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Conference Paper: The role of the tumor suppressor gene THY1 in suppression of epithelial-mesenchymal transition (EMT) in nasopharyngeal carcinoma
Title | The role of the tumor suppressor gene THY1 in suppression of epithelial-mesenchymal transition (EMT) in nasopharyngeal carcinoma |
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Other Titles | The role of the tumor suppressor gene THY1 in suppression of EMT in NPC |
Authors | |
Keywords | Medical sciences Oncology |
Issue Date | 2014 |
Publisher | American Association for Cancer Research. The Journal's web site is located at http://cancerres.aacrjournals.org/ |
Citation | The 105th Annual Meeting of the American Association for Cancer Research (AACR 2014), San Diego, CA., 5-9 April 2014. In Cancer Research, 2014, v. 74 n. 19 suppl., abstract no. 1150 How to Cite? |
Abstract | BACKGROUND AND AIMS: THY1 has been successfully identified as a tumor suppressor gene (TSG) in nasopharyngeal carcinoma (NPC) in our laboratory. The frequency of down-regulated THY1 protein expression was found to be significantly associated with NPC lymph node metastases. However, direct functional evidence is still lacking for THY1 being anti-metastatic in NPC. Epithelial-Mesenchymal Transition (EMT) is well-known as a closely associated process for stem cells as well as tumor cells to invade to other part of the body. We aim to study the association of EMT with the THY1 in NPC. METHODS: Functional analysis of ... |
Description | Tumor Biology - Poster Presentations - Proffered Abstracts - Poster Presentations - Epithelial-Mesenchymal Transitions in Tumor Metastasis: abstract no. 1150 This journal suppl. entitled: Proceedings: AACR Annual Meeting 2014; April 5-9, 2014 ... |
Persistent Identifier | http://hdl.handle.net/10722/198638 |
ISSN | 2023 Impact Factor: 12.5 2023 SCImago Journal Rankings: 3.468 |
ISI Accession Number ID |
DC Field | Value | Language |
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dc.contributor.author | Lung, HL | - |
dc.contributor.author | Lung, ML | - |
dc.date.accessioned | 2014-07-07T08:26:46Z | - |
dc.date.available | 2014-07-07T08:26:46Z | - |
dc.date.issued | 2014 | - |
dc.identifier.citation | The 105th Annual Meeting of the American Association for Cancer Research (AACR 2014), San Diego, CA., 5-9 April 2014. In Cancer Research, 2014, v. 74 n. 19 suppl., abstract no. 1150 | - |
dc.identifier.issn | 0008-5472 | - |
dc.identifier.uri | http://hdl.handle.net/10722/198638 | - |
dc.description | Tumor Biology - Poster Presentations - Proffered Abstracts - Poster Presentations - Epithelial-Mesenchymal Transitions in Tumor Metastasis: abstract no. 1150 | - |
dc.description | This journal suppl. entitled: Proceedings: AACR Annual Meeting 2014; April 5-9, 2014 ... | - |
dc.description.abstract | BACKGROUND AND AIMS: THY1 has been successfully identified as a tumor suppressor gene (TSG) in nasopharyngeal carcinoma (NPC) in our laboratory. The frequency of down-regulated THY1 protein expression was found to be significantly associated with NPC lymph node metastases. However, direct functional evidence is still lacking for THY1 being anti-metastatic in NPC. Epithelial-Mesenchymal Transition (EMT) is well-known as a closely associated process for stem cells as well as tumor cells to invade to other part of the body. We aim to study the association of EMT with the THY1 in NPC. METHODS: Functional analysis of ... | - |
dc.language | eng | - |
dc.publisher | American Association for Cancer Research. The Journal's web site is located at http://cancerres.aacrjournals.org/ | - |
dc.relation.ispartof | Cancer Research | - |
dc.subject | Medical sciences | - |
dc.subject | Oncology | - |
dc.title | The role of the tumor suppressor gene THY1 in suppression of epithelial-mesenchymal transition (EMT) in nasopharyngeal carcinoma | - |
dc.title.alternative | The role of the tumor suppressor gene THY1 in suppression of EMT in NPC | - |
dc.type | Conference_Paper | - |
dc.identifier.email | Lung, HL: hllung2@hku.hk | - |
dc.identifier.email | Lung, ML: mlilung@hku.hk | - |
dc.identifier.authority | Lung, HL=rp00299 | - |
dc.identifier.authority | Lung, ML=rp00300 | - |
dc.description.nature | postprint | - |
dc.identifier.doi | 10.1158/1538-7445.AM2014-1150 | - |
dc.identifier.hkuros | 229926 | - |
dc.identifier.volume | 74 | - |
dc.identifier.issue | 19 suppl., abstract no. 1150 | - |
dc.identifier.isi | WOS:000349906900388 | - |
dc.publisher.place | United States | - |
dc.identifier.issnl | 0008-5472 | - |