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Article: Effects of moxa smoke on monoamine neurotransmitters in SAMP8 mice

TitleEffects of moxa smoke on monoamine neurotransmitters in SAMP8 mice
Authors
Issue Date2013
PublisherHindawi Publishing Corporation. The Journal's web site is located at http://www.hindawi.com/journals/ecam/
Citation
Evidence-Based Complementary And Alternative Medicine, 2013, v. 2013 How to Cite?
AbstractObjectives. To investigate the anti-aging effects of moxa smoke on SAMP8 mice. Methods. Using 2 × 3 factorial design, exposure length (15 or 30 minutes daily), and concentration (low, 5-15 mg/m3; middle, 25-35 mg/m3; high, 85-95 mg/m3), 70 SAMP8 mice were randomly assigned, n = 10 /group, to a model group or one of six moxa smoke groups: L1, L2, M1, M2, H1, or H2. Ten SAMR1 mice were used as normal control. Mice in moxa smoke groups were exposed to moxa smoke at respective concentrations and exposure lengths; the model and normal control mice were not exposed. Cerebral 5-HT, DA, and NE levels were determined using ELISA. Results. Compared to normal control, the model group showed a significant decrease in 5-HT, DA, and NE. Compared to model group, 5-HT and NE were significantly higher in groups L2, M1, and M2 and DA was significantly so in L2 and M1. 5-HT, DA, and NE levels were the highest in group M 1 among moxa smoke groups. A marked exposure length × concentration interaction was observed for 5-HT, DA, and NE. Conclusion. Moxa smoke increases monoamine neurotransmitter levels, which varies according to concentration and exposure length. Our finding suggests that the middle concentration of moxa smoke for 15 minutes seems the most beneficial. © 2013 Huanfang Xu et al.
Persistent Identifierhttp://hdl.handle.net/10722/188667
ISSN
2015 Impact Factor: 1.931
2015 SCImago Journal Rankings: 0.615
ISI Accession Number ID
References

 

DC FieldValueLanguage
dc.contributor.authorXu, Hen_US
dc.contributor.authorZhao, Ben_US
dc.contributor.authorCui, Yen_US
dc.contributor.authorLim, MYen_US
dc.contributor.authorLiu, Pen_US
dc.contributor.authorHan, Len_US
dc.contributor.authorGuo, Hen_US
dc.contributor.authorLao, Len_US
dc.date.accessioned2013-09-03T04:10:58Z-
dc.date.available2013-09-03T04:10:58Z-
dc.date.issued2013en_US
dc.identifier.citationEvidence-Based Complementary And Alternative Medicine, 2013, v. 2013en_US
dc.identifier.issn1741-427Xen_US
dc.identifier.urihttp://hdl.handle.net/10722/188667-
dc.description.abstractObjectives. To investigate the anti-aging effects of moxa smoke on SAMP8 mice. Methods. Using 2 × 3 factorial design, exposure length (15 or 30 minutes daily), and concentration (low, 5-15 mg/m3; middle, 25-35 mg/m3; high, 85-95 mg/m3), 70 SAMP8 mice were randomly assigned, n = 10 /group, to a model group or one of six moxa smoke groups: L1, L2, M1, M2, H1, or H2. Ten SAMR1 mice were used as normal control. Mice in moxa smoke groups were exposed to moxa smoke at respective concentrations and exposure lengths; the model and normal control mice were not exposed. Cerebral 5-HT, DA, and NE levels were determined using ELISA. Results. Compared to normal control, the model group showed a significant decrease in 5-HT, DA, and NE. Compared to model group, 5-HT and NE were significantly higher in groups L2, M1, and M2 and DA was significantly so in L2 and M1. 5-HT, DA, and NE levels were the highest in group M 1 among moxa smoke groups. A marked exposure length × concentration interaction was observed for 5-HT, DA, and NE. Conclusion. Moxa smoke increases monoamine neurotransmitter levels, which varies according to concentration and exposure length. Our finding suggests that the middle concentration of moxa smoke for 15 minutes seems the most beneficial. © 2013 Huanfang Xu et al.en_US
dc.languageengen_US
dc.publisherHindawi Publishing Corporation. The Journal's web site is located at http://www.hindawi.com/journals/ecam/en_US
dc.relation.ispartofEvidence-based Complementary and Alternative Medicineen_US
dc.titleEffects of moxa smoke on monoamine neurotransmitters in SAMP8 miceen_US
dc.typeArticleen_US
dc.identifier.emailLao, L: lxlao1@hku.hken_US
dc.identifier.authorityLao, L=rp01784en_US
dc.description.naturelink_to_subscribed_fulltexten_US
dc.identifier.doi10.1155/2013/178067en_US
dc.identifier.scopuseid_2-s2.0-84878705266en_US
dc.relation.referenceshttp://www.scopus.com/mlt/select.url?eid=2-s2.0-84878705266&selection=ref&src=s&origin=recordpageen_US
dc.identifier.volume2013en_US
dc.identifier.isiWOS:000319569700001-
dc.publisher.placeUnited Kingdomen_US
dc.identifier.scopusauthoridXu, H=55286339400en_US
dc.identifier.scopusauthoridZhao, B=55469024300en_US
dc.identifier.scopusauthoridCui, Y=55758564900en_US
dc.identifier.scopusauthoridLim, MY=55757964500en_US
dc.identifier.scopusauthoridLiu, P=55207220800en_US
dc.identifier.scopusauthoridHan, L=55286839600en_US
dc.identifier.scopusauthoridGuo, H=7404403702en_US
dc.identifier.scopusauthoridLao, L=7005681883en_US

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