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Article: Analysis of Ventricular Performance as a Function of Pacing Site and Mode

TitleAnalysis of Ventricular Performance as a Function of Pacing Site and Mode
Authors
Issue Date2008
PublisherWB Saunders Co. The Journal's web site is located at http://www.elsevier.com/locate/pcad
Citation
Progress In Cardiovascular Diseases, 2008, v. 51 n. 2, p. 171-182 How to Cite?
AbstractEmerging data from experimental and clinical studies have shown that right ventricular (RV) apical pacing led to abnormalities of ventricular activation and contraction, and impairment of myocardial perfusion with adverse left ventricular (LV) remodeling, which was associated with increased risk of cardiac morbidity and mortality. As a result, there is a growing interest in searching for methods to minimize unnecessary RV pacing and preserving normal ventricular activation with alternative ventricular pacing sites. The risk of developing heart failure (HF) after RV apical pacing depends on the interactions between patient-specific factors (baseline atrial rhythm, intrinsic atrioventricular and ventricular conduction, LV ejection fraction, and the presence of HF and myocardial infarction) and pacing-related factors (mode of pacing, site of ventricular pacing, paced QRS duration, and percentage and duration of pacing). In patients with intact atrioventricular conduction, atrial-based pacing should be used to avoid unnecessary ventricular pacing. In patients requiring ventricular pacing, the potential benefits of alternate ventricular pacing sites, such as RV or LV septa, or even biventricular pacing in different patient populations remain unclear and warrant further long-term prospective clinical trial evaluations especially in those patients who are at a higher risk of developing HF after RV apical pacing. © 2008 Elsevier Inc. All rights reserved.
Persistent Identifierhttp://hdl.handle.net/10722/59346
ISSN
2015 Impact Factor: 4.635
2015 SCImago Journal Rankings: 1.847
ISI Accession Number ID
References

 

DC FieldValueLanguage
dc.contributor.authorSiu, CWen_HK
dc.contributor.authorWang, Men_HK
dc.contributor.authorZhang, XHen_HK
dc.contributor.authorLau, CPen_HK
dc.contributor.authorTse, HFen_HK
dc.date.accessioned2010-05-31T03:48:09Z-
dc.date.available2010-05-31T03:48:09Z-
dc.date.issued2008en_HK
dc.identifier.citationProgress In Cardiovascular Diseases, 2008, v. 51 n. 2, p. 171-182en_HK
dc.identifier.issn0033-0620en_HK
dc.identifier.urihttp://hdl.handle.net/10722/59346-
dc.description.abstractEmerging data from experimental and clinical studies have shown that right ventricular (RV) apical pacing led to abnormalities of ventricular activation and contraction, and impairment of myocardial perfusion with adverse left ventricular (LV) remodeling, which was associated with increased risk of cardiac morbidity and mortality. As a result, there is a growing interest in searching for methods to minimize unnecessary RV pacing and preserving normal ventricular activation with alternative ventricular pacing sites. The risk of developing heart failure (HF) after RV apical pacing depends on the interactions between patient-specific factors (baseline atrial rhythm, intrinsic atrioventricular and ventricular conduction, LV ejection fraction, and the presence of HF and myocardial infarction) and pacing-related factors (mode of pacing, site of ventricular pacing, paced QRS duration, and percentage and duration of pacing). In patients with intact atrioventricular conduction, atrial-based pacing should be used to avoid unnecessary ventricular pacing. In patients requiring ventricular pacing, the potential benefits of alternate ventricular pacing sites, such as RV or LV septa, or even biventricular pacing in different patient populations remain unclear and warrant further long-term prospective clinical trial evaluations especially in those patients who are at a higher risk of developing HF after RV apical pacing. © 2008 Elsevier Inc. All rights reserved.en_HK
dc.languageengen_HK
dc.publisherWB Saunders Co. The Journal's web site is located at http://www.elsevier.com/locate/pcaden_HK
dc.relation.ispartofProgress in Cardiovascular Diseasesen_HK
dc.subject.meshAnimalsen_HK
dc.subject.meshAtrioventricular Block - physiopathology - therapyen_HK
dc.subject.meshBundle-Branch Block - physiopathology - therapyen_HK
dc.subject.meshCardiac Pacing, Artificial - adverse effects - methods - trendsen_HK
dc.subject.meshHeart Conduction System - physiopathologyen_HK
dc.subject.meshHeart Failure - etiology - physiopathologyen_HK
dc.subject.meshHemodynamicsen_HK
dc.subject.meshHumansen_HK
dc.subject.meshMyocardial Contractionen_HK
dc.subject.meshPatient Selectionen_HK
dc.subject.meshRisk Assessmenten_HK
dc.subject.meshVentricular Dysfunction, Left - etiology - physiopathologyen_HK
dc.subject.meshVentricular Function, Righten_HK
dc.subject.meshVentricular Remodelingen_HK
dc.titleAnalysis of Ventricular Performance as a Function of Pacing Site and Modeen_HK
dc.typeArticleen_HK
dc.identifier.openurlhttp://library.hku.hk:4550/resserv?sid=HKU:IR&issn=0033-0620&volume=51&issue=2&spage=171&epage=82&date=2008&atitle=Analysis+of+ventricular+performance+as+a+function+of+pacing+site+and+modeen_HK
dc.identifier.emailSiu, CW:cwdsiu@hkucc.hku.hken_HK
dc.identifier.emailWang, M:meiwang@hkucc.hku.hken_HK
dc.identifier.emailTse, HF:hftse@hkucc.hku.hken_HK
dc.identifier.authoritySiu, CW=rp00534en_HK
dc.identifier.authorityWang, M=rp00281en_HK
dc.identifier.authorityTse, HF=rp00428en_HK
dc.description.naturelink_to_subscribed_fulltext-
dc.identifier.doi10.1016/j.pcad.2008.01.001en_HK
dc.identifier.pmid18774015-
dc.identifier.scopuseid_2-s2.0-50549101623en_HK
dc.identifier.hkuros151520en_HK
dc.relation.referenceshttp://www.scopus.com/mlt/select.url?eid=2-s2.0-50549101623&selection=ref&src=s&origin=recordpageen_HK
dc.identifier.volume51en_HK
dc.identifier.issue2en_HK
dc.identifier.spage171en_HK
dc.identifier.epage182en_HK
dc.identifier.isiWOS:000259466400007-
dc.publisher.placeUnited Statesen_HK
dc.identifier.scopusauthoridSiu, CW=7006550690en_HK
dc.identifier.scopusauthoridWang, M=7406690398en_HK
dc.identifier.scopusauthoridZhang, XH=16425051500en_HK
dc.identifier.scopusauthoridLau, CP=7401968501en_HK
dc.identifier.scopusauthoridTse, HF=7006070805en_HK

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