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Article: Gene expression in cardiac hypertrophy

TitleGene expression in cardiac hypertrophy
Authors
Issue Date1992
Citation
Trends in Cardiovascular Medicine, 1992, v. 2 n. 5, p. 176-182 How to Cite?
AbstractCardiac hypertrophy due to hemodynamic overload is a harbinger of morbidity and mortality in humans. The development of hypertrophy involves both quantitative and qualitative changes in gene expression that are thought to produce an enlarged organ more capable of meeting its new functional requirements. The genes are normal, but the way in which they are regulated is modified. Analysis of these changes and the mechanisms involved are essential if we are to understand the role that hypertrophy plays in the pathogenesis of heart failure.
Persistent Identifierhttp://hdl.handle.net/10722/195229
ISSN
2015 Impact Factor: 3.075
2015 SCImago Journal Rankings: 1.323
ISI Accession Number ID

 

DC FieldValueLanguage
dc.contributor.authorBoheler, KR-
dc.contributor.authorSchwartz, K-
dc.date.accessioned2014-02-25T01:40:20Z-
dc.date.available2014-02-25T01:40:20Z-
dc.date.issued1992-
dc.identifier.citationTrends in Cardiovascular Medicine, 1992, v. 2 n. 5, p. 176-182-
dc.identifier.issn1050-1738-
dc.identifier.urihttp://hdl.handle.net/10722/195229-
dc.description.abstractCardiac hypertrophy due to hemodynamic overload is a harbinger of morbidity and mortality in humans. The development of hypertrophy involves both quantitative and qualitative changes in gene expression that are thought to produce an enlarged organ more capable of meeting its new functional requirements. The genes are normal, but the way in which they are regulated is modified. Analysis of these changes and the mechanisms involved are essential if we are to understand the role that hypertrophy plays in the pathogenesis of heart failure.-
dc.languageeng-
dc.relation.ispartofTrends in Cardiovascular Medicine-
dc.titleGene expression in cardiac hypertrophy-
dc.typeArticle-
dc.description.naturelink_to_subscribed_fulltext-
dc.identifier.doi10.1016/1050-1738(92)90046-U-
dc.identifier.pmid21239251-
dc.identifier.scopuseid_2-s2.0-0026760462-
dc.identifier.volume2-
dc.identifier.issue5-
dc.identifier.spage176-
dc.identifier.epage182-
dc.identifier.isiWOS:A1992JN05700003-

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