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Conference Paper: Nitric oxide and vascular smooth muscle homeostasis | LE MONOXYDE D'AZOTE ET L'HOMEOSTASIE DU MUSCLE LISSE VASCULAIRE

TitleNitric oxide and vascular smooth muscle homeostasis | LE MONOXYDE D'AZOTE ET L'HOMEOSTASIE DU MUSCLE LISSE VASCULAIRE
Authors
Issue Date1993
Citation
Archives Des Maladies Du Coeur Et Des Vaisseaux, 1993, v. 86 SPEC. ISS. 1, p. 83-89 How to Cite?
AbstractNitric oxide is synthesised from an amino acid, L-arginine, by a family of enzymes called nitric oxide (NO) synthase, by most cells in the vessel wall. In healthy vessels, the production of NO is due to the constitutive and calcium dependent NO synthase present in the endothelial cells. On the other hand, when the vascular system is diseased and defense mechanisms are activated, the mediators of inflammatory and immunitary reactions induce and NO synthase non-responsive to calcium which produces large quantities of NO in most of the cells of the vessel wall. Nitric oxide is a liposoluble radical with a short half-life. It plays a central role in the regulation of the motricity and proliferation of blood vessels and in the interaction of the blood cells with the vessel wall. An inadequate production of nitric oxide could play a role in many vascular diseases such as hypertension, atherosclerosis, restenosis or vascular hyporeactivity associated with septicaemic shock.
Persistent Identifierhttp://hdl.handle.net/10722/173514
ISSN
2009 Impact Factor: 0.398
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DC FieldValueLanguage
dc.contributor.authorSchini, VBen_US
dc.contributor.authorVanhoutte, PMen_US
dc.date.accessioned2012-10-30T06:32:27Z-
dc.date.available2012-10-30T06:32:27Z-
dc.date.issued1993en_US
dc.identifier.citationArchives Des Maladies Du Coeur Et Des Vaisseaux, 1993, v. 86 SPEC. ISS. 1, p. 83-89en_US
dc.identifier.issn0003-9683en_US
dc.identifier.urihttp://hdl.handle.net/10722/173514-
dc.description.abstractNitric oxide is synthesised from an amino acid, L-arginine, by a family of enzymes called nitric oxide (NO) synthase, by most cells in the vessel wall. In healthy vessels, the production of NO is due to the constitutive and calcium dependent NO synthase present in the endothelial cells. On the other hand, when the vascular system is diseased and defense mechanisms are activated, the mediators of inflammatory and immunitary reactions induce and NO synthase non-responsive to calcium which produces large quantities of NO in most of the cells of the vessel wall. Nitric oxide is a liposoluble radical with a short half-life. It plays a central role in the regulation of the motricity and proliferation of blood vessels and in the interaction of the blood cells with the vessel wall. An inadequate production of nitric oxide could play a role in many vascular diseases such as hypertension, atherosclerosis, restenosis or vascular hyporeactivity associated with septicaemic shock.en_US
dc.languageengen_US
dc.relation.ispartofArchives des Maladies du Coeur et des Vaisseauxen_US
dc.subject.meshAmino Acid Oxidoreductases - Metabolismen_US
dc.subject.meshBlood Platelets - Enzymologyen_US
dc.subject.meshEndothelium, Vascular - Metabolismen_US
dc.subject.meshHumansen_US
dc.subject.meshMacrophages - Enzymologyen_US
dc.subject.meshMuscle, Smooth, Vascular - Metabolismen_US
dc.subject.meshNitric Oxide - Biosynthesis - Metabolism - Physiologyen_US
dc.subject.meshNitric Oxide Synthaseen_US
dc.titleNitric oxide and vascular smooth muscle homeostasis | LE MONOXYDE D'AZOTE ET L'HOMEOSTASIE DU MUSCLE LISSE VASCULAIREen_US
dc.typeConference_Paperen_US
dc.identifier.emailVanhoutte, PM:vanhoutt@hku.hken_US
dc.identifier.authorityVanhoutte, PM=rp00238en_US
dc.description.naturelink_to_subscribed_fulltexten_US
dc.identifier.pmid7692833-
dc.identifier.scopuseid_2-s2.0-0027500256en_US
dc.identifier.volume86en_US
dc.identifier.issueSPEC. ISS. 1en_US
dc.identifier.spage83en_US
dc.identifier.epage89en_US
dc.identifier.isiWOS:A1993KK57000011-
dc.identifier.scopusauthoridSchini, VB=7004113565en_US
dc.identifier.scopusauthoridVanhoutte, PM=7202304247en_US

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