File Download

There are no files associated with this item.

  Links for fulltext
     (May Require Subscription)
Supplementary

Article: Periodontal disease progression in subjects with orofacial clefts over a 25-year follow-up period

TitlePeriodontal disease progression in subjects with orofacial clefts over a 25-year follow-up period
Authors
Issue Date2009
PublisherBlackwell Munksgaard. The Journal's web site is located at http://www.blackwellpublishing.com/journals/CPE
Citation
Journal Of Clinical Periodontology, 2009, v. 36 n. 10, p. 836-842 How to Cite?
AbstractAims: To assess rates of periodontal disease progression in subjects with cleft lip, alveolus and palate (CLAP) over a 25-year period without regular maintenance care in a specialist setting and to compare those with those of subjects without alveolar clefts, i.e. cleft lip (CL) or cleft palate (CP). Material and Methods: Ten subjects with CLAP and 10 subjects with CL/CP were examined in 1979, 1987, 1993 and 2004. Probing pocket depth (PPD), clinical attachment level (CAL), bleeding on probing (BoP) and plaque control record (PCR) scores were recorded in all 20 subjects. Results: High plaque and BoP scores were recorded at all examinations in both groups. Over 25 years, a statistically significant loss of mean full-mouth CAL of 1.52 ± 0.12 mm (SD) and 1.66 ± 0.15 mm occurred in the CLAP and CL/CP group respectively (p<0.05). A statistically significant increase (p<0.05) in mean full-mouth PPD of 0.35 ± 0.12 mm was observed in the CL/CP group, whereas only a trend for a mean full-mouth increase in PPD of 0.09 ± 0.11 mm was observed in the CLAP group. In subjects with CLAP, a statistically significant increase (p<0.05) in PPD of 0.92 ± 1.13 mm at cleft sites was observed compared with that of 0.17 ± 0.76 mm at control sites. With respect to CAL, the loss at the corresponding sites amounted to 2.71 ± 1.46 and to 2.27 ± 1.62 mm, respectively (p=0.36). Conclusions: When stringent and well-defined supportive periodontal therapy was not provided, subjects with orofacial clefts were at high risk for periodontal disease progression. Over 25 years, alveolar cleft sites tended to have more periodontal tissue destruction compared with control sites. © 2009 John Wiley & Sons A/S.
Persistent Identifierhttp://hdl.handle.net/10722/67260
ISSN
2015 Impact Factor: 3.915
2015 SCImago Journal Rankings: 1.848
ISI Accession Number ID
Funding AgencyGrant Number
Clinical Research Foundation (CRF)
Promotion of Oral Health, Brienz, Switzerland
Funding Information:

This study was supported by the Clinical Research Foundation (CRF) for the Promotion of Oral Health, Brienz, Switzerland.

References

 

DC FieldValueLanguage
dc.contributor.authorHuynhBa, Gen_HK
dc.contributor.authorBrägger, Uen_HK
dc.contributor.authorZwahlen, Men_HK
dc.contributor.authorLang, NPen_HK
dc.contributor.authorSalvi, GEen_HK
dc.date.accessioned2010-09-06T05:53:20Z-
dc.date.available2010-09-06T05:53:20Z-
dc.date.issued2009en_HK
dc.identifier.citationJournal Of Clinical Periodontology, 2009, v. 36 n. 10, p. 836-842en_HK
dc.identifier.issn0303-6979en_HK
dc.identifier.urihttp://hdl.handle.net/10722/67260-
dc.description.abstractAims: To assess rates of periodontal disease progression in subjects with cleft lip, alveolus and palate (CLAP) over a 25-year period without regular maintenance care in a specialist setting and to compare those with those of subjects without alveolar clefts, i.e. cleft lip (CL) or cleft palate (CP). Material and Methods: Ten subjects with CLAP and 10 subjects with CL/CP were examined in 1979, 1987, 1993 and 2004. Probing pocket depth (PPD), clinical attachment level (CAL), bleeding on probing (BoP) and plaque control record (PCR) scores were recorded in all 20 subjects. Results: High plaque and BoP scores were recorded at all examinations in both groups. Over 25 years, a statistically significant loss of mean full-mouth CAL of 1.52 ± 0.12 mm (SD) and 1.66 ± 0.15 mm occurred in the CLAP and CL/CP group respectively (p<0.05). A statistically significant increase (p<0.05) in mean full-mouth PPD of 0.35 ± 0.12 mm was observed in the CL/CP group, whereas only a trend for a mean full-mouth increase in PPD of 0.09 ± 0.11 mm was observed in the CLAP group. In subjects with CLAP, a statistically significant increase (p<0.05) in PPD of 0.92 ± 1.13 mm at cleft sites was observed compared with that of 0.17 ± 0.76 mm at control sites. With respect to CAL, the loss at the corresponding sites amounted to 2.71 ± 1.46 and to 2.27 ± 1.62 mm, respectively (p=0.36). Conclusions: When stringent and well-defined supportive periodontal therapy was not provided, subjects with orofacial clefts were at high risk for periodontal disease progression. Over 25 years, alveolar cleft sites tended to have more periodontal tissue destruction compared with control sites. © 2009 John Wiley & Sons A/S.en_HK
dc.languageengen_HK
dc.publisherBlackwell Munksgaard. The Journal's web site is located at http://www.blackwellpublishing.com/journals/CPEen_HK
dc.relation.ispartofJournal of Clinical Periodontologyen_HK
dc.subject.meshAdulten_HK
dc.subject.meshAlveolar Process - abnormalitiesen_HK
dc.subject.meshChronic Periodontitis - physiopathologyen_HK
dc.subject.meshCleft Lip - complicationsen_HK
dc.subject.meshCleft Palate - complicationsen_HK
dc.subject.meshCohort Studiesen_HK
dc.subject.meshDental Plaque - physiopathologyen_HK
dc.subject.meshDisease Progressionen_HK
dc.subject.meshFemaleen_HK
dc.subject.meshFollow-Up Studiesen_HK
dc.subject.meshGingival Hemorrhage - physiopathologyen_HK
dc.subject.meshHumansen_HK
dc.subject.meshMaleen_HK
dc.subject.meshMiddle Ageden_HK
dc.subject.meshPeriodontal Attachment Loss - physiopathologyen_HK
dc.subject.meshPeriodontal Pocket - physiopathologyen_HK
dc.subject.meshRisk Factorsen_HK
dc.subject.meshTooth Loss - physiopathologyen_HK
dc.titlePeriodontal disease progression in subjects with orofacial clefts over a 25-year follow-up perioden_HK
dc.typeArticleen_HK
dc.identifier.openurlhttp://library.hku.hk:4550/resserv?sid=HKU:IR&issn=0303-6979&volume=36&spage=836&epage=842&date=2009&atitle=Periodontal+disease+progression+in+subjects+with+orofacial+clefts+over+a+25-year+follow-up+perioden_HK
dc.identifier.emailLang, NP:nplang@hkucc.hku.hken_HK
dc.identifier.authorityLang, NP=rp00031en_HK
dc.description.naturelink_to_subscribed_fulltext-
dc.identifier.doi10.1111/j.1600-051X.2009.01469.xen_HK
dc.identifier.pmid19703238-
dc.identifier.scopuseid_2-s2.0-70349149352en_HK
dc.identifier.hkuros169065en_HK
dc.relation.referenceshttp://www.scopus.com/mlt/select.url?eid=2-s2.0-70349149352&selection=ref&src=s&origin=recordpageen_HK
dc.identifier.volume36en_HK
dc.identifier.issue10en_HK
dc.identifier.spage836en_HK
dc.identifier.epage842en_HK
dc.identifier.isiWOS:000269728700004-
dc.publisher.placeDenmarken_HK
dc.identifier.scopusauthoridHuynhBa, G=25825004200en_HK
dc.identifier.scopusauthoridBrägger, U=7005538598en_HK
dc.identifier.scopusauthoridZwahlen, M=7004748418en_HK
dc.identifier.scopusauthoridLang, NP=7201577367en_HK
dc.identifier.scopusauthoridSalvi, GE=35600695300en_HK
dc.identifier.citeulike5789916-

Export via OAI-PMH Interface in XML Formats


OR


Export to Other Non-XML Formats