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Conference Paper: PR prolongation strongly predicts new-onset myocardial infarction, ischaemic stroke, heart failure, and cardiovascular death in coronary patients or risk equivalent: a 5-year clinical-patholophysiological study

TitlePR prolongation strongly predicts new-onset myocardial infarction, ischaemic stroke, heart failure, and cardiovascular death in coronary patients or risk equivalent: a 5-year clinical-patholophysiological study
Authors
KeywordsMedical sciences
Issue Date2013
PublisherHong Kong Academy of Medicine Press. The Journal's web site is located at http://www.hkmj.org.hk
Citation
The 18th Medical Research Conference (MRC 2013); The University of Hong Kong, Hong Kong, 12 January 2013. In Hong Kong Medical Journal, 2013, v. 19 suppl. 1, p. 13, abstract no. 7 How to Cite?
AbstractOBJECTIVES: To investigate the role of PR prolongation in new-onset adverse cardiovascular (CV) events in high-risk CV patients, and the underlying pathophysiological mechanisms in terms of vascular phenotypes. METHODS: We prospectively followed up 597 high-risk CV out-patients (mean age 66±11 years; male 67%; coronary disease 55%, stroke 22%, diabetes 52%) for new-onset ischaemic stroke, myocardial infarction (MI), congestive heart failure (CHF), and CV death. Vascular phenotypes were asses…
DescriptionOral Presentation
Persistent Identifierhttp://hdl.handle.net/10722/181769
ISSN
2015 Impact Factor: 0.887
2015 SCImago Journal Rankings: 0.279

 

DC FieldValueLanguage
dc.contributor.authorChan, YHen_US
dc.contributor.authorLau, KKen_US
dc.contributor.authorYiu, KHen_US
dc.contributor.authorYiu, YFen_US
dc.contributor.authorLi, SWen_US
dc.contributor.authorLam, THen_US
dc.contributor.authorLau, CPen_US
dc.contributor.authorSiu, CWen_US
dc.contributor.authorTse, HFen_US
dc.date.accessioned2013-03-19T03:56:55Z-
dc.date.available2013-03-19T03:56:55Z-
dc.date.issued2013en_US
dc.identifier.citationThe 18th Medical Research Conference (MRC 2013); The University of Hong Kong, Hong Kong, 12 January 2013. In Hong Kong Medical Journal, 2013, v. 19 suppl. 1, p. 13, abstract no. 7en_US
dc.identifier.issn1024-2708-
dc.identifier.urihttp://hdl.handle.net/10722/181769-
dc.descriptionOral Presentation-
dc.description.abstractOBJECTIVES: To investigate the role of PR prolongation in new-onset adverse cardiovascular (CV) events in high-risk CV patients, and the underlying pathophysiological mechanisms in terms of vascular phenotypes. METHODS: We prospectively followed up 597 high-risk CV out-patients (mean age 66±11 years; male 67%; coronary disease 55%, stroke 22%, diabetes 52%) for new-onset ischaemic stroke, myocardial infarction (MI), congestive heart failure (CHF), and CV death. Vascular phenotypes were asses…-
dc.languageengen_US
dc.publisherHong Kong Academy of Medicine Press. The Journal's web site is located at http://www.hkmj.org.hk-
dc.relation.ispartofHong Kong Medical Journalen_US
dc.rightsHong Kong Medical Journal. Copyright © Hong Kong Academy of Medicine Press.-
dc.rightsCreative Commons: Attribution 3.0 Hong Kong License-
dc.subjectMedical sciences-
dc.titlePR prolongation strongly predicts new-onset myocardial infarction, ischaemic stroke, heart failure, and cardiovascular death in coronary patients or risk equivalent: a 5-year clinical-patholophysiological studyen_US
dc.typeConference_Paperen_US
dc.identifier.emailChan, YH: chanwill@hku.hken_US
dc.identifier.emailLau, KK: gkklau@hku.hken_US
dc.identifier.emailYiu, KH: khkyiu@hku.hken_US
dc.identifier.emailLi, SW: lswli@hkucc.hku.hken_US
dc.identifier.emailLam, TH: hrmrlth@hkucc.hku.hken_US
dc.identifier.emailLau, CP: cplau@hku.hken_US
dc.identifier.emailSiu, CW: cwdsiu@hkucc.hku.hken_US
dc.identifier.emailTse, HF: hftse@hkucc.hku.hken_US
dc.identifier.authorityChan, YH=rp01313en_US
dc.identifier.authorityLau, KK=rp01499en_US
dc.identifier.authorityYiu, KH=rp01490en_US
dc.identifier.authorityLam, TH=rp00326en_US
dc.identifier.authoritySiu, CW=rp00534en_US
dc.identifier.authorityTse, HF=rp00428en_US
dc.description.naturepublished_or_final_version-
dc.identifier.hkuros213418en_US
dc.identifier.volume19-
dc.identifier.issuesuppl. 1-
dc.identifier.spage13, abstract no. 7-
dc.identifier.epage13, abstract no. 7-
dc.publisher.placeHong Kong-
dc.customcontrol.immutablesml 130417-

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