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Article: Ulcer and gastritis

TitleUlcer and gastritis
Authors
Issue Date2001
PublisherGeorg Thieme Verlag. The Journal's web site is located at http://www.thieme.de/endoscopy/index.html
Citation
Endoscopy, 2001, v. 33 n. 1, p. 8-15 How to Cite?
AbstractRecent progress in the area of ulcer and gastritis is still dominated by findings and reports on Helicobacter pylori and nonsteroidal anti-inflammatory drugs, which in turn are the two major causes of peptic ulcers. Although the prevalence of H. pylori is declining in most developed countries, it is still contributing to a significant proportion of peptic ulcers globally. The interrelationship of H. pylori gastritis in patients with gastroesophageal reflux has become more apparent. H. pylori-induced gastric body gastritis is associated with reduced acid production, and thus with reduced reflux and esophagitis. The controversies regarding the interactions between H. pylori and NSAIDs have still not been settled. With the availability of the new COX-2-specific inhibitors, the current scenario of NSAID-related gastroduodenal complications will certainly change. Short-term usage of these agents has significantly reduced the incidence of endoscopic ulcers, but the benefits in terms of clinical outcomes, such as bleeding or perforation, remain to be determined. This review summarizes the recent literature on peptic ulcer and gastritis.
Persistent Identifierhttp://hdl.handle.net/10722/162487
ISSN
2015 Impact Factor: 5.634
2015 SCImago Journal Rankings: 1.692
ISI Accession Number ID
References

 

DC FieldValueLanguage
dc.contributor.authorLeung, WKen_US
dc.contributor.authorGraham, DYen_US
dc.date.accessioned2012-09-05T05:20:28Z-
dc.date.available2012-09-05T05:20:28Z-
dc.date.issued2001en_US
dc.identifier.citationEndoscopy, 2001, v. 33 n. 1, p. 8-15en_US
dc.identifier.issn0013-726Xen_US
dc.identifier.urihttp://hdl.handle.net/10722/162487-
dc.description.abstractRecent progress in the area of ulcer and gastritis is still dominated by findings and reports on Helicobacter pylori and nonsteroidal anti-inflammatory drugs, which in turn are the two major causes of peptic ulcers. Although the prevalence of H. pylori is declining in most developed countries, it is still contributing to a significant proportion of peptic ulcers globally. The interrelationship of H. pylori gastritis in patients with gastroesophageal reflux has become more apparent. H. pylori-induced gastric body gastritis is associated with reduced acid production, and thus with reduced reflux and esophagitis. The controversies regarding the interactions between H. pylori and NSAIDs have still not been settled. With the availability of the new COX-2-specific inhibitors, the current scenario of NSAID-related gastroduodenal complications will certainly change. Short-term usage of these agents has significantly reduced the incidence of endoscopic ulcers, but the benefits in terms of clinical outcomes, such as bleeding or perforation, remain to be determined. This review summarizes the recent literature on peptic ulcer and gastritis.en_US
dc.languageengen_US
dc.publisherGeorg Thieme Verlag. The Journal's web site is located at http://www.thieme.de/endoscopy/index.htmlen_US
dc.relation.ispartofEndoscopyen_US
dc.subject.meshGastric Mucosa - Pathologyen_US
dc.subject.meshGastritis - Diagnosis - Pathologyen_US
dc.subject.meshHelicobacter Infections - Diagnosis - Pathologyen_US
dc.subject.meshHelicobacter Pylorien_US
dc.subject.meshHumansen_US
dc.subject.meshPeptic Ulcer - Diagnosis - Pathologyen_US
dc.subject.meshPrecancerous Conditions - Diagnosis - Pathologyen_US
dc.subject.meshRisk Factorsen_US
dc.titleUlcer and gastritisen_US
dc.typeArticleen_US
dc.identifier.emailLeung, WK:waikleung@hku.hken_US
dc.identifier.authorityLeung, WK=rp01479en_US
dc.description.naturelink_to_subscribed_fulltexten_US
dc.identifier.doi10.1055/s-2001-11183en_US
dc.identifier.pmid11204993-
dc.identifier.scopuseid_2-s2.0-0034960970en_US
dc.relation.referenceshttp://www.scopus.com/mlt/select.url?eid=2-s2.0-0034960970&selection=ref&src=s&origin=recordpageen_US
dc.identifier.volume33en_US
dc.identifier.issue1en_US
dc.identifier.spage8en_US
dc.identifier.epage15en_US
dc.identifier.isiWOS:000166433100002-
dc.publisher.placeGermanyen_US
dc.identifier.scopusauthoridLeung, WK=7201504523en_US
dc.identifier.scopusauthoridGraham, DY=7403477677en_US

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