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Article: Molecular characterization of a catalase-negative Staphylococcus aureus subsp. aureus strain collected from a patient with mitral valve endocarditis and pericarditis revealed a novel nonsense mutation in the katA gene

TitleMolecular characterization of a catalase-negative Staphylococcus aureus subsp. aureus strain collected from a patient with mitral valve endocarditis and pericarditis revealed a novel nonsense mutation in the katA gene
Authors
KeywordsCatalase
Adolescent
Antibiotic sensitivity
Antibody titer
Bacterial endocarditis
Issue Date2011
PublisherAmerican Society for Microbiology.
Citation
Journal Of Clinical Microbiology, 2011, v. 49 n. 9, p. 3398-3402 How to Cite?
Abstract
We report a case of endocarditis and pericarditis caused by catalase-negative Staphylococcus aureus. Molecular characterization revealed a novel nonsense mutation in the katA gene, leading to a loss of 238 amino acids (47% of the wild-type catalase protein), including the heme-binding site, NADPH-binding region, and Tyr-337, essential for catalysis. Copyright © 2011, American Society for Microbiology. All Rights Reserved.
Persistent Identifierhttp://hdl.handle.net/10722/139510
ISSN
2013 Impact Factor: 4.232
PubMed Central ID
ISI Accession Number ID
Funding AgencyGrant Number
University Development Fund
Committee on Research and Conference Grants, The University of Hong Kong
Funding Information:

This work is partly supported by the University Development Fund and the Committee on Research and Conference Grants, The University of Hong Kong.

References

 

Author Affiliations
  1. The University of Hong Kong
  2. United Christian Hospital Hong Kong
  3. Queen Mary Hospital Hong Kong
DC FieldValueLanguage
dc.contributor.authorTo, KKWen_HK
dc.contributor.authorCheng, VCCen_HK
dc.contributor.authorChan, JFWen_HK
dc.contributor.authorWong, ACYen_HK
dc.contributor.authorChau, Sen_HK
dc.contributor.authorTsang, FHFen_HK
dc.contributor.authorCurreem, SOTen_HK
dc.contributor.authorLau, SKPen_HK
dc.contributor.authorYuen, KYen_HK
dc.contributor.authorWoo, PCYen_HK
dc.date.accessioned2011-09-23T05:50:53Z-
dc.date.available2011-09-23T05:50:53Z-
dc.date.issued2011en_HK
dc.identifier.citationJournal Of Clinical Microbiology, 2011, v. 49 n. 9, p. 3398-3402en_HK
dc.identifier.issn0095-1137en_HK
dc.identifier.urihttp://hdl.handle.net/10722/139510-
dc.description.abstractWe report a case of endocarditis and pericarditis caused by catalase-negative Staphylococcus aureus. Molecular characterization revealed a novel nonsense mutation in the katA gene, leading to a loss of 238 amino acids (47% of the wild-type catalase protein), including the heme-binding site, NADPH-binding region, and Tyr-337, essential for catalysis. Copyright © 2011, American Society for Microbiology. All Rights Reserved.en_HK
dc.languageengen_US
dc.publisherAmerican Society for Microbiology.-
dc.relation.ispartofJournal of Clinical Microbiologyen_HK
dc.rightsJournal of Clinical Microbiology. Copyright © American Society for Microbiology.-
dc.subjectCatalase-
dc.subjectAdolescent-
dc.subjectAntibiotic sensitivity-
dc.subjectAntibody titer-
dc.subjectBacterial endocarditis-
dc.titleMolecular characterization of a catalase-negative Staphylococcus aureus subsp. aureus strain collected from a patient with mitral valve endocarditis and pericarditis revealed a novel nonsense mutation in the katA geneen_HK
dc.typeArticleen_HK
dc.identifier.openurlhttp://library.hku.hk:4550/resserv?sid=HKU:IR&issn=0095-1137&volume=49&issue=9&spage=3398&epage=3402&date=2011&atitle=Molecular+characterization+of+a+catalase-negative+Staphylococcus+aureus+subsp.+aureus+from+a+patient+with+mitral+valve+endocarditis+and+pericarditis+revealed+a+novel+nonsense+mutation+in+katA+gene-
dc.identifier.emailTo, KKW: kelvinto@hkucc.hku.hken_HK
dc.identifier.emailChan, JFW: jfwchan@hku.hken_HK
dc.identifier.emailLau, SKP: skplau@hkucc.hku.hken_HK
dc.identifier.emailYuen, KY: kyyuen@hkucc.hku.hken_HK
dc.identifier.emailWoo, PCY: pcywoo@hkucc.hku.hken_HK
dc.identifier.authorityTo, KKW=rp01384en_HK
dc.identifier.authorityChan, JFW=rp01736en_HK
dc.identifier.authorityLau, SKP=rp00486en_HK
dc.identifier.authorityYuen, KY=rp00366en_HK
dc.identifier.authorityWoo, PCY=rp00430en_HK
dc.description.naturelink_to_OA_fulltexten_US
dc.identifier.doi10.1128/JCM.00849-11en_HK
dc.identifier.pmid21715584en_HK
dc.identifier.pmcidPMC3165592-
dc.identifier.scopuseid_2-s2.0-80052202334en_HK
dc.identifier.hkuros192140en_US
dc.identifier.hkuros190821en_US
dc.relation.referenceshttp://www.scopus.com/mlt/select.url?eid=2-s2.0-80052202334&selection=ref&src=s&origin=recordpageen_HK
dc.identifier.volume49en_HK
dc.identifier.issue9en_HK
dc.identifier.spage3398en_HK
dc.identifier.epage3402en_HK
dc.identifier.isiWOS:000294416000057-
dc.publisher.placeUnited Statesen_HK
dc.identifier.scopusauthoridTo, KKW=14323807300en_HK
dc.identifier.scopusauthoridCheng, VCC=23670479400en_HK
dc.identifier.scopusauthoridChan, JFW=24278817900en_HK
dc.identifier.scopusauthoridWong, ACY=36921626000en_HK
dc.identifier.scopusauthoridChau, S=55151628900en_HK
dc.identifier.scopusauthoridTsang, FHF=7003264965en_HK
dc.identifier.scopusauthoridCurreem, SOT=16416762100en_HK
dc.identifier.scopusauthoridLau, SKP=7401596211en_HK
dc.identifier.scopusauthoridYuen, KY=36078079100en_HK
dc.identifier.scopusauthoridWoo, PCY=7201801340en_HK

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