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Article: Aetiological factors in the genesis of pregnancy hydronephrosis

TitleAetiological factors in the genesis of pregnancy hydronephrosis
Authors
Issue Date1985
PublisherBlackwell Publishing Asia. The Journal's web site is located at http://www.blackwellpublishing.com/journals/ANZJOG
Citation
Australian And New Zealand Journal Of Obstetrics And Gynaecology, 1985, v. 25 n. 4, p. 248-251 How to Cite?
AbstractIn order to identify possible aetiological factors in the genesis of physiological hydronephrosis in pregnancy, the degree of pelvic-calyceal dilatation in 90 asymptomatic pregnant women was correlated with levels of plasma oestradiol, progesterone, 24-hour urinary oestriol, the site of the placenta, birth-weight of the fetus, and pelvic inlet measurements. A grading system based on maximum calyceal diameter was used; 90% of the patients were found to have at least mild dilatation on the right side. No correlation was demonstrated between the degree of hydronephrosis and the levels of oestradiol, progesterone and 24-hour urinary oestriol excretion. The birth-weight of the fetus and its relationship with the pelvic inlet measurements also did not correlate with the occurrence of hydronephrotic changes in the kidneys. The only significant positive finding was a higher incidence of moderate and severe hydronephrosis occurring in patients with a right-sided placenta than compared with the left (x2 = 4.77; p<0.05), although the sensitivity and specificity in predicting hydronephrosis from a right-sided placenta is low (53% and 66% respectively). Our results support the hypothesis of a mechanical aetiology in the genesis of pregnancy hydronephrosis, where vascular compression on the ureters may be an important contributory factor. Our study has also shown that urinary tract infection and reduction of creatinine clearance were not more common in patients with moderate or severe pelvic-calyceal dilatation.
Persistent Identifierhttp://hdl.handle.net/10722/184175
ISSN
2015 Impact Factor: 1.738
2015 SCImago Journal Rankings: 0.814
ISI Accession Number ID

 

DC FieldValueLanguage
dc.contributor.authorAu, KKLen_US
dc.contributor.authorWoo, JSKen_US
dc.contributor.authorTang, LCHen_US
dc.contributor.authorLiang, STen_US
dc.date.accessioned2013-06-25T03:01:01Z-
dc.date.available2013-06-25T03:01:01Z-
dc.date.issued1985en_US
dc.identifier.citationAustralian And New Zealand Journal Of Obstetrics And Gynaecology, 1985, v. 25 n. 4, p. 248-251en_US
dc.identifier.issn0004-8666en_US
dc.identifier.urihttp://hdl.handle.net/10722/184175-
dc.description.abstractIn order to identify possible aetiological factors in the genesis of physiological hydronephrosis in pregnancy, the degree of pelvic-calyceal dilatation in 90 asymptomatic pregnant women was correlated with levels of plasma oestradiol, progesterone, 24-hour urinary oestriol, the site of the placenta, birth-weight of the fetus, and pelvic inlet measurements. A grading system based on maximum calyceal diameter was used; 90% of the patients were found to have at least mild dilatation on the right side. No correlation was demonstrated between the degree of hydronephrosis and the levels of oestradiol, progesterone and 24-hour urinary oestriol excretion. The birth-weight of the fetus and its relationship with the pelvic inlet measurements also did not correlate with the occurrence of hydronephrotic changes in the kidneys. The only significant positive finding was a higher incidence of moderate and severe hydronephrosis occurring in patients with a right-sided placenta than compared with the left (x2 = 4.77; p<0.05), although the sensitivity and specificity in predicting hydronephrosis from a right-sided placenta is low (53% and 66% respectively). Our results support the hypothesis of a mechanical aetiology in the genesis of pregnancy hydronephrosis, where vascular compression on the ureters may be an important contributory factor. Our study has also shown that urinary tract infection and reduction of creatinine clearance were not more common in patients with moderate or severe pelvic-calyceal dilatation.en_US
dc.languageengen_US
dc.publisherBlackwell Publishing Asia. The Journal's web site is located at http://www.blackwellpublishing.com/journals/ANZJOGen_US
dc.relation.ispartofAustralian and New Zealand Journal of Obstetrics and Gynaecologyen_US
dc.subject.meshBacteriuria - Etiologyen_US
dc.subject.meshBirth Weighten_US
dc.subject.meshDilatation, Pathologic - Pathologyen_US
dc.subject.meshEstradiol - Blooden_US
dc.subject.meshEstriol - Urineen_US
dc.subject.meshFemaleen_US
dc.subject.meshHumansen_US
dc.subject.meshHydronephrosis - Etiology - Pathologyen_US
dc.subject.meshInfant, Newbornen_US
dc.subject.meshKidney Calices - Pathologyen_US
dc.subject.meshPregnancyen_US
dc.subject.meshPregnancy Complications - Etiology - Pathologyen_US
dc.subject.meshPregnancy Complications, Infectious - Etiologyen_US
dc.subject.meshProgesterone - Blooden_US
dc.subject.meshUreteral Obstruction - Etiologyen_US
dc.titleAetiological factors in the genesis of pregnancy hydronephrosisen_US
dc.typeArticleen_US
dc.identifier.emailTang, LCH: lchtang@hku.hken_US
dc.identifier.authorityTang, LCH=rp01756en_US
dc.description.naturelink_to_subscribed_fulltexten_US
dc.identifier.doi10.1111/j.1479-828X.1985.tb00737.x-
dc.identifier.pmid3869447-
dc.identifier.scopuseid_2-s2.0-0022400305en_US
dc.identifier.volume25en_US
dc.identifier.issue4en_US
dc.identifier.spage248en_US
dc.identifier.epage251en_US
dc.identifier.isiWOS:A1985AXU9200003-
dc.publisher.placeAustraliaen_US
dc.identifier.scopusauthoridAu, KKL=7006641980en_US
dc.identifier.scopusauthoridWoo, JSK=7401753072en_US
dc.identifier.scopusauthoridTang, LCH=7402081111en_US
dc.identifier.scopusauthoridLiang, ST=7402146735en_US

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