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Article: Recent advances in the aetiology of adolescent idiopathic scoliosis

TitleRecent advances in the aetiology of adolescent idiopathic scoliosis
Authors
Issue Date2007
PublisherSpringer. The Journal's web site is located at http://www.springer.com/medicine/orthopedics/journal/264
Citation
International Orthopaedics, 2007, v. 32 n. 6, p. 729-734 How to Cite?
AbstractThe aetiology of adolescent idiopathic scoliosis (AIS) is still unknown despite many years of research effort. Theories on AIS's aetiology have included mechanical, hormonal, metabolic, neuromuscular, growth, and genetic abnormalities. Amongst these, some factors may be epiphenomena rather than the cause itself. Other factors may even contribute to curve progression, rather than curve initiation. Current views maintain that AIS is a multifactorial disease with genetic predisposing factors [Lowe et al. in J Bone Joint Surg [Am] 82:1157-1168, 2000]. With improvements in diagnostic methods, imaging and genomics, there has been considerable recent work on aetiology. This review aims to bring readers up-to-date with the latest developments in scoliosis research. © 2007 Springer-Verlag.
Persistent Identifierhttp://hdl.handle.net/10722/62602
ISSN
2015 Impact Factor: 2.387
2015 SCImago Journal Rankings: 1.508
ISI Accession Number ID
References

 

DC FieldValueLanguage
dc.contributor.authorCheung, KMCen_HK
dc.contributor.authorWang, Ten_HK
dc.contributor.authorQiu, GXen_HK
dc.contributor.authorLuk, KDKen_HK
dc.date.accessioned2010-07-13T04:04:51Z-
dc.date.available2010-07-13T04:04:51Z-
dc.date.issued2007en_HK
dc.identifier.citationInternational Orthopaedics, 2007, v. 32 n. 6, p. 729-734en_HK
dc.identifier.issn0341-2695en_HK
dc.identifier.urihttp://hdl.handle.net/10722/62602-
dc.description.abstractThe aetiology of adolescent idiopathic scoliosis (AIS) is still unknown despite many years of research effort. Theories on AIS's aetiology have included mechanical, hormonal, metabolic, neuromuscular, growth, and genetic abnormalities. Amongst these, some factors may be epiphenomena rather than the cause itself. Other factors may even contribute to curve progression, rather than curve initiation. Current views maintain that AIS is a multifactorial disease with genetic predisposing factors [Lowe et al. in J Bone Joint Surg [Am] 82:1157-1168, 2000]. With improvements in diagnostic methods, imaging and genomics, there has been considerable recent work on aetiology. This review aims to bring readers up-to-date with the latest developments in scoliosis research. © 2007 Springer-Verlag.en_HK
dc.languageengen_HK
dc.publisherSpringer. The Journal's web site is located at http://www.springer.com/medicine/orthopedics/journal/264en_HK
dc.relation.ispartofInternational Orthopaedicsen_HK
dc.rightsThe original publication is available at www.springerlink.com-
dc.subject.meshScoliosis - etiology - physiopathologyen_HK
dc.subject.meshHumansen_HK
dc.subject.meshBiomechanicsen_HK
dc.subject.meshAdolescent Development - physiologyen_HK
dc.subject.meshAdolescenten_HK
dc.titleRecent advances in the aetiology of adolescent idiopathic scoliosisen_HK
dc.typeArticleen_HK
dc.identifier.openurlhttp://library.hku.hk:4550/resserv?sid=HKU:IR&issn=0341-2695&volume=32&issue=6&spage=729&epage=734&date=2008&atitle=Recent+advances+in+the+aetiology+of+adolescent+idiopathic+scoliosisen_HK
dc.identifier.emailCheung, KMC: cheungmc@hku.hken_HK
dc.identifier.emailLuk, KDK: cm21000@hku.hken_HK
dc.identifier.authorityCheung, KMC=rp00387en_HK
dc.identifier.authorityLuk, KDK=rp00333en_HK
dc.description.naturelink_to_subscribed_fulltext-
dc.identifier.doi10.1007/s00264-007-0393-yen_HK
dc.identifier.pmid17572891-
dc.identifier.scopuseid_2-s2.0-58249119468en_HK
dc.identifier.hkuros166636-
dc.relation.referenceshttp://www.scopus.com/mlt/select.url?eid=2-s2.0-58249119468&selection=ref&src=s&origin=recordpageen_HK
dc.identifier.volume32en_HK
dc.identifier.issue6en_HK
dc.identifier.spage729en_HK
dc.identifier.epage734en_HK
dc.identifier.isiWOS:000262490700002-
dc.publisher.placeGermanyen_HK
dc.identifier.scopusauthoridLuk, KDK=7201921573en_HK
dc.identifier.scopusauthoridQiu, GX=7103291762en_HK
dc.identifier.scopusauthoridWang, T=7405564819en_HK
dc.identifier.scopusauthoridCheung, KMC=7402406754en_HK
dc.customcontrol.immutablesml 140807-

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