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Article: The aetiology of idiopathic Parkinson's disease

TitleThe aetiology of idiopathic Parkinson's disease
Authors
Keywordsα synuclein
Environmental agents
Parkin
Parkinson's disease
Transcription factors
Ubiquitin carboxy terminal hydroxylase L1
Issue Date2001
PublisherB M J Publishing Group. The Journal's web site is located at http://mp.bmjjournals.com/
Citation
Journal Of Clinical Pathology - Molecular Pathology, 2001, v. 54 n. 6, p. 369-380 How to Cite?
AbstractAgents potentially involved in the aetiology of idiopathic Parkinson's disease are discussed. These include factors regulating dopaminergic neurogenesis (Nurr 1, Ptx-3, and Lmx1b) and related proteins, together with genes involved in familial Parkinson's disease (α synuclein, parkin, and ubiquitin carboxy terminal hydroxylase L1), and endogenous and environmental agents.
Persistent Identifierhttp://hdl.handle.net/10722/45134
ISSN
2005 Impact Factor: 2.576
2005 SCImago Journal Rankings: 0.122
PubMed Central ID
References

 

DC FieldValueLanguage
dc.contributor.authorRamsden, DBen_HK
dc.contributor.authorParsons, RBen_HK
dc.contributor.authorHo, SLen_HK
dc.contributor.authorWaring, RHen_HK
dc.date.accessioned2007-10-30T06:18:06Z-
dc.date.available2007-10-30T06:18:06Z-
dc.date.issued2001en_HK
dc.identifier.citationJournal Of Clinical Pathology - Molecular Pathology, 2001, v. 54 n. 6, p. 369-380en_HK
dc.identifier.issn1366-8714en_HK
dc.identifier.urihttp://hdl.handle.net/10722/45134-
dc.description.abstractAgents potentially involved in the aetiology of idiopathic Parkinson's disease are discussed. These include factors regulating dopaminergic neurogenesis (Nurr 1, Ptx-3, and Lmx1b) and related proteins, together with genes involved in familial Parkinson's disease (α synuclein, parkin, and ubiquitin carboxy terminal hydroxylase L1), and endogenous and environmental agents.en_HK
dc.format.extent351839 bytes-
dc.format.extent9894 bytes-
dc.format.mimetypeapplication/pdf-
dc.format.mimetypetext/plain-
dc.languageengen_HK
dc.publisherB M J Publishing Group. The Journal's web site is located at http://mp.bmjjournals.com/en_HK
dc.relation.ispartofJournal of Clinical Pathology - Molecular Pathologyen_HK
dc.rightsCreative Commons: Attribution 3.0 Hong Kong License-
dc.rightsJournal of Clinical Pathology. Copyright © B M J Publishing Group.en_HK
dc.subjectα synucleinen_HK
dc.subjectEnvironmental agentsen_HK
dc.subjectParkinen_HK
dc.subjectParkinson's diseaseen_HK
dc.subjectTranscription factorsen_HK
dc.subjectUbiquitin carboxy terminal hydroxylase L1en_HK
dc.subject.meshDioxygenasesen_HK
dc.subject.meshParkinson Disease - etiology - genetics - metabolismen_HK
dc.subject.meshC-Reactive Protein - genetics - metabolismen_HK
dc.subject.meshHomeodomain Proteins - genetics - metabolismen_HK
dc.subject.meshSerum Amyloid P-Component - genetics - metabolismen_HK
dc.titleThe aetiology of idiopathic Parkinson's diseaseen_HK
dc.typeArticleen_HK
dc.identifier.openurlhttp://library.hku.hk:4550/resserv?sid=HKU:IR&issn=0021-9746&volume=54&issue=6&spage=369&epage=380&date=2001&atitle=The+aetiology+of+idiopathic+Parkinson%27s+diseaseen_HK
dc.identifier.emailHo, SL:slho@hku.hken_HK
dc.identifier.authorityHo, SL=rp00240en_HK
dc.description.naturepublished_or_final_versionen_HK
dc.identifier.pmid11724911-
dc.identifier.pmcidPMC1187126-
dc.identifier.scopuseid_2-s2.0-0035659556en_HK
dc.identifier.hkuros82356-
dc.relation.referenceshttp://www.scopus.com/mlt/select.url?eid=2-s2.0-0035659556&selection=ref&src=s&origin=recordpageen_HK
dc.identifier.volume54en_HK
dc.identifier.issue6en_HK
dc.identifier.spage369en_HK
dc.identifier.epage380en_HK
dc.publisher.placeUnited Kingdomen_HK
dc.identifier.scopusauthoridRamsden, DB=7102612805en_HK
dc.identifier.scopusauthoridParsons, RB=7202030894en_HK
dc.identifier.scopusauthoridHo, SL=25959633500en_HK
dc.identifier.scopusauthoridWaring, RH=7102650741en_HK

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