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Article: Periodontal conditions in adult patients with cleft lip, alveolus, and palate

TitlePeriodontal conditions in adult patients with cleft lip, alveolus, and palate
Authors
Issue Date1992
PublisherAllen Press Inc. The Journal's web site is located at http://cpcj.allenpress.com
Citation
Cleft Palate-Craniofacial Journal, 1992, v. 29 n. 2, p. 179-185 How to Cite?
AbstractThe present study assessed the progression rate of periodontal disease over 8 years in a group of 52 adult patients with various forms of cleft lip, alveolus, and palate considered at risk for progression of periodontal disease. Of special interest was the evaluation of periodontal disease progression at sites adjacent to cleft regions compared to changes found at control sites not directly affected by such defects. High incidences of generalized plaque accumulation and bleeding on probing were noted at both examinations in 1979 and in 1987. A mean apical shift of the clinical attachment level amounting to 0.2 mm had occurred over the 8-year observation period. A slight apical displacement of the mesial and distal mean crestal alveolar bone was also noted. The rate of progression of periodontal disease over the 8 years was not found to be different at statistically significant levels at cleft sites compared to control sites. However, the results of this study documented that the cumulative periodontal destruction at 26 to 28 years of age was statistically significant and more pronounced at cleft sites as revealed by greater probing pocket depth and loss of clinical attachment. The differences between test and control sites amounted to 0.3 and 0.4 mm respectively for probing depth and 0.6 mm for loss of clinical attachment. In addition, the discrepancy between alveolar bone height and the levels of the clinical attachment at cleft sites demonstrated the presence of a long supracrestal connective tissue attachment adjacent to cleft defects. Therefore, the alveolar bone height as visualized in radiographs at such sites was considered an unreliable diagnostic tool for the assessment of the degree of periodontal destruction.
Persistent Identifierhttp://hdl.handle.net/10722/153776
ISSN
2010 Impact Factor: 0.77
2015 SCImago Journal Rankings: 0.685
ISI Accession Number ID

 

DC FieldValueLanguage
dc.contributor.authorBragger, Uen_US
dc.contributor.authorSchurch Jr, Een_US
dc.contributor.authorSalvi, Gen_US
dc.contributor.authorVon Wyttenbach, Ten_US
dc.contributor.authorLang, NPen_US
dc.date.accessioned2012-08-08T08:21:31Z-
dc.date.available2012-08-08T08:21:31Z-
dc.date.issued1992en_US
dc.identifier.citationCleft Palate-Craniofacial Journal, 1992, v. 29 n. 2, p. 179-185en_US
dc.identifier.issn1055-6656en_US
dc.identifier.urihttp://hdl.handle.net/10722/153776-
dc.description.abstractThe present study assessed the progression rate of periodontal disease over 8 years in a group of 52 adult patients with various forms of cleft lip, alveolus, and palate considered at risk for progression of periodontal disease. Of special interest was the evaluation of periodontal disease progression at sites adjacent to cleft regions compared to changes found at control sites not directly affected by such defects. High incidences of generalized plaque accumulation and bleeding on probing were noted at both examinations in 1979 and in 1987. A mean apical shift of the clinical attachment level amounting to 0.2 mm had occurred over the 8-year observation period. A slight apical displacement of the mesial and distal mean crestal alveolar bone was also noted. The rate of progression of periodontal disease over the 8 years was not found to be different at statistically significant levels at cleft sites compared to control sites. However, the results of this study documented that the cumulative periodontal destruction at 26 to 28 years of age was statistically significant and more pronounced at cleft sites as revealed by greater probing pocket depth and loss of clinical attachment. The differences between test and control sites amounted to 0.3 and 0.4 mm respectively for probing depth and 0.6 mm for loss of clinical attachment. In addition, the discrepancy between alveolar bone height and the levels of the clinical attachment at cleft sites demonstrated the presence of a long supracrestal connective tissue attachment adjacent to cleft defects. Therefore, the alveolar bone height as visualized in radiographs at such sites was considered an unreliable diagnostic tool for the assessment of the degree of periodontal destruction.en_US
dc.languageengen_US
dc.publisherAllen Press Inc. The Journal's web site is located at http://cpcj.allenpress.comen_US
dc.relation.ispartofCleft Palate-Craniofacial Journalen_US
dc.subject.meshAdolescenten_US
dc.subject.meshAdulten_US
dc.subject.meshAlveolar Bone Loss - Etiologyen_US
dc.subject.meshAlveolar Process - Abnormalitiesen_US
dc.subject.meshCleft Lip - Complicationsen_US
dc.subject.meshCleft Palate - Complicationsen_US
dc.subject.meshDental Calculus - Etiologyen_US
dc.subject.meshDental Plaque - Etiologyen_US
dc.subject.meshDenture, Partialen_US
dc.subject.meshFemaleen_US
dc.subject.meshGingival Hemorrhage - Etiologyen_US
dc.subject.meshGingival Pocket - Etiologyen_US
dc.subject.meshHumansen_US
dc.subject.meshMaleen_US
dc.subject.meshOral Hygieneen_US
dc.subject.meshPeriodontal Diseases - Etiology - Physiopathologyen_US
dc.subject.meshRisk Factorsen_US
dc.subject.meshTime Factorsen_US
dc.subject.meshTooth Loss - Etiologyen_US
dc.titlePeriodontal conditions in adult patients with cleft lip, alveolus, and palateen_US
dc.typeArticleen_US
dc.identifier.emailLang, NP:nplang@hkucc.hku.hken_US
dc.identifier.authorityLang, NP=rp00031en_US
dc.description.naturelink_to_subscribed_fulltexten_US
dc.identifier.doi10.1597/1545-1569(1992)029<0179:PCIAPW>2.3.CO;2en_US
dc.identifier.pmid1571353-
dc.identifier.scopuseid_2-s2.0-0026526103en_US
dc.identifier.volume29en_US
dc.identifier.issue2en_US
dc.identifier.spage179en_US
dc.identifier.epage185en_US
dc.identifier.isiWOS:A1992HL07900012-
dc.publisher.placeUnited Statesen_US
dc.identifier.scopusauthoridBragger, U=7005538598en_US
dc.identifier.scopusauthoridSchurch Jr, E=6603625444en_US
dc.identifier.scopusauthoridSalvi, G=35600695300en_US
dc.identifier.scopusauthoridVon Wyttenbach, T=6508084024en_US
dc.identifier.scopusauthoridLang, NP=7201577367en_US

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