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Article: Innate immunity and systemic lupus erythematosus
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TitleInnate immunity and systemic lupus erythematosus
 
AuthorsJin, O2
Sun, L1
Kavikondala, S2
Lau, CS2
 
KeywordsDisease Aetiology And Pathogenesis
Innate Immunity
Lymphocyte Activation
Systemic Lupus Erythematous
 
Issue Date2006
 
PublisherBlackwell Publishing Asia. The Journal's web site is located at http://www.blackwellpublishing.com/journals/APL
 
CitationAplar Journal Of Rheumatology, 2006, v. 9 n. 4, p. 359-364 [How to Cite?]
DOI: http://dx.doi.org/10.1111/j.1479-8077.2006.00229.x
 
AbstractInnate immunity is the first-line host defence against pathogens and damaged host cells, and the major cellular components are phagocytes such as monocytes/macrophages, polymorphonuclear cells and dendritic cells. Systemic lupus erythematosus (SLE) is an autoimmune disease characterized by the loss of tolerance to self-antigens, the source of which has been suggested to be apoptotic cells. In this article, we will review studies on apoptosis in SLE and discuss the contribution of innate immunity abnormalities in the development of this condition. © 2006 Asia Pacific League of Associations for Rheumatology.
 
ISSN0219-0494
 
DOIhttp://dx.doi.org/10.1111/j.1479-8077.2006.00229.x
 
ReferencesReferences in Scopus
 
DC FieldValue
dc.contributor.authorJin, O
 
dc.contributor.authorSun, L
 
dc.contributor.authorKavikondala, S
 
dc.contributor.authorLau, CS
 
dc.date.accessioned2012-09-05T05:26:54Z
 
dc.date.available2012-09-05T05:26:54Z
 
dc.date.issued2006
 
dc.description.abstractInnate immunity is the first-line host defence against pathogens and damaged host cells, and the major cellular components are phagocytes such as monocytes/macrophages, polymorphonuclear cells and dendritic cells. Systemic lupus erythematosus (SLE) is an autoimmune disease characterized by the loss of tolerance to self-antigens, the source of which has been suggested to be apoptotic cells. In this article, we will review studies on apoptosis in SLE and discuss the contribution of innate immunity abnormalities in the development of this condition. © 2006 Asia Pacific League of Associations for Rheumatology.
 
dc.description.naturelink_to_subscribed_fulltext
 
dc.identifier.citationAplar Journal Of Rheumatology, 2006, v. 9 n. 4, p. 359-364 [How to Cite?]
DOI: http://dx.doi.org/10.1111/j.1479-8077.2006.00229.x
 
dc.identifier.doihttp://dx.doi.org/10.1111/j.1479-8077.2006.00229.x
 
dc.identifier.epage364
 
dc.identifier.issn0219-0494
 
dc.identifier.issue4
 
dc.identifier.scopuseid_2-s2.0-33750991860
 
dc.identifier.spage359
 
dc.identifier.urihttp://hdl.handle.net/10722/163044
 
dc.identifier.volume9
 
dc.languageeng
 
dc.publisherBlackwell Publishing Asia. The Journal's web site is located at http://www.blackwellpublishing.com/journals/APL
 
dc.publisher.placeAustralia
 
dc.relation.ispartofAPLAR Journal of Rheumatology
 
dc.relation.referencesReferences in Scopus
 
dc.subjectDisease Aetiology And Pathogenesis
 
dc.subjectInnate Immunity
 
dc.subjectLymphocyte Activation
 
dc.subjectSystemic Lupus Erythematous
 
dc.titleInnate immunity and systemic lupus erythematosus
 
dc.typeArticle
 
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Author Affiliations
  1. Nanjing University
  2. The University of Hong Kong