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Conference Paper: Venous disease: From pathophysiology to quality of life

TitleVenous disease: From pathophysiology to quality of life
Authors
Issue Date1997
PublisherSage Publications, Inc. The Journal's web site is located at http://ang.sagepub.com
Citation
Angiology, 1997, v. 48 n. 7, p. 559-567 How to Cite?
AbstractVenous insufficiency is a multifactorial pathology that has an important impact on the quality of life of the patients. The primary factor of venous disease is an abnormal wall distensibility, which seems to be correlated with genetic factors. Facilitating factors include hormonal impregnation and prolonged hydrostatic load, particularly under conditions where the control of the sympathetic nervous system is reduced by an increase in local temperature. The resulting valvular incompetence, combined with the augmented hydrostatic load, leads to varicosis and venous stasis. The ensuing tissue hypoxia and local edema favor inflammation and infection, which ultimately favor the occurrence of ulcers. The available data on the impact of the disease suggest a relation between the physiopathological phenomena and some parameters of health-related quality of life.
Persistent Identifierhttp://hdl.handle.net/10722/173529
ISSN
2015 Impact Factor: 2.931
2015 SCImago Journal Rankings: 0.598
ISI Accession Number ID
References

 

DC FieldValueLanguage
dc.contributor.authorVanhoutte, PMen_US
dc.contributor.authorCorcaud, Sen_US
dc.contributor.authorDe Montrion, Cen_US
dc.date.accessioned2012-10-30T06:32:31Z-
dc.date.available2012-10-30T06:32:31Z-
dc.date.issued1997en_US
dc.identifier.citationAngiology, 1997, v. 48 n. 7, p. 559-567en_US
dc.identifier.issn0003-3197en_US
dc.identifier.urihttp://hdl.handle.net/10722/173529-
dc.description.abstractVenous insufficiency is a multifactorial pathology that has an important impact on the quality of life of the patients. The primary factor of venous disease is an abnormal wall distensibility, which seems to be correlated with genetic factors. Facilitating factors include hormonal impregnation and prolonged hydrostatic load, particularly under conditions where the control of the sympathetic nervous system is reduced by an increase in local temperature. The resulting valvular incompetence, combined with the augmented hydrostatic load, leads to varicosis and venous stasis. The ensuing tissue hypoxia and local edema favor inflammation and infection, which ultimately favor the occurrence of ulcers. The available data on the impact of the disease suggest a relation between the physiopathological phenomena and some parameters of health-related quality of life.en_US
dc.languageengen_US
dc.publisherSage Publications, Inc. The Journal's web site is located at http://ang.sagepub.comen_US
dc.relation.ispartofAngiologyen_US
dc.subject.meshBody Temperature Regulationen_US
dc.subject.meshHumansen_US
dc.subject.meshHydrostatic Pressureen_US
dc.subject.meshMuscle Contraction - Physiologyen_US
dc.subject.meshPliabilityen_US
dc.subject.meshQuality Of Lifeen_US
dc.subject.meshRisk Factorsen_US
dc.subject.meshSex Factorsen_US
dc.subject.meshVaricose Veins - Physiopathologyen_US
dc.subject.meshVeins - Physiopathologyen_US
dc.subject.meshVenous Insufficiency - Epidemiology - Physiopathologyen_US
dc.titleVenous disease: From pathophysiology to quality of lifeen_US
dc.typeConference_Paperen_US
dc.identifier.emailVanhoutte, PM:vanhoutt@hku.hken_US
dc.identifier.authorityVanhoutte, PM=rp00238en_US
dc.description.naturelink_to_subscribed_fulltexten_US
dc.identifier.doi10.1177/000331979704800702-
dc.identifier.pmid9242153-
dc.identifier.scopuseid_2-s2.0-0030840024en_US
dc.relation.referenceshttp://www.scopus.com/mlt/select.url?eid=2-s2.0-0030840024&selection=ref&src=s&origin=recordpageen_US
dc.identifier.volume48en_US
dc.identifier.issue7en_US
dc.identifier.spage559en_US
dc.identifier.epage567en_US
dc.identifier.isiWOS:A1997XM63700002-
dc.publisher.placeUnited Statesen_US
dc.identifier.scopusauthoridVanhoutte, PM=7202304247en_US
dc.identifier.scopusauthoridCorcaud, S=6507592103en_US
dc.identifier.scopusauthoridDe Montrion, C=22943373000en_US

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