author | Völzke, H |
| dc.contributor.author | Hampe, J |
| dc.contributor.author | Schreiber, S |
| dc.contributor.author | John, U |
| dc.contributor.author | Koessler, T |
| dc.contributor.author | Pharoah, P |
| dc.contributor.author | Van Wezel, T |
| dc.contributor.author | Morreau, H |
| dc.contributor.author | Wijnen, JT |
| dc.contributor.author | Hopper, JL |
| dc.contributor.author | Southey, MC |
| dc.contributor.author | Giles, GG |
| dc.contributor.author | Severi, G |
| dc.contributor.author | CastellvíBel, S |
| dc.contributor.author | RuizPonte, C |
| dc.contributor.author | Carracedo, A |
| dc.contributor.author | Castells, A |
| dc.contributor.author | Försti, A |
| dc.contributor.author | Hemminki, K |
| dc.contributor.author | Vodicka, P |
| dc.contributor.author | Naccarati, A |
| dc.contributor.author | Lipton, L |
| dc.contributor.author | Ho, JWC |
| dc.contributor.author | Cheng, KK |
| dc.contributor.author | Sham, PC |
| dc.contributor.author | Luk, J |
| dc.contributor.author | Agúndez, JAG |
| dc.contributor.author | Ladero, JM |
| dc.contributor.author | De La Hoya, M |
| dc.contributor.author | Caldés, T |
| dc.contributor.author | Niittymäki, I |
| dc.contributor.author | Tuupanen, S |
| dc.contributor.author | Karhu, A |
| dc.contributor.author | Aaltonen, L |
| dc.contributor.author | Cazier, JB |
| dc.contributor.author | Campbell, H |
| dc.contributor.author | Dunlop, MG |
| dc.contributor.author | Houlston, RS |
| dc.date.accessioned | 2010-09-06T08:20:38Z |
| dc.date.available | 2010-09-06T08:20:38Z |
| dc.date.issued | 2008 |
| dc.description.abstract | To identify colorectal cancer (CRC) susceptibility alleles, we conducted a genome-wide association study. In phase 1, we genotyped 550,163 tagSNPs in 940 familial colorectal tumor cases (627 CRC, 313 high-risk adenoma) and 965 controls. In phase 2, we genotyped 42,708 selected SNPs in 2,873 CRC cases and 2,871 controls. In phase 3, we evaluated 11 SNPs showing association at P < 10 -4 in a joint analysis of phases 1 and 2 in 4,287 CRC cases and 3,743 controls. Two SNPs were taken forward to phase 4 genotyping (10,731 CRC cases and 10,961 controls from eight centers). In addition to the previously reported 8q24, 15q13 and 18q21 CRC risk loci, we identified two previously unreported associations: rs10795668, located at 10p14 (P = 2.5 × 10 -13 overall; P = 6.9 × 10 -12 replication), and rs16892766, at 8q23.3 (P = 3.3 × 10 -18 overall; P = 9.6 × 10 -17 replication), which tags a plausible causative gene, EIF3H. These data provide further evidence for the 'common-disease common-variant' model of CRC predisposition. © 2008 Nature Publishing Group. |
| dc.description.nature | link_to_subscribed_fulltext |
| dc.identifier.citation | Nature Genetics, 2008, v. 40 n. 5, p. 623-630 [How to Cite?] DOI: http://dx.doi.org/10.1038/ng.111 |
| dc.identifier.citeulike | 2731132 |
| dc.identifier.doi | http://dx.doi.org/10.1038/ng.111 |
| dc.identifier.epage | 630 |
| dc.identifier.hkuros | 161726 |
| dc.identifier.isi | WOS:000255366700031 |
| dc.identifier.issn | 1061-4036 2011 Impact Factor: 35.532 2011 SCImago Journal Rankings: 8.923 |
| dc.identifier.issue | 5 |
| dc.identifier.openurl | ![]() |
| dc.identifier.pmid | 18372905 |
| dc.identifier.scopus | eid_2-s2.0-42649136554 |
| dc.identifier.spage | 623 |
| dc.identifier.uri | http://hdl.handle.net/10722/81673 |
| dc.identifier.volume | 40 |
| dc.language | eng |
| dc.publisher | Nature Publishing Group. The Journal's web site is located at http://www.genetics.nature.com |
| dc.publisher.place | United States |
| dc.relation.ispartof | Nature Genetics |
| dc.relation.references | References in Scopus |
| dc.title | A genome-wide association study identifies colorectal cancer susceptibility loci on chromosomes 10p14 and 8q23.3 |
| dc.type | Article |
Author Affiliations
- Hospital Clinic Barcelona
- Cancer Council Victoria
- University of Southampton
- Institute of Cancer Research London
- Complejo Hospitalario Universitario de Santiago
- The University of Hong Kong
- Universidad de Extremadura
- Hospital Clinico San Carlos de Madrid
- London School of Hygiene & Tropical Medicine
- University of Helsinki Faculty of Medicine
- Karolinska Institutet
- University of Birmingham
- University of Melbourne School of Medicine Dentistry and Health Sciences
- Universitätsklinikum Schleswig-Holstein Campus Kiel
- University of Cambridge
- Cancer Research UK
- University of Leeds
- University of Oxford
- Western Hospital, Footscray
- Ernst-Moritz-Arndt-Universität Greifswald
- University of Edinburgh
- University of Melbourne
- Leiden University Medical Center - LUMC
- Institute of Experimental Medicine of the Academy of Sciences of the Czech Republic
- St. Mark's Hospital and Academic Institute
- St Mary's Hospital London
- German Cancer Research Center


