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Conference Paper: Inhibition by chromium and cadmium of anaerobic acidogenesis

TitleInhibition by chromium and cadmium of anaerobic acidogenesis
Authors
Issue Date2001
PublisherI W A Publishing. The Journal's web site is located at http://www.iwapublishing.com/template.cfm?name=iwapwst
Citation
Water Science And Technology, 2001, v. 43 n. 11, p. 267-274 How to Cite?
AbstractThe effects of chromium (III) and cadmium on the anaerobic acidogenesis of a simulated dairy waste were examined using serum vials. At Cd dosages less than 20 mg/l, the acidogenesis process was enhanced by the dosage, resulting in a higher degree of acidification, protein conversion, and hydrogen production than the control. At dosages over 20-mg/l, Cd inhibited the acidogenesis. The Cr (III) dosage of 5 mg/l reduced overall volatile fatty acid and alcohol generation, degree of acidification, conversions of lactose, lipid and protein, and total biogas production, with the exception of accumulation of hydrogen and propionate. At dosages exceeding 5 mg/l, Cr (III) had a severe inhibition on the acidogenesis. The Cd concentrations which caused a 50% reduction in total volatile fatty acid and alcohol production, degree of acidification and cumulative gas production were higher than the corresponding values caused by Cr (III), suggesting that Cr (III) was more toxic to acidogenic bacteria than Cd.
Persistent Identifierhttp://hdl.handle.net/10722/152115
ISSN
2015 Impact Factor: 1.064
2015 SCImago Journal Rankings: 0.469
ISI Accession Number ID
References

 

DC FieldValueLanguage
dc.contributor.authorYu, HQen_US
dc.contributor.authorFang, HHPen_US
dc.date.accessioned2012-06-26T06:35:16Z-
dc.date.available2012-06-26T06:35:16Z-
dc.date.issued2001en_US
dc.identifier.citationWater Science And Technology, 2001, v. 43 n. 11, p. 267-274en_US
dc.identifier.issn0273-1223en_US
dc.identifier.urihttp://hdl.handle.net/10722/152115-
dc.description.abstractThe effects of chromium (III) and cadmium on the anaerobic acidogenesis of a simulated dairy waste were examined using serum vials. At Cd dosages less than 20 mg/l, the acidogenesis process was enhanced by the dosage, resulting in a higher degree of acidification, protein conversion, and hydrogen production than the control. At dosages over 20-mg/l, Cd inhibited the acidogenesis. The Cr (III) dosage of 5 mg/l reduced overall volatile fatty acid and alcohol generation, degree of acidification, conversions of lactose, lipid and protein, and total biogas production, with the exception of accumulation of hydrogen and propionate. At dosages exceeding 5 mg/l, Cr (III) had a severe inhibition on the acidogenesis. The Cd concentrations which caused a 50% reduction in total volatile fatty acid and alcohol production, degree of acidification and cumulative gas production were higher than the corresponding values caused by Cr (III), suggesting that Cr (III) was more toxic to acidogenic bacteria than Cd.en_US
dc.languageengen_US
dc.publisherI W A Publishing. The Journal's web site is located at http://www.iwapublishing.com/template.cfm?name=iwapwsten_US
dc.relation.ispartofWater Science and Technologyen_US
dc.subject.meshAlcohols - Metabolismen_US
dc.subject.meshAnaerobiosisen_US
dc.subject.meshBacteria - Drug Effectsen_US
dc.subject.meshCadmium - Pharmacologyen_US
dc.subject.meshChromium - Pharmacologyen_US
dc.subject.meshDairyingen_US
dc.subject.meshFatty Acids, Volatile - Metabolismen_US
dc.subject.meshHydrogen - Metabolismen_US
dc.subject.meshHydrogen-Ion Concentrationen_US
dc.subject.meshIndustrial Waste - Analysisen_US
dc.subject.meshProteins - Metabolismen_US
dc.subject.meshSewage - Chemistry - Microbiologyen_US
dc.subject.meshWater Microbiologyen_US
dc.titleInhibition by chromium and cadmium of anaerobic acidogenesisen_US
dc.typeConference_Paperen_US
dc.identifier.emailFang, HHP:hrechef@hkucc.hku.hken_US
dc.identifier.authorityFang, HHP=rp00115en_US
dc.description.naturelink_to_subscribed_fulltexten_US
dc.identifier.pmid11443972-
dc.identifier.scopuseid_2-s2.0-0034747966en_US
dc.relation.referenceshttp://www.scopus.com/mlt/select.url?eid=2-s2.0-0034747966&selection=ref&src=s&origin=recordpageen_US
dc.identifier.volume43en_US
dc.identifier.issue11en_US
dc.identifier.spage267en_US
dc.identifier.epage274en_US
dc.identifier.isiWOS:000170142600033-
dc.publisher.placeUnited Kingdomen_US
dc.identifier.scopusauthoridYu, HQ=13008678100en_US
dc.identifier.scopusauthoridFang, HHP=7402542625en_US

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