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Conference Paper: Electrical remodelling of chronic atrial fibrillation

TitleElectrical remodelling of chronic atrial fibrillation
Authors
Issue Date1997
PublisherBlackwell Publishing Asia. The Journal's web site is located at http://www.blackwellpublishing.com/journals/CEP
Citation
Clinical And Experimental Pharmacology And Physiology, 1997, v. 24 n. 12, p. 982-983 How to Cite?
Abstract1. It is now recognized that atrial fibrillation (AF) is not a benign condition, as it is associated with a 40% increase in mortality and a doubling of the risk of stroke. 2. The development of AF leads to mechanical, electrophysiological and cellular changes in the atria that fend to sustain AF. This process is known as atrial remodelling. 3. The three electrophysiological elements in the atria that initiate and sustain AF are: (i) shortening of the refractory period and an increase in dispersion; (ii) slowing of conduction velocity; and (iii) the presence of triggering foci. 4. As AF is a heterogeneous disorder, therapeutic strategies include the use of devices (pacemakers and atrial defibrillators), radiofrequency ablation (focal ablation or the creation of linear lines) and drug therapy that may reverse a remodelled atrium.
DescriptionProceedings of the Symposium on Cardiovascular Science and Medicine: From Bench to Bedside, Hong Kong, April 1997.
Persistent Identifierhttp://hdl.handle.net/10722/77424
ISSN
2012 Impact Factor: 2.16
2015 SCImago Journal Rankings: 0.944
ISI Accession Number ID
References

 

DC FieldValueLanguage
dc.contributor.authorLau, CPen_HK
dc.contributor.authorTse, HFen_HK
dc.date.accessioned2010-09-06T07:31:45Z-
dc.date.available2010-09-06T07:31:45Z-
dc.date.issued1997en_HK
dc.identifier.citationClinical And Experimental Pharmacology And Physiology, 1997, v. 24 n. 12, p. 982-983en_HK
dc.identifier.issn0305-1870en_HK
dc.identifier.urihttp://hdl.handle.net/10722/77424-
dc.descriptionProceedings of the Symposium on Cardiovascular Science and Medicine: From Bench to Bedside, Hong Kong, April 1997.-
dc.description.abstract1. It is now recognized that atrial fibrillation (AF) is not a benign condition, as it is associated with a 40% increase in mortality and a doubling of the risk of stroke. 2. The development of AF leads to mechanical, electrophysiological and cellular changes in the atria that fend to sustain AF. This process is known as atrial remodelling. 3. The three electrophysiological elements in the atria that initiate and sustain AF are: (i) shortening of the refractory period and an increase in dispersion; (ii) slowing of conduction velocity; and (iii) the presence of triggering foci. 4. As AF is a heterogeneous disorder, therapeutic strategies include the use of devices (pacemakers and atrial defibrillators), radiofrequency ablation (focal ablation or the creation of linear lines) and drug therapy that may reverse a remodelled atrium.en_HK
dc.languageengen_HK
dc.publisherBlackwell Publishing Asia. The Journal's web site is located at http://www.blackwellpublishing.com/journals/CEPen_HK
dc.relation.ispartofClinical and Experimental Pharmacology and Physiologyen_HK
dc.subject.meshAtrial Fibrillation - physiopathologyen_HK
dc.subject.meshChronic Diseaseen_HK
dc.subject.meshElectrophysiologyen_HK
dc.subject.meshHumansen_HK
dc.titleElectrical remodelling of chronic atrial fibrillationen_HK
dc.typeConference_Paperen_HK
dc.identifier.openurlhttp://library.hku.hk:4550/resserv?sid=HKU:IR&issn=0305-1870&volume=24&spage=982&epage=&date=1997&atitle=Electrical+Remodelling+of+Chronic+Atrial+Fibrillationen_HK
dc.identifier.emailTse, HF:hftse@hkucc.hku.hken_HK
dc.identifier.authorityTse, HF=rp00428en_HK
dc.description.naturelink_to_subscribed_fulltext-
dc.identifier.doi10.1111/j.1440-1681.1997.tb02734.x-
dc.identifier.pmid9406671-
dc.identifier.scopuseid_2-s2.0-0030831918en_HK
dc.identifier.hkuros32857en_HK
dc.identifier.hkuros28864-
dc.relation.referenceshttp://www.scopus.com/mlt/select.url?eid=2-s2.0-0030831918&selection=ref&src=s&origin=recordpageen_HK
dc.identifier.volume24en_HK
dc.identifier.issue12en_HK
dc.identifier.spage982en_HK
dc.identifier.epage983en_HK
dc.identifier.isiWOS:A1997YJ03600018-
dc.publisher.placeAustraliaen_HK
dc.identifier.scopusauthoridLau, CP=7401968501en_HK
dc.identifier.scopusauthoridTse, HF=7006070805en_HK

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