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Article: Tuberculous Constrictive Pericarditis

TitleTuberculous Constrictive Pericarditis
Authors
KeywordsConstrictive Pericarditides
Pericardial Effusion
Tuberculosis
Issue Date2015
PublisherRajaie Cardiovascular, Medical & Research Center.
Citation
Research in Cardiovascular Medicine, 2015, v. 4 n. 4, p. e29614 How to Cite?
AbstractIntroduction: Constrictive pericarditis is characterized by constriction of the heart secondary to pericardial inflammation. Cardiovascular magnetic resonance (CMR) imaging is useful imaging modality for addressing the challenges of confirming this diagnosis. It can be used to exclude other causes of right heart failure, such as pulmonary hypertension or myocardial infarction, determine whether the pericardium is causing constriction and differentiate it from restrictive cardiomyopathy, which also causes impaired cardiac filling. Case Presentation: A 77-year-old man from a country with high incidence of tuberculosis presented with severe dyspnea. Echocardiography revealed a small left ventricle with normal systolic and mildly impaired diastolic function. Left heart catheterization revealed non-obstructive coronary disease, not felt contributory to the dyspnea. Anatomy imaging with cardiovascular magnetic resonance imaging (CMR) showed global, severely thickened pericardium. Short tau inversion recovery (STIR) sequences for detection of oedema/ inflammation showed increased signal intensity and free breathing sequences confirmed septal flattening on inspiration. Late gadolinium imaging confirmed enhancement in the pericardium, with all findings suggestive of pericardial inflammation and constriction. Conclusions: CMR with STIR sequences, free breathing sequences and late gadolinium imaging can prove extremely useful for diagnosing constrictive pericarditis.
Persistent Identifierhttp://hdl.handle.net/10722/221453
ISSN

 

DC FieldValueLanguage
dc.contributor.authorTse, G-
dc.contributor.authorAli, A-
dc.contributor.authorAlpendurada, F-
dc.contributor.authorPrasad, S-
dc.contributor.authorRaphael, CE-
dc.contributor.authorVassiliou, V-
dc.date.accessioned2015-11-19T07:11:08Z-
dc.date.available2015-11-19T07:11:08Z-
dc.date.issued2015-
dc.identifier.citationResearch in Cardiovascular Medicine, 2015, v. 4 n. 4, p. e29614-
dc.identifier.issn2251-9572-
dc.identifier.urihttp://hdl.handle.net/10722/221453-
dc.description.abstractIntroduction: Constrictive pericarditis is characterized by constriction of the heart secondary to pericardial inflammation. Cardiovascular magnetic resonance (CMR) imaging is useful imaging modality for addressing the challenges of confirming this diagnosis. It can be used to exclude other causes of right heart failure, such as pulmonary hypertension or myocardial infarction, determine whether the pericardium is causing constriction and differentiate it from restrictive cardiomyopathy, which also causes impaired cardiac filling. Case Presentation: A 77-year-old man from a country with high incidence of tuberculosis presented with severe dyspnea. Echocardiography revealed a small left ventricle with normal systolic and mildly impaired diastolic function. Left heart catheterization revealed non-obstructive coronary disease, not felt contributory to the dyspnea. Anatomy imaging with cardiovascular magnetic resonance imaging (CMR) showed global, severely thickened pericardium. Short tau inversion recovery (STIR) sequences for detection of oedema/ inflammation showed increased signal intensity and free breathing sequences confirmed septal flattening on inspiration. Late gadolinium imaging confirmed enhancement in the pericardium, with all findings suggestive of pericardial inflammation and constriction. Conclusions: CMR with STIR sequences, free breathing sequences and late gadolinium imaging can prove extremely useful for diagnosing constrictive pericarditis.-
dc.languageeng-
dc.publisherRajaie Cardiovascular, Medical & Research Center.-
dc.relation.ispartofResearch in Cardiovascular Medicine-
dc.subjectConstrictive Pericarditides-
dc.subjectPericardial Effusion-
dc.subjectTuberculosis-
dc.titleTuberculous Constrictive Pericarditis-
dc.typeArticle-
dc.identifier.emailTse, G: tseg@hku.hk-
dc.identifier.authorityTse, G=rp02073-
dc.description.naturelink_to_subscribed_fulltext-
dc.identifier.doi10.5812/cardiovascmed.29614-
dc.identifier.volume4-
dc.identifier.issue4-
dc.identifier.spagee29614-
dc.identifier.epagee29614-
dc.publisher.placeIran-

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