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- Publisher Website: 10.1016/0022-3956(92)90029-N
- Scopus: eid_2-s2.0-0027103179
- PMID: 1491349
- WOS: WOS:A1992KH01900002
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Conference Paper: Genes, viruses and neurodevelopmental schizophrenia
Title | Genes, viruses and neurodevelopmental schizophrenia |
---|---|
Authors | |
Issue Date | 1992 |
Publisher | Pergamon. The Journal's web site is located at http://www.elsevier.com/locate/jpsychires |
Citation | Journal of Psychiatric Research, 1992, v. 26 n. 4, p. 225-235 How to Cite? |
Abstract | Recent neuroimaging and neuropathological studies suggest a developmental origin for schizophrenia. Some cases may, therefore, be caused by a genetic defect in the specification of brain development. Early environmental hazards such as obstetric complications, and maternal exposure during pregnancy to influenza epidemics, have also been found to increase the risk of later schizophrenia. The relationship between the prevalence of influenza and birth date has been found more consistently for female than male schizophrenics. Female schizophrenia is also associated with a higher risk of schizophrenia in first degree relatives. This raises the question of whether part of the genetic predisposition to schizophrenia may comprise an abnormal reaction to maternal influenza. |
Persistent Identifier | http://hdl.handle.net/10722/176088 |
ISSN | 2023 Impact Factor: 3.7 2023 SCImago Journal Rankings: 1.553 |
ISI Accession Number ID |
DC Field | Value | Language |
---|---|---|
dc.contributor.author | Murray, RM | en_US |
dc.contributor.author | Jones, P | en_US |
dc.contributor.author | O'callaghan, E | en_US |
dc.contributor.author | Takei, N | en_US |
dc.contributor.author | Sham, P | en_US |
dc.date.accessioned | 2012-11-26T09:05:32Z | - |
dc.date.available | 2012-11-26T09:05:32Z | - |
dc.date.issued | 1992 | en_US |
dc.identifier.citation | Journal of Psychiatric Research, 1992, v. 26 n. 4, p. 225-235 | en_US |
dc.identifier.issn | 0022-3956 | en_US |
dc.identifier.uri | http://hdl.handle.net/10722/176088 | - |
dc.description.abstract | Recent neuroimaging and neuropathological studies suggest a developmental origin for schizophrenia. Some cases may, therefore, be caused by a genetic defect in the specification of brain development. Early environmental hazards such as obstetric complications, and maternal exposure during pregnancy to influenza epidemics, have also been found to increase the risk of later schizophrenia. The relationship between the prevalence of influenza and birth date has been found more consistently for female than male schizophrenics. Female schizophrenia is also associated with a higher risk of schizophrenia in first degree relatives. This raises the question of whether part of the genetic predisposition to schizophrenia may comprise an abnormal reaction to maternal influenza. | en_US |
dc.language | eng | en_US |
dc.publisher | Pergamon. The Journal's web site is located at http://www.elsevier.com/locate/jpsychires | en_US |
dc.relation.ispartof | Journal of Psychiatric Research | en_US |
dc.subject.mesh | Brain Damage, Chronic - Genetics - Psychology | en_US |
dc.subject.mesh | Cerebral Cortex - Pathology | en_US |
dc.subject.mesh | Cerebral Ventricles - Pathology | en_US |
dc.subject.mesh | Delirium, Dementia, Amnestic, Cognitive Disorders - Genetics - Psychology | en_US |
dc.subject.mesh | Female | en_US |
dc.subject.mesh | Humans | en_US |
dc.subject.mesh | Orthomyxoviridae - Genetics | en_US |
dc.subject.mesh | Personality Development | en_US |
dc.subject.mesh | Pregnancy | en_US |
dc.subject.mesh | Prenatal Exposure Delayed Effects | en_US |
dc.subject.mesh | Risk Factors | en_US |
dc.subject.mesh | Schizophrenia - Genetics | en_US |
dc.subject.mesh | Schizophrenic Psychology | en_US |
dc.subject.mesh | Social Environment | en_US |
dc.title | Genes, viruses and neurodevelopmental schizophrenia | en_US |
dc.type | Conference_Paper | en_US |
dc.identifier.email | Sham, P: pcsham@hku.hk | en_US |
dc.identifier.authority | Sham, P=rp00459 | en_US |
dc.description.nature | link_to_subscribed_fulltext | en_US |
dc.identifier.doi | 10.1016/0022-3956(92)90029-N | en_US |
dc.identifier.pmid | 1491349 | - |
dc.identifier.scopus | eid_2-s2.0-0027103179 | en_US |
dc.identifier.volume | 26 | en_US |
dc.identifier.issue | 4 | en_US |
dc.identifier.spage | 225 | en_US |
dc.identifier.epage | 235 | en_US |
dc.identifier.isi | WOS:A1992KH01900002 | - |
dc.publisher.place | United Kingdom | en_US |
dc.identifier.scopusauthorid | Murray, RM=35406239400 | en_US |
dc.identifier.scopusauthorid | Jones, P=36078972900 | en_US |
dc.identifier.scopusauthorid | O'Callaghan, E=7005694363 | en_US |
dc.identifier.scopusauthorid | Takei, N=35874982900 | en_US |
dc.identifier.scopusauthorid | Sham, P=34573429300 | en_US |
dc.customcontrol.immutable | sml 170602 amended | - |
dc.identifier.issnl | 0022-3956 | - |