Article: Naturally occurring inhibitors against the formation of advanced glycation end-products
| Title | Naturally occurring inhibitors against the formation of advanced glycation end-products |
|---|---|
| Authors | Peng, X1 Ma, J1 Chen, SF Wang, M1 |
| Issue Date | 2011 |
| Publisher | Royal Society of Chemistry. The Journal's web site is located at http://pubs.rsc.org/en/journals/journalissues/fo |
| Citation | Food & Function, 2011, v. 2 n. 6, p. 289-301 [How to Cite?] DOI: http://dx.doi.org/10.1039/c1fo10034c |
| Abstract | Advanced glycation end-products (AGEs) are the final products of the non-enzymatic reaction between reducing sugars and amino groups in proteins, lipids and nucleic acids. Recently, the accumulation of AGEs in vivo has been implicated as a major pathogenic process in diabetic complications, atherosclerosis, Alzheimer's disease and normal aging. The early recognition of AGEs can ascend to the late 1960s when a non-enzymatic glycation process was found in human body which is similar to the Maillard reaction. To some extent, AGEs can be regarded as products of the Maillard reaction. This review firstly introduces the Maillard reaction, the formation process of AGEs and harmful effects of AGEs to human health. As AGEs can cause undesirable diseases or disorders, it is necessary to investigate AGE inhibitors to offer a potential therapeutic approach for the prevention of diabetic or other pathogenic complications induced by AGEs. Typical effective AGE inhibitors with different inhibition mechanisms are also reviewed in this paper. Both synthetic compounds and natural products have been evaluated as inhibitors against the formation of AGEs. However, considering toxic or side effects of synthetic molecules present in clinical trials, natural products are more promising to be developed as potent AGE inhibitors. |
| ISSN | 2042-6496 2011 Impact Factor: 1.179 |
| DOI | http://dx.doi.org/10.1039/c1fo10034c |
| ISI Accession Number ID | WOS:000291992800001 |
| dc.contributor.author | Peng, X |
|---|---|
| dc.contributor.author | Ma, J |
| dc.contributor.author | Chen, SF |
| dc.contributor.author | Wang, M |
| dc.date.accessioned | 2011-07-27T02:11:39Z |
| dc.date.available | 2011-07-27T02:11:39Z |
| dc.date.issued | 2011 |
| dc.description.abstract | Advanced glycation end-products (AGEs) are the final products of the non-enzymatic reaction between reducing sugars and amino groups in proteins, lipids and nucleic acids. Recently, the accumulation of AGEs in vivo has been implicated as a major pathogenic process in diabetic complications, atherosclerosis, Alzheimer's disease and normal aging. The early recognition of AGEs can ascend to the late 1960s when a non-enzymatic glycation process was found in human body which is similar to the Maillard reaction. To some extent, AGEs can be regarded as products of the Maillard reaction. This review firstly introduces the Maillard reaction, the formation process of AGEs and harmful effects of AGEs to human health. As AGEs can cause undesirable diseases or disorders, it is necessary to investigate AGE inhibitors to offer a potential therapeutic approach for the prevention of diabetic or other pathogenic complications induced by AGEs. Typical effective AGE inhibitors with different inhibition mechanisms are also reviewed in this paper. Both synthetic compounds and natural products have been evaluated as inhibitors against the formation of AGEs. However, considering toxic or side effects of synthetic molecules present in clinical trials, natural products are more promising to be developed as potent AGE inhibitors. |
| dc.identifier.citation | Food & Function, 2011, v. 2 n. 6, p. 289-301 [How to Cite?] DOI: http://dx.doi.org/10.1039/c1fo10034c |
| dc.identifier.doi | http://dx.doi.org/10.1039/c1fo10034c |
| dc.identifier.epage | 301 |
| dc.identifier.hkuros | 186787 |
| dc.identifier.isi | WOS:000291992800001 |
| dc.identifier.issn | 2042-6496 2011 Impact Factor: 1.179 |
| dc.identifier.issue | 6 |
| dc.identifier.pmid | 21779567 |
| dc.identifier.scopus | eid_2-s2.0-84862833280 |
| dc.identifier.spage | 289 |
| dc.identifier.uri | http://hdl.handle.net/10722/136246 |
| dc.identifier.volume | 2 |
| dc.language | eng |
| dc.publisher | Royal Society of Chemistry. The Journal's web site is located at http://pubs.rsc.org/en/journals/journalissues/fo |
| dc.publisher.place | United Kingdom |
| dc.relation.ispartof | Food & Function |
| dc.subject.mesh | Alzheimer Disease |
| dc.subject.mesh | Atherosclerosis |
| dc.subject.mesh | Biological Agents - pharmacology |
| dc.subject.mesh | Diabetes Complications |
| dc.subject.mesh | Glycosylation End Products, Advanced - adverse effects - antagonists and inhibitors - metabolism |
| dc.title | Naturally occurring inhibitors against the formation of advanced glycation end-products |
| dc.type | Article |
Author Affiliations
- The University of Hong Kong
- Peking University

