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Article: Snaring of the target vessel in less invasive bypass operations does not cause endothelial dysfunction

TitleSnaring of the target vessel in less invasive bypass operations does not cause endothelial dysfunction
Authors
Issue Date1997
PublisherElsevier Inc. The Journal's web site is located at http://www.elsevier.com/locate/athoracsur
Citation
Annals Of Thoracic Surgery, 1997, v. 63 n. 3, p. 751-755 How to Cite?
AbstractBackground. Minimally invasive coronary artery bypass grafting aims to achieve less patient discomfort and a more rapid return to active life. Most approaches have used maintenance of the beating heart and control of the target coronary vessel by different hemostatic devices. The purpose of this study was to assess the effects of commonly used coronary artery snares and of the occlusion of the coronary vessel necessary for minimally invasive coronary artery operations on coronary endothelial function. Methods. Coronary artery bypass grafting with an internal mammary artery to left anterior descending artery anastomosis was performed in a porcine model with a 30-minute period of ischemia and a subsequent 30-minute period of reperfusion, using snares on either side of the anastomotic site to achieve hemostasis of the operative field. Endothelium-dependent relaxation to serotonin was studied in conventional organ chamber experiments with rings taken from the left anterior descending artery at the proximal snare site, the anastomotic site in the segment that underwent the ischemia reperfusion cycle, the distal snare site, and at a control segment. Responses to potassium chloride and bradykinin were also compared. Results. There were no significant differences in endothelium-dependent relaxation values among the four sites studied. Conclusions. These results confirm that snaring of the coronary artery for achieving hemostasis at the anastomotic site when performing coronary artery bypass grafting on the beating heart does not cause endothelial dysfunction.
Persistent Identifierhttp://hdl.handle.net/10722/171201
ISSN
2015 Impact Factor: 2.975
2015 SCImago Journal Rankings: 1.490
ISI Accession Number ID
References

 

DC FieldValueLanguage
dc.contributor.authorPerrault, LPen_US
dc.contributor.authorMenasché, Pen_US
dc.contributor.authorBidouard, JPen_US
dc.contributor.authorJacquemin, Cen_US
dc.contributor.authorVilleneuve, Nen_US
dc.contributor.authorVilaine, JPen_US
dc.contributor.authorVanhoutte, PMen_US
dc.date.accessioned2012-10-30T06:12:40Z-
dc.date.available2012-10-30T06:12:40Z-
dc.date.issued1997en_US
dc.identifier.citationAnnals Of Thoracic Surgery, 1997, v. 63 n. 3, p. 751-755en_US
dc.identifier.issn0003-4975en_US
dc.identifier.urihttp://hdl.handle.net/10722/171201-
dc.description.abstractBackground. Minimally invasive coronary artery bypass grafting aims to achieve less patient discomfort and a more rapid return to active life. Most approaches have used maintenance of the beating heart and control of the target coronary vessel by different hemostatic devices. The purpose of this study was to assess the effects of commonly used coronary artery snares and of the occlusion of the coronary vessel necessary for minimally invasive coronary artery operations on coronary endothelial function. Methods. Coronary artery bypass grafting with an internal mammary artery to left anterior descending artery anastomosis was performed in a porcine model with a 30-minute period of ischemia and a subsequent 30-minute period of reperfusion, using snares on either side of the anastomotic site to achieve hemostasis of the operative field. Endothelium-dependent relaxation to serotonin was studied in conventional organ chamber experiments with rings taken from the left anterior descending artery at the proximal snare site, the anastomotic site in the segment that underwent the ischemia reperfusion cycle, the distal snare site, and at a control segment. Responses to potassium chloride and bradykinin were also compared. Results. There were no significant differences in endothelium-dependent relaxation values among the four sites studied. Conclusions. These results confirm that snaring of the coronary artery for achieving hemostasis at the anastomotic site when performing coronary artery bypass grafting on the beating heart does not cause endothelial dysfunction.en_US
dc.languageengen_US
dc.publisherElsevier Inc. The Journal's web site is located at http://www.elsevier.com/locate/athoracsuren_US
dc.relation.ispartofAnnals of Thoracic Surgeryen_US
dc.subject.meshAnimalsen_US
dc.subject.meshCoronary Vessels - Physiology - Surgeryen_US
dc.subject.meshEndothelium, Vascular - Physiologyen_US
dc.subject.meshFemaleen_US
dc.subject.meshHemostasis, Surgical - Adverse Effects - Instrumentation - Methodsen_US
dc.subject.meshInternal Mammary-Coronary Artery Anastomosis - Adverse Effects - Methodsen_US
dc.subject.meshIntraoperative Careen_US
dc.subject.meshMaleen_US
dc.subject.meshMyocardial Reperfusion Injury - Physiopathologyen_US
dc.subject.meshSwineen_US
dc.titleSnaring of the target vessel in less invasive bypass operations does not cause endothelial dysfunctionen_US
dc.typeArticleen_US
dc.identifier.emailVanhoutte, PM:vanhoutt@hku.hken_US
dc.identifier.authorityVanhoutte, PM=rp00238en_US
dc.description.naturelink_to_subscribed_fulltexten_US
dc.identifier.doi10.1016/S0003-4975(96)01118-6en_US
dc.identifier.pmid9066396-
dc.identifier.scopuseid_2-s2.0-0030933335en_US
dc.relation.referenceshttp://www.scopus.com/mlt/select.url?eid=2-s2.0-0030933335&selection=ref&src=s&origin=recordpageen_US
dc.identifier.volume63en_US
dc.identifier.issue3en_US
dc.identifier.spage751en_US
dc.identifier.epage755en_US
dc.identifier.isiWOS:A1997WM32600030-
dc.publisher.placeUnited Statesen_US
dc.identifier.scopusauthoridPerrault, LP=7004370552en_US
dc.identifier.scopusauthoridMenasché, P=7102635294en_US
dc.identifier.scopusauthoridBidouard, JP=6601955808en_US
dc.identifier.scopusauthoridJacquemin, C=7004759803en_US
dc.identifier.scopusauthoridVilleneuve, N=7003458215en_US
dc.identifier.scopusauthoridVilaine, JP=7004617134en_US
dc.identifier.scopusauthoridVanhoutte, PM=7202304247en_US

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