File Download
  Links for fulltext
     (May Require Subscription)
  • Find via Find It@HKUL
Supplementary

Conference Paper: PR interval prolongation is associated with endothelial dysfunction and activation of vascular repair in high risk cardiovascular patients

TitlePR interval prolongation is associated with endothelial dysfunction and activation of vascular repair in high risk cardiovascular patients
Authors
Chan, YHSiu, CWYiu, YFYiu, KHLi, SWLam, THLau, CPTse, HF
KeywordsMedical sciences
Cardiovascular diseases
Issue Date2011
PublisherLippincott Williams & Wilkins. The Journal's web site is located at http://circ.ahajournals.org
Citation
The 2011 Scientific Sessions of the American Heart Association (AHA), Orlando, FL., 12-16 November 2011. In Circulation, 2011, v. 124 n. 21 suppl., abstract 8488 How to Cite?
AbstractBACKGROUND: Recent studies showed that PR prolongation is associated with substantially increased risk of adverse clinical outcomes due to unclear mechanisms. OBJECTIVE: To investigate the relations between PR interval and indices of vascular function and endothelial repair in patients at high risk of cardiovascular events. METHODS: A total of 149 patients (mean age 63.6 ± 10.2 years …
DescriptionCore 7. Vascular Disease: Biology and Clinical Science
Session Title: Endothelial Function in Hyperglycemia and Type II Diabetes: abstract 8488
Open Access Journal
Persistent Identifierhttp://hdl.handle.net/10722/140185
ISSN
0009-7322
2023 Impact Factor: 35.5
2023 SCImago Journal Rankings: 8.415

 

DC FieldValueLanguage
dc.contributor.authorChan, YHen_US
dc.contributor.authorSiu, CWen_US
dc.contributor.authorYiu, YFen_US
dc.contributor.authorYiu, KHen_US
dc.contributor.authorLi, SWen_US
dc.contributor.authorLam, THen_US
dc.contributor.authorLau, CPen_US
dc.contributor.authorTse, HFen_US
dc.date.accessioned2011-09-23T06:08:13Z-
dc.date.available2011-09-23T06:08:13Z-
dc.date.issued2011en_US
dc.identifier.citationThe 2011 Scientific Sessions of the American Heart Association (AHA), Orlando, FL., 12-16 November 2011. In Circulation, 2011, v. 124 n. 21 suppl., abstract 8488en_US
dc.identifier.issn0009-7322-
dc.identifier.urihttp://hdl.handle.net/10722/140185-
dc.descriptionCore 7. Vascular Disease: Biology and Clinical Science-
dc.descriptionSession Title: Endothelial Function in Hyperglycemia and Type II Diabetes: abstract 8488-
dc.descriptionOpen Access Journal-
dc.description.abstractBACKGROUND: Recent studies showed that PR prolongation is associated with substantially increased risk of adverse clinical outcomes due to unclear mechanisms. OBJECTIVE: To investigate the relations between PR interval and indices of vascular function and endothelial repair in patients at high risk of cardiovascular events. METHODS: A total of 149 patients (mean age 63.6 ± 10.2 years …-
dc.languageengen_US
dc.publisherLippincott Williams & Wilkins. The Journal's web site is located at http://circ.ahajournals.org-
dc.relation.ispartofCirculationen_US
dc.subjectMedical sciences-
dc.subjectCardiovascular diseases-
dc.titlePR interval prolongation is associated with endothelial dysfunction and activation of vascular repair in high risk cardiovascular patientsen_US
dc.typeConference_Paperen_US
dc.identifier.emailChan, YH: chanwill@hku.hken_US
dc.identifier.emailSiu, CW: cwdsiu@hkucc.hku.hken_US
dc.identifier.emailYiu, KH: khkyiu@hku.hken_US
dc.identifier.emailLi, SW: lswli@hkucc.hku.hken_US
dc.identifier.emailLam, TH: hrmrlth@hkucc.hku.hken_US
dc.identifier.emailLau, CP: cplau@hku.hken_US
dc.identifier.emailTse, HF: hftse@hkucc.hku.hk-
dc.identifier.authorityChan, YH=rp01313en_US
dc.identifier.authoritySiu, CW=rp00534en_US
dc.identifier.authorityYiu, KH=rp01490en_US
dc.identifier.authorityLam, TH=rp00326en_US
dc.identifier.authorityTse, HF=rp00428en_US
dc.description.naturelink_to_OA_fulltext-
dc.identifier.hkuros194927en_US
dc.identifier.hkuros198851-
dc.identifier.volume124-
dc.identifier.issue21 suppl.-
dc.publisher.placeUnited States-
dc.customcontrol.immutablesml 140123-
dc.identifier.issnl0009-7322-

Export via OAI-PMH Interface in XML Formats

OR

Export to Other Non-XML Formats