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Article: Viral nephropathy

TitleViral nephropathy
Authors
KeywordsGlomerulonephritis
Hepatitis B
Hepatitis C
HIV-associated nephropathy
Viral infection
Issue Date2006
PublisherNature Publishing Group.
Citation
Nature Clinical Practice Nephrology, 2006, v. 2 n. 5, p. 254-262 How to Cite?
AbstractViral infections can cause many glomerular diseases. The diagnostic criteria for virus-related nephropathy include detailed clinical and laboratory data, and tissue molecular analysis. Several mechanisms are involved in the pathogenesis of virus-related nephropathy, including tropism of the virus in the kidney, induction of abnormal immune complexes, direct cytopathogenic effects, and multiorgan failure. Hepatitis B virus is associated with membranous nephropathy and mesangiocapillary glomerulonephritis in endemic areas. Hepatitis C virus causes various forms of glomerulonephritis, including cryoglobulinemia-mediated glomerulonephritis. Infection with HIV is associated with a collapsing focal segmental glomerulosclerosis, a distinctive disease that affects mainly Africans and African Americans. In the course of HIV infection, other types of immune complex glomerulonephritis can occur, most frequently in whites. Recent reports indicate a role for parvovirus B19 in 'idiopathic' collapsing focal segmental glomerulosclerosis. Both hantaviruses, and coronaviruses associated with severe acute respiratory syndrome, can lead to acute renal failure. Renal biopsy followed by appropriate serological and molecular testing is essential for defining virus-related glomerular lesions and guiding prognostic and therapeutic evaluation. ©2006 Nature Publishing Group.
Persistent Identifierhttp://hdl.handle.net/10722/78078
ISSN
2011 Impact Factor: 6.083
ISI Accession Number ID
References

 

DC FieldValueLanguage
dc.contributor.authorLai, ASHen_HK
dc.contributor.authorLai, KNen_HK
dc.date.accessioned2010-09-06T07:38:55Z-
dc.date.available2010-09-06T07:38:55Z-
dc.date.issued2006en_HK
dc.identifier.citationNature Clinical Practice Nephrology, 2006, v. 2 n. 5, p. 254-262en_HK
dc.identifier.issn1745-8323en_HK
dc.identifier.urihttp://hdl.handle.net/10722/78078-
dc.description.abstractViral infections can cause many glomerular diseases. The diagnostic criteria for virus-related nephropathy include detailed clinical and laboratory data, and tissue molecular analysis. Several mechanisms are involved in the pathogenesis of virus-related nephropathy, including tropism of the virus in the kidney, induction of abnormal immune complexes, direct cytopathogenic effects, and multiorgan failure. Hepatitis B virus is associated with membranous nephropathy and mesangiocapillary glomerulonephritis in endemic areas. Hepatitis C virus causes various forms of glomerulonephritis, including cryoglobulinemia-mediated glomerulonephritis. Infection with HIV is associated with a collapsing focal segmental glomerulosclerosis, a distinctive disease that affects mainly Africans and African Americans. In the course of HIV infection, other types of immune complex glomerulonephritis can occur, most frequently in whites. Recent reports indicate a role for parvovirus B19 in 'idiopathic' collapsing focal segmental glomerulosclerosis. Both hantaviruses, and coronaviruses associated with severe acute respiratory syndrome, can lead to acute renal failure. Renal biopsy followed by appropriate serological and molecular testing is essential for defining virus-related glomerular lesions and guiding prognostic and therapeutic evaluation. ©2006 Nature Publishing Group.en_HK
dc.languageengen_HK
dc.publisherNature Publishing Group.en_HK
dc.relation.ispartofNature Clinical Practice Nephrologyen_HK
dc.subjectGlomerulonephritisen_HK
dc.subjectHepatitis Ben_HK
dc.subjectHepatitis Cen_HK
dc.subjectHIV-associated nephropathyen_HK
dc.subjectViral infectionen_HK
dc.titleViral nephropathyen_HK
dc.typeArticleen_HK
dc.identifier.openurlhttp://library.hku.hk:4550/resserv?sid=HKU:IR&issn=1745-8323&volume=2&issue=5&spage=254&epage=262&date=2006&atitle=Viral+nephropathyen_HK
dc.identifier.emailLai, KN: knlai@hku.hken_HK
dc.identifier.authorityLai, KN=rp00324en_HK
dc.description.naturelink_to_subscribed_fulltext-
dc.identifier.doi10.1038/ncpneph0166en_HK
dc.identifier.pmid16932438-
dc.identifier.scopuseid_2-s2.0-33646864778en_HK
dc.identifier.hkuros121481en_HK
dc.relation.referenceshttp://www.scopus.com/mlt/select.url?eid=2-s2.0-33646864778&selection=ref&src=s&origin=recordpageen_HK
dc.identifier.volume2en_HK
dc.identifier.issue5en_HK
dc.identifier.spage254en_HK
dc.identifier.epage262en_HK
dc.identifier.isiWOS:000237084300010-
dc.publisher.placeUnited Kingdomen_HK
dc.identifier.scopusauthoridLai, ASH=8711668800en_HK
dc.identifier.scopusauthoridLai, KN=7402135706en_HK

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