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Conference Paper: Reduction of intracellular calcium increase provides partial protection against β-amyloid peptide neurotoxicity

TitleReduction of intracellular calcium increase provides partial protection against β-amyloid peptide neurotoxicity
Authors
Suen, KCHugon, JSo, KFChang, RCC
KeywordsAmyloid
Apoptosis
Calcium
Caspases
Stress
Issue Date2003
PublisherBlackwell Publishing Ltd.
Citation
Journal of Neurochemistry, 2003, v. 87 suppl. 1, p. 96, abstract no. P09-20 How to Cite?
DOI: http://dx.doi.org/10.1046/j.1474-1644.2003.2175_9.x
Persistent Identifierhttp://hdl.handle.net/10722/67957
ISSN
0022-3042
2023 Impact Factor: 4.2
2023 SCImago Journal Rankings: 1.476

 

DC FieldValueLanguage
dc.contributor.authorSuen, KCen_HK
dc.contributor.authorHugon, Jen_HK
dc.contributor.authorSo, KFen_HK
dc.contributor.authorChang, RCCen_HK
dc.date.accessioned2010-09-06T05:59:48Z-
dc.date.available2010-09-06T05:59:48Z-
dc.date.issued2003en_HK
dc.identifier.citationJournal of Neurochemistry, 2003, v. 87 suppl. 1, p. 96, abstract no. P09-20en_HK
dc.identifier.issn0022-3042en_HK
dc.identifier.urihttp://hdl.handle.net/10722/67957-
dc.languageengen_HK
dc.publisherBlackwell Publishing Ltd.en_HK
dc.relation.ispartofJournal of Neurochemistryen_HK
dc.rightsThe definitive version is available at www.blackwell-synergy.comen_HK
dc.subjectAmyloid-
dc.subjectApoptosis-
dc.subjectCalcium-
dc.subjectCaspases-
dc.subjectStress-
dc.titleReduction of intracellular calcium increase provides partial protection against β-amyloid peptide neurotoxicityen_HK
dc.typeConference_Paperen_HK
dc.identifier.emailHugon, J: jhugon@hkucc.hku.hken_HK
dc.identifier.emailSo, KF: hrmaskf@hkucc.hku.hken_HK
dc.identifier.emailChang, RCC: rccchang@hkucc.hku.hken_HK
dc.identifier.authoritySo, KF=rp00329en_HK
dc.identifier.authorityChang, RCC=rp00470en_HK
dc.description.naturelink_to_OA_fulltext-
dc.identifier.doi10.1046/j.1474-1644.2003.2175_9.x-
dc.identifier.hkuros85229en_HK
dc.identifier.volume87-
dc.identifier.issueSuppl. 1-
dc.identifier.spage96, abstract no. P09-
dc.identifier.epage20-
dc.publisher.placeUnited Kingdom-
dc.identifier.issnl0022-3042-

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