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Article: Immunohistochemical observation on luteinizing hormone in rat testes before and after testicular capsulotomy

TitleImmunohistochemical observation on luteinizing hormone in rat testes before and after testicular capsulotomy
Authors
KeywordsImmunohistochemistry
Luteinizing hormone
Testicular capsulotomy
Testis
Issue Date2001
PublisherBlackwell Publishing, Inc. The Journal's web site is located at http://www.blackwellpublishing.com/journal.asp?ref=1008-682X&site=1
Citation
Asian Journal Of Andrology, 2001, v. 3 n. 3, p. 227-230 How to Cite?
AbstractAim: In the testicular capsulotomized rats, although there was a significant increase in the luteinizing hormone (LH) levels, the secretion of testosterone remained low. In order to clarify the mechanisms of this phenomenon, the binding of endogenous LH to the testes were observed before and after testicular capsulotomy. Methods: Peroxidase-antiperoxidase (PAP) method was used to detect the binding of LH to the testes in rats. Results: An intense positive staining of LH was found in the Leydig cells of both the normal and sham-operated control testes. However, at 40 d after operation, the LH immunoreactivity was decreased in the Leydig cells of the capsulotomized testis. By d 60, only very weak positive staining could be observed in these cells. Conclusion: A progressive reduction of endogenous LH binding to the testis occurred in the capsulotomized rat.
Persistent Identifierhttp://hdl.handle.net/10722/81278
ISSN
2015 Impact Factor: 2.644
2015 SCImago Journal Rankings: 0.879
ISI Accession Number ID
References

 

DC FieldValueLanguage
dc.contributor.authorOin, DNen_HK
dc.contributor.authorLung, MAen_HK
dc.date.accessioned2010-09-06T08:15:51Z-
dc.date.available2010-09-06T08:15:51Z-
dc.date.issued2001en_HK
dc.identifier.citationAsian Journal Of Andrology, 2001, v. 3 n. 3, p. 227-230en_HK
dc.identifier.issn1008-682Xen_HK
dc.identifier.urihttp://hdl.handle.net/10722/81278-
dc.description.abstractAim: In the testicular capsulotomized rats, although there was a significant increase in the luteinizing hormone (LH) levels, the secretion of testosterone remained low. In order to clarify the mechanisms of this phenomenon, the binding of endogenous LH to the testes were observed before and after testicular capsulotomy. Methods: Peroxidase-antiperoxidase (PAP) method was used to detect the binding of LH to the testes in rats. Results: An intense positive staining of LH was found in the Leydig cells of both the normal and sham-operated control testes. However, at 40 d after operation, the LH immunoreactivity was decreased in the Leydig cells of the capsulotomized testis. By d 60, only very weak positive staining could be observed in these cells. Conclusion: A progressive reduction of endogenous LH binding to the testis occurred in the capsulotomized rat.en_HK
dc.languageengen_HK
dc.publisherBlackwell Publishing, Inc. The Journal's web site is located at http://www.blackwellpublishing.com/journal.asp?ref=1008-682X&site=1en_HK
dc.relation.ispartofAsian Journal of Andrologyen_HK
dc.subjectImmunohistochemistryen_HK
dc.subjectLuteinizing hormoneen_HK
dc.subjectTesticular capsulotomyen_HK
dc.subjectTestisen_HK
dc.titleImmunohistochemical observation on luteinizing hormone in rat testes before and after testicular capsulotomyen_HK
dc.typeArticleen_HK
dc.identifier.openurlhttp://library.hku.hk:4550/resserv?sid=HKU:IR&issn=1008-682X&volume=3&spage=227&epage=30&date=2001&atitle=Immunohistochemical+observation+on+luteinizing+hormone+in+rat+testes+before+and+after+testicular+capsulotomyen_HK
dc.identifier.emailLung, MA: makylung@hkucc.hku.hken_HK
dc.identifier.authorityLung, MA=rp00319en_HK
dc.description.naturelink_to_subscribed_fulltext-
dc.identifier.pmid11561195en_HK
dc.identifier.scopuseid_2-s2.0-0034784946en_HK
dc.identifier.hkuros71852en_HK
dc.relation.referenceshttp://www.scopus.com/mlt/select.url?eid=2-s2.0-0034784946&selection=ref&src=s&origin=recordpageen_HK
dc.identifier.volume3en_HK
dc.identifier.issue3en_HK
dc.identifier.spage227en_HK
dc.identifier.epage230en_HK
dc.identifier.isiWOS:000171402900012-
dc.publisher.placeUnited Statesen_HK
dc.identifier.scopusauthoridOin, DN=22948680400en_HK
dc.identifier.scopusauthoridLung, MA=7006411781en_HK

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