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Article: Spinal Primitive neuroectodermal tumor mimicking as chronic inflammatory demyelination polyneuropathy: A case report and review of literature

TitleSpinal Primitive neuroectodermal tumor mimicking as chronic inflammatory demyelination polyneuropathy: A case report and review of literature
Authors
KeywordsChronic inflammatory demyelinating polyneuropathy
Spinal primitive neuroectodermal tumor
Issue Date2015
Citation
Journal of Child Neurology, 2015, v. 30, n. 2, p. 254-258 How to Cite?
Abstract© The Author(s) 2014.We report a young boy who presented with progressive weakness of lower extremities associated with areflexia and abnormal electrophysiological findings initially suggestive of chronic inflammatory demyelinating polyneuropathy. Initial lumbosacral spinal magnetic resonance imaging (MRI) showed thickened descending spinal nerve roots only. Immunomodulating therapy was given but with limited clinical response. Repeated spine magnetic resonance imaging showed cauda equina and also new spinal cord extramedullary contrast enhancement. The initial extensive investigations including open biopsy did not point to any specific diagnosis. Only through pursuing a repeated biopsy, the diagnosis of the spinal peripheral primitive neuroectodermal tumor was confirmed. This case highlights the diagnostic challenges of the spinal peripheral primitive neuroectodermal tumor that could have an initial chronic inflammatory demyelinating polyneuropathy-like presentation. The literature review confirms that this is a rare condition and cauda equina origin has only been reported in adults and teenagers, and this is the first reported case in a young child.
Persistent Identifierhttp://hdl.handle.net/10722/233749
ISSN
2015 Impact Factor: 1.434
2015 SCImago Journal Rankings: 0.694

 

DC FieldValueLanguage
dc.contributor.authorChan, Sophelia H S-
dc.contributor.authorTsang, Dickson S F-
dc.contributor.authorWong, Virginia C N-
dc.contributor.authorChan, Godfrey C F-
dc.date.accessioned2016-09-27T07:21:32Z-
dc.date.available2016-09-27T07:21:32Z-
dc.date.issued2015-
dc.identifier.citationJournal of Child Neurology, 2015, v. 30, n. 2, p. 254-258-
dc.identifier.issn0883-0738-
dc.identifier.urihttp://hdl.handle.net/10722/233749-
dc.description.abstract© The Author(s) 2014.We report a young boy who presented with progressive weakness of lower extremities associated with areflexia and abnormal electrophysiological findings initially suggestive of chronic inflammatory demyelinating polyneuropathy. Initial lumbosacral spinal magnetic resonance imaging (MRI) showed thickened descending spinal nerve roots only. Immunomodulating therapy was given but with limited clinical response. Repeated spine magnetic resonance imaging showed cauda equina and also new spinal cord extramedullary contrast enhancement. The initial extensive investigations including open biopsy did not point to any specific diagnosis. Only through pursuing a repeated biopsy, the diagnosis of the spinal peripheral primitive neuroectodermal tumor was confirmed. This case highlights the diagnostic challenges of the spinal peripheral primitive neuroectodermal tumor that could have an initial chronic inflammatory demyelinating polyneuropathy-like presentation. The literature review confirms that this is a rare condition and cauda equina origin has only been reported in adults and teenagers, and this is the first reported case in a young child.-
dc.languageeng-
dc.relation.ispartofJournal of Child Neurology-
dc.subjectChronic inflammatory demyelinating polyneuropathy-
dc.subjectSpinal primitive neuroectodermal tumor-
dc.titleSpinal Primitive neuroectodermal tumor mimicking as chronic inflammatory demyelination polyneuropathy: A case report and review of literature-
dc.typeArticle-
dc.description.natureLink_to_subscribed_fulltext-
dc.identifier.doi10.1177/0883073814527160-
dc.identifier.pmid24659733-
dc.identifier.scopuseid_2-s2.0-84927761924-
dc.identifier.hkuros229006-
dc.identifier.volume30-
dc.identifier.issue2-
dc.identifier.spage254-
dc.identifier.epage258-
dc.identifier.eissn1708-8283-

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