Article: DSM-IV combined type ADHD shows familial association with sibling trait scores: A sampling strategy for QTL linkage
| Title | DSM-IV combined type ADHD shows familial association with sibling trait scores: A sampling strategy for QTL linkage | ||||
|---|---|---|---|---|---|
| Authors | Chen, W3 Zhou, K3 Sham, P3 4 Franke, B18 Kuntsi, J3 Campbell, D3 Fleischman, K3 Knight, J3 Andreou, P3 Arnold, R9 Altink, M18 Boer, F5 Boholst, MJ3 Buschgens, C18 Butler, L1 Christiansen, H16 Fliers, E11 18 HoweForbes, R3 Gabriëls, I7 Heise, A10 KornLubetzki, I19 Marco, R13 Medad, S2 Minderaa, R8 Müller, UC20 Mulligan, A1 Psychogiou, L6 Rommelse, N17 Sethna, V3 Uebel, H10 McGuffin, P3 Plomin, R3 Banaschewski, T10 14 Buitelaar, J18 Ebstein, R12 Eisenberg, J12 Gill, M1 Manor, I2 Miranda, A13 Mulas, F15 Oades, RD16 Roeyers, H7 Rothenberger, A10 Sergeant, J17 SonugaBarke, E6 Steinhausen, HC20 Taylor, E3 Thompson, M6 Faraone, SV21 Asherson, P3 | ||||
| Keywords | Attention deficit hyperactivity disorder (ADHD) DF analysis Linkage study Quantitative genetics Quantitative trait locus (QTL) | ||||
| Issue Date | 2008 | ||||
| Publisher | John Wiley & Sons, Inc. The Journal's web site is located at http://www.interscience.wiley.com/jpages/0148-7299:1 | ||||
| Citation | American Journal Of Medical Genetics, Part B: Neuropsychiatric Genetics, 2008, v. 147 n. 8, p. 1450-1460 [How to Cite?] DOI: http://dx.doi.org/10.1002/ajmg.b.30672 | ||||
| Abstract | Attention deficit hyperactivity disorder (ADHD) is a discrete clinical syndrome characterized by the triad of inattention, hyperactivity, and impulsivity in the context of marked impairments. Molecular genetic studies have been successful in identifying genetic variants associated with ADHD, particularly with DSM-IV inattentive and combined subtypes. Quantitative trait locus (QTL) approaches to linkage and association mapping have yet to be widely used in ADHD research, although twin studies investigating individual differences suggest that genetic liability for ADHD is continuously distributedthroughout the population, underscoring the applicability of quantitative dimensional approaches. To investigate the appropriateness of QTL approaches, we tested the familial association between 894 probands with a research diagnosis of DSM-IV ADHD combined type and continuous trait measures among 1,135 of their siblings unselected for phenotype. The sibling recurrence rate for ADHD combined subtype was 12.7%, yielding a sibling recurrence risk ratio (λsib) of 9.0. Estimated sibling correlations around 0.2-0.3 are similar to those estimated from the analysis of fraternal twins in population twin samples. We further show that there are no threshold effects on the sibling risk for ADHD among the ADHD probands; and that both affected and unaffected siblings contributed to the association with ADHD trait scores. In conclusion, these data confirm the main requirement for QTL mapping of ADHD by demonstrating that narrowly defined DSM-IV combined type probands show familial association with dimensional ADHD symptom scores amongst their siblings. © 2008 Wiley-Liss, Inc. | ||||
| ISSN | 1552-485X 2011 SCImago Journal Rankings: 0.288 | ||||
| DOI | http://dx.doi.org/10.1002/ajmg.b.30672 | ||||
| ISI Accession Number ID | WOS:000261415800018
Funding Information: This work was funded by NIMH Grant R01MH062873 to Stephen V. Faraone. | ||||
| References | References in Scopus |
| dc.contributor.author | Chen, W | ||||
|---|---|---|---|---|---|
| dc.contributor.author | Zhou, K | ||||
| dc.contributor.author | Sham, P | ||||
| dc.contributor.author | Franke, B | ||||
| dc.contributor.author | Kuntsi, J | ||||
| dc.contributor.author | Campbell, D | ||||
| dc.contributor.author | Fleischman, K | ||||
| dc.contributor.author | Knight, J | ||||
| dc.contributor.author | Andreou, P | ||||
| dc.contributor.author | Arnold, R | ||||
| dc.contributor.author | Altink, M | ||||
| dc.contributor.author | Boer, F | ||||
| dc.contributor.author | Boholst, MJ | ||||
| dc.contributor.author | Buschgens, C | ||||
| dc.contributor.author | Butler, L | ||||
| dc.contributor.author | Christiansen, H | ||||
| dc.contributor.author | Fliers, E | ||||
| dc.contributor.author | HoweForbes, R | ||||
| dc.contributor.author | Gabriëls, I | ||||
| dc.contributor.author | Heise, A | ||||
| dc.contributor.author | KornLubetzki, I | ||||
| dc.contributor.author | Marco, R | ||||
| dc.contributor.author | Medad, S | ||||
| dc.contributor.author | Minderaa, R | ||||
| dc.contributor.author | Müller, UC | ||||
| dc.contributor.author | Mulligan, A | ||||
| dc.contributor.author | Psychogiou, L | ||||
| dc.contributor.author | Rommelse, N | ||||
| dc.contributor.author | Sethna, V | ||||
| dc.contributor.author | Uebel, H | ||||
| dc.contributor.author | McGuffin, P | ||||
| dc.contributor.author | Plomin, R | ||||
| dc.contributor.author | Banaschewski, T | ||||
| dc.contributor.author | Buitelaar, J | ||||
| dc.contributor.author | Ebstein, R | ||||
| dc.contributor.author | Eisenberg, J | ||||
| dc.contributor.author | Gill, M | ||||
| dc.contributor.author | Manor, I | ||||
| dc.contributor.author | Miranda, A | ||||
| dc.contributor.author | Mulas, F | ||||
| dc.contributor.author | Oades, RD | ||||
| dc.contributor.author | Roeyers, H | ||||
| dc.contributor.author | Rothenberger, A | ||||
| dc.contributor.author | Sergeant, J | ||||
| dc.contributor.author | SonugaBarke, E | ||||
| dc.contributor.author | Steinhausen, HC | ||||
| dc.contributor.author | Taylor, E | ||||
| dc.contributor.author | Thompson, M | ||||
| dc.contributor.author | Faraone, SV | ||||
| dc.contributor.author | Asherson, P | ||||
| dc.date.accessioned | 2010-05-31T03:55:38Z | ||||
| dc.date.available | 2010-05-31T03:55:38Z | ||||
| dc.date.issued | 2008 | ||||
| dc.description.abstract | Attention deficit hyperactivity disorder (ADHD) is a discrete clinical syndrome characterized by the triad of inattention, hyperactivity, and impulsivity in the context of marked impairments. Molecular genetic studies have been successful in identifying genetic variants associated with ADHD, particularly with DSM-IV inattentive and combined subtypes. Quantitative trait locus (QTL) approaches to linkage and association mapping have yet to be widely used in ADHD research, although twin studies investigating individual differences suggest that genetic liability for ADHD is continuously distributedthroughout the population, underscoring the applicability of quantitative dimensional approaches. To investigate the appropriateness of QTL approaches, we tested the familial association between 894 probands with a research diagnosis of DSM-IV ADHD combined type and continuous trait measures among 1,135 of their siblings unselected for phenotype. The sibling recurrence rate for ADHD combined subtype was 12.7%, yielding a sibling recurrence risk ratio (λsib) of 9.0. Estimated sibling correlations around 0.2-0.3 are similar to those estimated from the analysis of fraternal twins in population twin samples. We further show that there are no threshold effects on the sibling risk for ADHD among the ADHD probands; and that both affected and unaffected siblings contributed to the association with ADHD trait scores. In conclusion, these data confirm the main requirement for QTL mapping of ADHD by demonstrating that narrowly defined DSM-IV combined type probands show familial association with dimensional ADHD symptom scores amongst their siblings. © 2008 Wiley-Liss, Inc. | ||||
| dc.description.nature | Link_to_subscribed_fulltext | ||||
| dc.identifier.citation | American Journal Of Medical Genetics, Part B: Neuropsychiatric Genetics, 2008, v. 147 n. 8, p. 1450-1460 [How to Cite?] DOI: http://dx.doi.org/10.1002/ajmg.b.30672 | ||||
| dc.identifier.citeulike | 10600645 | ||||
| dc.identifier.doi | http://dx.doi.org/10.1002/ajmg.b.30672 | ||||
| dc.identifier.epage | 1460 | ||||
| dc.identifier.hkuros | 162575 | ||||
| dc.identifier.isi | WOS:000261415800018
Funding Information: This work was funded by NIMH Grant R01MH062873 to Stephen V. Faraone. | ||||
| dc.identifier.issn | 1552-485X 2011 SCImago Journal Rankings: 0.288 | ||||
| dc.identifier.issue | 8 | ||||
| dc.identifier.openurl | ![]() | ||||
| dc.identifier.pmid | 18189238 | ||||
| dc.identifier.scopus | eid_2-s2.0-57349174136 | ||||
| dc.identifier.spage | 1450 | ||||
| dc.identifier.uri | http://hdl.handle.net/10722/59698 | ||||
| dc.identifier.volume | 147 | ||||
| dc.language | eng | ||||
| dc.publisher | John Wiley & Sons, Inc. The Journal's web site is located at http://www.interscience.wiley.com/jpages/0148-7299:1 | ||||
| dc.relation.ispartof | American Journal of Medical Genetics, Part B: Neuropsychiatric Genetics | ||||
| dc.relation.references | References in Scopus | ||||
| dc.rights | American Journal of Medical Genetics Part B: Neuropsychiatric Genetics. Copyright © John Wiley & Sons, Inc. | ||||
| dc.subject | Attention deficit hyperactivity disorder (ADHD) | ||||
| dc.subject | DF analysis | ||||
| dc.subject | Linkage study | ||||
| dc.subject | Quantitative genetics | ||||
| dc.subject | Quantitative trait locus (QTL) | ||||
| dc.title | DSM-IV combined type ADHD shows familial association with sibling trait scores: A sampling strategy for QTL linkage | ||||
| dc.type | Article |
Author Affiliations
- Trinity College Dublin
- Geha Mental Health Center
- King's College London
- The University of Hong Kong Li Ka Shing Faculty of Medicine
- Academic Medical Centre, University of Amsterdam
- University of Southampton
- Universiteit Gent
- Universitair Medisch Centrum Groningen
- Center for Child and Adolescent Psychiatry
- Universität Göttingen
- BAVO-Europoort
- Herzog Hospital Jerusalem
- Universitat de Valencia
- Zentralinstitut für Seelische Gesundheit
- Hospital Universitario La Fe
- Universitäts Klinikum Essen und Medizinische Fakultät
- Vrije Universiteit Amsterdam
- Radboud University Nijmegen Medical Centre
- Shaare Zedek Medical Center
- Universität Zürich
- State University of New York Upstate Medical University


