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Conference Paper: Periimplant diseases: Where are we now? - Consensus of the Seventh European Workshop on Periodontology

TitlePeriimplant diseases: Where are we now? - Consensus of the Seventh European Workshop on Periodontology
Authors
Issue Date2011
PublisherBlackwell Munksgaard. The Journal's web site is located at http://www.blackwellpublishing.com/journals/CPE
Citation
Journal Of Clinical Periodontology, 2011, v. 38 SUPPL. 11, p. 178-181 How to Cite?
AbstractBackground: Peri-implant diseases present in two forms - peri-implant mucositis and peri-implantitis. Materials and Methods: The literature was systematically searched and critically reviewed. Four manuscripts were produced in specific topics identified as key areas to understand the microbial aetiology and the pathogenesis of peri-implant diseases and how the implant surface structure may affect pathogenesis. Results: While peri-implant mucositis represents the host response of the peri-implant tissues to the bacterial challenge that is not fundamentally different from gingivitis representing the host response to the bacterial challenge in the gingiva, peri-implantitis may differ from periodontitis both in the extent and the composition of cells in the lesion as well as the progression rate. A self-limiting process with a "protective" connective tissue capsule developing appears to dominate the periodontitis lesion while such a process may occasionally be lacking in peri-implantitis lesions. Bacterial biofilm formation on implant surfaces does not differ from that on tooth surfaces, but may be influenced by surface roughness. Nevertheless there is no evidence that such differences may influence the development of peri-implantitis. Conclusion: It was agreed that clinical and radiographic data should routinely be obtained after prosthesis installation on implants in order to establish a baseline for the diagnosis of peri-implantitis during maintenance of implant patients. © 2011 John Wiley & Sons A/S.
Persistent Identifierhttp://hdl.handle.net/10722/157958
ISSN
2015 Impact Factor: 3.915
2015 SCImago Journal Rankings: 1.848
ISI Accession Number ID
Funding AgencyGrant Number
Colgate
Funding Information:

This workshop has been financially supported by an unrestricted educational grant from Colgate. The sponsor had no impact on the program or on the deliberations of the European Workshop. Group participants declared that they had no conflict of interests.

References

 

DC FieldValueLanguage
dc.contributor.authorLang, NPen_US
dc.contributor.authorBerglundh, Ten_US
dc.date.accessioned2012-08-08T08:57:11Z-
dc.date.available2012-08-08T08:57:11Z-
dc.date.issued2011en_US
dc.identifier.citationJournal Of Clinical Periodontology, 2011, v. 38 SUPPL. 11, p. 178-181en_US
dc.identifier.issn0303-6979en_US
dc.identifier.urihttp://hdl.handle.net/10722/157958-
dc.description.abstractBackground: Peri-implant diseases present in two forms - peri-implant mucositis and peri-implantitis. Materials and Methods: The literature was systematically searched and critically reviewed. Four manuscripts were produced in specific topics identified as key areas to understand the microbial aetiology and the pathogenesis of peri-implant diseases and how the implant surface structure may affect pathogenesis. Results: While peri-implant mucositis represents the host response of the peri-implant tissues to the bacterial challenge that is not fundamentally different from gingivitis representing the host response to the bacterial challenge in the gingiva, peri-implantitis may differ from periodontitis both in the extent and the composition of cells in the lesion as well as the progression rate. A self-limiting process with a "protective" connective tissue capsule developing appears to dominate the periodontitis lesion while such a process may occasionally be lacking in peri-implantitis lesions. Bacterial biofilm formation on implant surfaces does not differ from that on tooth surfaces, but may be influenced by surface roughness. Nevertheless there is no evidence that such differences may influence the development of peri-implantitis. Conclusion: It was agreed that clinical and radiographic data should routinely be obtained after prosthesis installation on implants in order to establish a baseline for the diagnosis of peri-implantitis during maintenance of implant patients. © 2011 John Wiley & Sons A/S.en_US
dc.languageengen_US
dc.publisherBlackwell Munksgaard. The Journal's web site is located at http://www.blackwellpublishing.com/journals/CPEen_US
dc.relation.ispartofJournal of Clinical Periodontologyen_US
dc.subject.meshAlveolar Bone Loss - Etiology - Microbiologyen_US
dc.subject.meshBacterial Physiological Phenomenaen_US
dc.subject.meshBiofilmsen_US
dc.subject.meshDental Implants - Adverse Effects - Microbiologyen_US
dc.subject.meshDental Prosthesis Designen_US
dc.subject.meshDisease Progressionen_US
dc.subject.meshGingivitis - Microbiologyen_US
dc.subject.meshHumansen_US
dc.subject.meshPeriodontitis - Etiology - Microbiologyen_US
dc.subject.meshSurface Propertiesen_US
dc.titlePeriimplant diseases: Where are we now? - Consensus of the Seventh European Workshop on Periodontologyen_US
dc.typeConference_Paperen_US
dc.identifier.emailLang, NP:nplang@hkucc.hku.hken_US
dc.identifier.authorityLang, NP=rp00031en_US
dc.description.naturelink_to_subscribed_fulltexten_US
dc.identifier.doi10.1111/j.1600-051X.2010.01674.xen_US
dc.identifier.pmid21323713-
dc.identifier.scopuseid_2-s2.0-79951878893en_US
dc.relation.referenceshttp://www.scopus.com/mlt/select.url?eid=2-s2.0-79951878893&selection=ref&src=s&origin=recordpageen_US
dc.identifier.volume38en_US
dc.identifier.issueSUPPL. 11en_US
dc.identifier.spage178en_US
dc.identifier.epage181en_US
dc.identifier.isiWOS:000287402800017-
dc.publisher.placeDenmarken_US
dc.identifier.scopusauthoridLang, NP=7201577367en_US
dc.identifier.scopusauthoridBerglundh, T=26643204700en_US

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