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Conference Paper: Novel Ca2+ signalling mechanisms in vascular myocytes: Symposium overview

TitleNovel Ca2+ signalling mechanisms in vascular myocytes: Symposium overview
Authors
Issue Date2003
Citation
Acta Physiologica Scandinavica, 2003, v. 179 n. 4, p. 339-352 How to Cite?
AbstractThis commentary presents the proceedings of the symposium sponsored by Cardiovascular Section of American Physiological Society in San Diego, CA on 12 April 2003. The major focus of this symposium was on the actions and physiological relevance of several novel Ca2+ signalling mechanisms in vascular smooth muscle (VSM) cells. Five important topics were presented in this symposium including the discovery and roles of cyclic ADP-ribose (cADPR) and nicotinic acid adenine dinucleotide phosphate (NAADP) in mediating Ca 2+ release, Ca2+ sparks and activation of plasma membrane KCa channels in VSM cells, the role of cADPR-mediated activation of ryanodine receptors in the control of vascular tone, the role of [Ca 2+]i in mechano-transduction in the arterioles, and interactions of mitochondrial Ca2+ release and SR Ca2+ mobilization. The purpose of this symposium was to promote discussions and exchange of ideas between scientists with interests in Ca2+ signalling mechanisms and those with interests in vascular physiology and pharmacology. The cross-fertilization of ideas is expected to greatly advance our understanding of the physiological and pharmacological relevance of these new Ca2+ signalling mechanisms.
Persistent Identifierhttp://hdl.handle.net/10722/173446
ISSN
2007 Impact Factor: 2.554
ISI Accession Number ID
References

 

DC FieldValueLanguage
dc.contributor.authorLi, PLen_US
dc.contributor.authorLee, HCen_US
dc.contributor.authorNelson, MTen_US
dc.contributor.authorMeininger, GAen_US
dc.contributor.authorVan Breemen, Cen_US
dc.date.accessioned2012-10-30T06:31:28Z-
dc.date.available2012-10-30T06:31:28Z-
dc.date.issued2003en_US
dc.identifier.citationActa Physiologica Scandinavica, 2003, v. 179 n. 4, p. 339-352en_US
dc.identifier.issn0001-6772en_US
dc.identifier.urihttp://hdl.handle.net/10722/173446-
dc.description.abstractThis commentary presents the proceedings of the symposium sponsored by Cardiovascular Section of American Physiological Society in San Diego, CA on 12 April 2003. The major focus of this symposium was on the actions and physiological relevance of several novel Ca2+ signalling mechanisms in vascular smooth muscle (VSM) cells. Five important topics were presented in this symposium including the discovery and roles of cyclic ADP-ribose (cADPR) and nicotinic acid adenine dinucleotide phosphate (NAADP) in mediating Ca 2+ release, Ca2+ sparks and activation of plasma membrane KCa channels in VSM cells, the role of cADPR-mediated activation of ryanodine receptors in the control of vascular tone, the role of [Ca 2+]i in mechano-transduction in the arterioles, and interactions of mitochondrial Ca2+ release and SR Ca2+ mobilization. The purpose of this symposium was to promote discussions and exchange of ideas between scientists with interests in Ca2+ signalling mechanisms and those with interests in vascular physiology and pharmacology. The cross-fertilization of ideas is expected to greatly advance our understanding of the physiological and pharmacological relevance of these new Ca2+ signalling mechanisms.en_US
dc.languageengen_US
dc.relation.ispartofActa Physiologica Scandinavicaen_US
dc.subject.meshArterioles - Metabolismen_US
dc.subject.meshBiological Transporten_US
dc.subject.meshCalcium - Metabolismen_US
dc.subject.meshCalcium Signaling - Physiologyen_US
dc.subject.meshCyclic Adp-Ribose - Metabolismen_US
dc.subject.meshHumansen_US
dc.subject.meshIntegrins - Metabolismen_US
dc.subject.meshMitochondria - Metabolismen_US
dc.subject.meshMuscle, Smooth, Vascular - Metabolismen_US
dc.subject.meshNadp - Analogs & Derivatives - Metabolismen_US
dc.subject.meshPotassium Channels - Metabolismen_US
dc.titleNovel Ca2+ signalling mechanisms in vascular myocytes: Symposium overviewen_US
dc.typeConference_Paperen_US
dc.identifier.emailLee, HC:leehc@hku.hken_US
dc.identifier.authorityLee, HC=rp00545en_US
dc.description.naturelink_to_subscribed_fulltexten_US
dc.identifier.doi10.1046/j.0001-6772.2003.01216.xen_US
dc.identifier.pmid14656371-
dc.identifier.scopuseid_2-s2.0-0346249751en_US
dc.relation.referenceshttp://www.scopus.com/mlt/select.url?eid=2-s2.0-0346249751&selection=ref&src=s&origin=recordpageen_US
dc.identifier.volume179en_US
dc.identifier.issue4en_US
dc.identifier.spage339en_US
dc.identifier.epage352en_US
dc.identifier.isiWOS:000187366200005-
dc.identifier.scopusauthoridLi, PL=7404773769en_US
dc.identifier.scopusauthoridLee, HC=26642959100en_US
dc.identifier.scopusauthoridNelson, MT=7403461703en_US
dc.identifier.scopusauthoridMeininger, GA=7005594512en_US
dc.identifier.scopusauthoridVan Breemen, C=35571620500en_US

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