Article: Latent membrane protein-1 of Epstein-Barr virus inhibits cell growth and induces sensitivity to cisplatin in nasopharyngeal carcinoma cells
| Title | Latent membrane protein-1 of Epstein-Barr virus inhibits cell growth and induces sensitivity to cisplatin in nasopharyngeal carcinoma cells |
|---|---|
| Authors | Liu, Y1 Wang, X1 Lo, AKF1 Wong, YC1 Cheung, ALM1 Tsao, SW1 |
| Issue Date | 2002 |
| Publisher | John Wiley & Sons, Inc. The Journal's web site is located at http://www3.interscience.wiley.com/cgi-bin/jhome/32763 |
| Citation | Journal Of Medical Virology, 2002, v. 66 n. 1, p. 63-69 [How to Cite?] DOI: http://dx.doi.org/10.1002/jmv.2112 |
| Abstract | Nasopharyngeal carcinoma is closely associated with Epstein-Barr virus (EBV) and the EBV encoded latent membrane protein-1 expression (LMP1) is commonly found in the tumour cells. LMP1 has been shown to be involved in modulation of cell growth in B cells but the biological properties of LMP1 expression in nasopharyngeal carcinoma cells are less defined. In this study, a full length LMP1 gene was introduced into an EBV negative nasopharyngeal carcinoma cell line, CNE2, and five LMPl-expressing clones were isolated. Expression of LMP1 did not confer cell growth advantage in CNE2 cells; instead, it induced growth inhibition both in vitro and in vivo. In addition, the LMP1 transfected cells were more susceptible to cisplatin-induced cell death and showed 1.4-4.0-fold increased sensitivity to cisplatin compared to the vector infected control clones. The effect of LMP1 on the balance of Bcl-2 and Bax ratio may play a role in inducing susceptibility to cisplatin-induced cell death. These results demonstrated that LMP1 did not confer growth advantage in CNE2 cells, suggesting that expression of LMP1 may not be crucial in sustaining cell growth in established cell lines. Alternatively, LMP1 alone may not be sufficient to facilitate nasopharyngeal carcinoma cell growth and additional oncogenic factors may be needed along with LMP1 in modulating the malignant property of nasopharyngeal carcinoma. © 2002 Wiley-Liss, Inc. |
| ISSN | 0146-6615 2011 Impact Factor: 2.82 2011 SCImago Journal Rankings: 0.267 |
| DOI | http://dx.doi.org/10.1002/jmv.2112 |
| References | References in Scopus |
| dc.contributor.author | Liu, Y |
|---|---|
| dc.contributor.author | Wang, X |
| dc.contributor.author | Lo, AKF |
| dc.contributor.author | Wong, YC |
| dc.contributor.author | Cheung, ALM |
| dc.contributor.author | Tsao, SW |
| dc.date.accessioned | 2010-09-06T05:54:27Z |
| dc.date.available | 2010-09-06T05:54:27Z |
| dc.date.issued | 2002 |
| dc.description.abstract | Nasopharyngeal carcinoma is closely associated with Epstein-Barr virus (EBV) and the EBV encoded latent membrane protein-1 expression (LMP1) is commonly found in the tumour cells. LMP1 has been shown to be involved in modulation of cell growth in B cells but the biological properties of LMP1 expression in nasopharyngeal carcinoma cells are less defined. In this study, a full length LMP1 gene was introduced into an EBV negative nasopharyngeal carcinoma cell line, CNE2, and five LMPl-expressing clones were isolated. Expression of LMP1 did not confer cell growth advantage in CNE2 cells; instead, it induced growth inhibition both in vitro and in vivo. In addition, the LMP1 transfected cells were more susceptible to cisplatin-induced cell death and showed 1.4-4.0-fold increased sensitivity to cisplatin compared to the vector infected control clones. The effect of LMP1 on the balance of Bcl-2 and Bax ratio may play a role in inducing susceptibility to cisplatin-induced cell death. These results demonstrated that LMP1 did not confer growth advantage in CNE2 cells, suggesting that expression of LMP1 may not be crucial in sustaining cell growth in established cell lines. Alternatively, LMP1 alone may not be sufficient to facilitate nasopharyngeal carcinoma cell growth and additional oncogenic factors may be needed along with LMP1 in modulating the malignant property of nasopharyngeal carcinoma. © 2002 Wiley-Liss, Inc. |
| dc.description.nature | Link_to_subscribed_fulltext |
| dc.identifier.citation | Journal Of Medical Virology, 2002, v. 66 n. 1, p. 63-69 [How to Cite?] DOI: http://dx.doi.org/10.1002/jmv.2112 |
| dc.identifier.doi | http://dx.doi.org/10.1002/jmv.2112 |
| dc.identifier.epage | 69 |
| dc.identifier.hkuros | 74658 |
| dc.identifier.isi | WOS:000172560100010 |
| dc.identifier.issn | 0146-6615 2011 Impact Factor: 2.82 2011 SCImago Journal Rankings: 0.267 |
| dc.identifier.issue | 1 |
| dc.identifier.openurl | ![]() |
| dc.identifier.pmid | 11748660 |
| dc.identifier.scopus | eid_2-s2.0-0036186372 |
| dc.identifier.spage | 63 |
| dc.identifier.uri | http://hdl.handle.net/10722/67362 |
| dc.identifier.volume | 66 |
| dc.language | eng |
| dc.publisher | John Wiley & Sons, Inc. The Journal's web site is located at http://www3.interscience.wiley.com/cgi-bin/jhome/32763 |
| dc.publisher.place | United States |
| dc.relation.ispartof | Journal of Medical Virology |
| dc.relation.references | References in Scopus |
| dc.rights | Journal of Medical Virology. Copyright © John Wiley & Sons, Inc. |
| dc.subject.mesh | Antineoplastic Agents - pharmacology |
| dc.subject.mesh | Cell Division |
| dc.subject.mesh | Cisplatin - pharmacology |
| dc.subject.mesh | Herpesvirus 4, Human - metabolism |
| dc.subject.mesh | Humans |
| dc.subject.mesh | Intracellular Signaling Peptides and Proteins |
| dc.subject.mesh | Nasopharyngeal Neoplasms - genetics - metabolism - pathology |
| dc.subject.mesh | Nuclear Proteins |
| dc.subject.mesh | Proteins - genetics - metabolism |
| dc.subject.mesh | Proto-Oncogene Proteins - genetics - metabolism |
| dc.subject.mesh | Proto-Oncogene Proteins c-bcl-2 - genetics - metabolism |
| dc.subject.mesh | Transfection |
| dc.subject.mesh | Tumor Cells, Cultured |
| dc.subject.mesh | Viral Matrix Proteins - genetics - metabolism - physiology |
| dc.subject.mesh | bcl-2-Associated X Protein |
| dc.title | Latent membrane protein-1 of Epstein-Barr virus inhibits cell growth and induces sensitivity to cisplatin in nasopharyngeal carcinoma cells |
| dc.type | Article |
Author Affiliations
- The University of Hong Kong


